David A. Tulis

1.6k total citations
46 papers, 1.4k citations indexed

About

David A. Tulis is a scholar working on Molecular Biology, Physiology and Pharmacology. According to data from OpenAlex, David A. Tulis has authored 46 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 20 papers in Physiology and 7 papers in Pharmacology. Recurrent topics in David A. Tulis's work include Nitric Oxide and Endothelin Effects (18 papers), Heme Oxygenase-1 and Carbon Monoxide (13 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). David A. Tulis is often cited by papers focused on Nitric Oxide and Endothelin Effects (18 papers), Heme Oxygenase-1 and Carbon Monoxide (13 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). David A. Tulis collaborates with scholars based in United States, India and Czechia. David A. Tulis's co-authors include Kelly J. Peyton, William Durante, Alida J. Evans, Andrew I. Schafer, Russell L. Prewitt, Joseph L. Unthank, Xiaoming Liu, Andrew W. Holt, Jianming Xu and Yuhui Yuan and has published in prestigious journals such as Circulation, PLoS ONE and Biochemical and Biophysical Research Communications.

In The Last Decade

David A. Tulis

45 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David A. Tulis United States 21 827 316 218 179 151 46 1.4k
Jun Ni China 22 680 0.8× 254 0.8× 149 0.7× 180 1.0× 180 1.2× 58 1.6k
Saula de Kreutzenberg Italy 17 764 0.9× 302 1.0× 350 1.6× 267 1.5× 157 1.0× 23 1.7k
Jee‐Young Han South Korea 29 670 0.8× 293 0.9× 251 1.2× 341 1.9× 128 0.8× 56 2.1k
Xue-Liang Du United States 6 660 0.8× 549 1.7× 222 1.0× 213 1.2× 164 1.1× 6 2.0k
Xu Teng China 26 563 0.7× 272 0.9× 217 1.0× 246 1.4× 86 0.6× 88 1.9k
Noriko Takahara Japan 15 781 0.9× 512 1.6× 363 1.7× 223 1.2× 110 0.7× 26 1.9k
Sheng Zhong Duan United States 14 863 1.0× 285 0.9× 331 1.5× 344 1.9× 106 0.7× 19 1.5k
Yi Pan Canada 18 591 0.7× 392 1.2× 457 2.1× 242 1.4× 139 0.9× 41 1.7k
Franziska Theilig Germany 27 1.0k 1.2× 278 0.9× 385 1.8× 193 1.1× 94 0.6× 50 2.3k
Voahanginirina Randriamboavonjy Germany 22 539 0.7× 338 1.1× 241 1.1× 93 0.5× 183 1.2× 42 1.3k

Countries citing papers authored by David A. Tulis

Since Specialization
Citations

This map shows the geographic impact of David A. Tulis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Tulis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Tulis more than expected).

Fields of papers citing papers by David A. Tulis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Tulis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Tulis. The network helps show where David A. Tulis may publish in the future.

Co-authorship network of co-authors of David A. Tulis

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Tulis. A scholar is included among the top collaborators of David A. Tulis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Tulis. David A. Tulis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Tulis, David A., et al.. (2020). Increased AMP deaminase activity decreases ATP content and slows protein degradation in cultured skeletal muscle. Metabolism. 108. 154257–154257. 20 indexed citations
3.
Tulis, David A.. (2017). Novel protein kinase targets in vascular smooth muscle therapeutics. Current Opinion in Pharmacology. 33. 12–16. 4 indexed citations
4.
Holt, Andrew W., Patti R. Shaver, Shaquria Adderley, et al.. (2016). Soluble guanylyl cyclase-activated cyclic GMP-dependent protein kinase inhibits arterial smooth muscle cell migration independent of VASP-serine 239 phosphorylation. Cellular Signalling. 28(9). 1364–1379. 21 indexed citations
5.
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Dong, Lixue, et al.. (2013). Abstract 11587: Acidosis/GPR4 Signaling Regulates Inflammatory and Endoplasmic Reticulum Stress Responses in Vascular Endothelial Cells. Circulation. 128. 2 indexed citations
7.
Johnson, Tracy, et al.. (2013). The Dopamine D3 Receptor Knockout Mouse Mimics Aging-Related Changes in Autonomic Function and Cardiac Fibrosis. PLoS ONE. 8(8). e74116–e74116. 23 indexed citations
8.
Shaver, Patti R., et al.. (2012). Control of Vascular Smooth Muscle Cell Growth by Connexin 43. Frontiers in Physiology. 3. 220–220. 23 indexed citations
9.
Adderley, Shaquria, et al.. (2012). Phosphodiesterases Regulate BAY 41-2272-Induced VASP Phosphorylation in Vascular Smooth Muscle Cells. Frontiers in Pharmacology. 3. 10–10. 14 indexed citations
10.
Liu, Xiaoming, Kelly J. Peyton, Natalia Mendelev, et al.. (2008). YC-1 Stimulates the Expression of Gaseous Monoxide-Generating Enzymes in Vascular Smooth Muscle Cells. Molecular Pharmacology. 75(1). 208–217. 16 indexed citations
11.
Tulis, David A.. (2008). Novel Therapies for Cyclic GMP Control of Vascular Smooth Muscle Growth. American Journal of Therapeutics. 15(6). 551–564. 17 indexed citations
12.
Mukhopadhyay, Somnath & David A. Tulis. (2007). Endocannabinoid Regulation of Matrix Metalloproteinases: Implications in Ischemic Stroke. Cardiovascular & Hematological Agents in Medicinal Chemistry. 5(4). 311–318. 6 indexed citations
13.
Tulis, David A.. (2007). Histological and Morphometric Analyses for Rat Carotid Balloon Injury Model. Methods in molecular medicine. 139. 31–66. 29 indexed citations
14.
Tulis, David A.. (2007). Rat Carotid Artery Balloon Injury Model. Methods in molecular medicine. 139. 1–30. 106 indexed citations
15.
Tulis, David A.. (2006). Methods for Identifying Cardiovascular Agents: A Review. Recent Advances in Cardiovascular Drug Discovery (Formerly Recent Patents on Cardiovascular Drug Discovery). 1(1). 47–56. 7 indexed citations
16.
Masters, Kristyn S., Elizabeth A. Lipke, Heather Myler, et al.. (2005). Nitric oxide-generating hydrogels inhibit neointima formation. Journal of Biomaterials Science Polymer Edition. 16(5). 659–672. 40 indexed citations
17.
Tulis, David A.. (2004). Salutary Properties of YC-1 in the Cardiovascular and Hematological Systems. PubMed. 2(4). 343–359. 17 indexed citations
18.
Tulis, David A., Kristyn S. Masters, Elizabeth A. Lipke, et al.. (2002). YC-1-Mediated Vascular Protection through Inhibition of Smooth Muscle Cell Proliferation and Platelet Function. Biochemical and Biophysical Research Communications. 291(4). 1014–1021. 45 indexed citations
19.
Tulis, David A., William Durante, Kelly J. Peyton, Alida J. Evans, & Andrew I. Schafer. (2001). Heme oxygenase-1 attenuates vascular remodeling following balloon injury in rat carotid arteries. Atherosclerosis. 155(1). 113–122. 123 indexed citations
20.
Tulis, David A. & Russell L. Prewitt. (1998). Medial and Endothelial Platelet-Derived Growth Factor A Chain Expression Is Regulated by in vivo Exposure to Elevated Flow. Journal of Vascular Research. 35(6). 413–420. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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