David A. Rempe

1.9k total citations
20 papers, 1.2k citations indexed

About

David A. Rempe is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cancer Research. According to data from OpenAlex, David A. Rempe has authored 20 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 6 papers in Cancer Research. Recurrent topics in David A. Rempe's work include Neuroscience and Neuropharmacology Research (9 papers), Cancer, Hypoxia, and Metabolism (6 papers) and Mitochondrial Function and Pathology (5 papers). David A. Rempe is often cited by papers focused on Neuroscience and Neuropharmacology Research (9 papers), Cancer, Hypoxia, and Metabolism (6 papers) and Mitochondrial Function and Pathology (5 papers). David A. Rempe collaborates with scholars based in United States, Sweden and Czechia. David A. Rempe's co-authors include Yanxin Zhao, Howard J. Federoff, Patrick S. Mangan, Eric W. Lothman, Edward H. Bertram, Maiken Nedergaard, Albee Messing, Michael V. Sofroniew, Nihal DeLanerolle and Nathan B. Fountain and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and The Journal of Cell Biology.

In The Last Decade

David A. Rempe

20 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David A. Rempe United States 16 559 503 284 185 173 20 1.2k
Elena Pravettoni Italy 12 808 1.4× 875 1.7× 462 1.6× 177 1.0× 181 1.0× 12 1.8k
Julien Chuquet France 13 429 0.8× 633 1.3× 562 2.0× 217 1.2× 234 1.4× 23 1.3k
Rémi Bos France 14 475 0.8× 474 0.9× 106 0.4× 118 0.6× 176 1.0× 32 1.1k
Alessandra L. Scotti Switzerland 16 373 0.7× 573 1.1× 197 0.7× 112 0.6× 121 0.7× 28 921
Johanne Egge Rinholm Norway 12 763 1.4× 512 1.0× 258 0.9× 99 0.5× 222 1.3× 17 1.3k
P. Elyse Schauwecker United States 20 729 1.3× 1.0k 2.0× 238 0.8× 117 0.6× 137 0.8× 32 1.6k
Fredrik Blomstrand Sweden 20 742 1.3× 618 1.2× 351 1.2× 101 0.5× 249 1.4× 29 1.4k
Évelyne Tremblay France 17 746 1.3× 1.0k 2.0× 113 0.4× 205 1.1× 287 1.7× 22 1.6k
Sean J. Mulligan Canada 15 499 0.9× 651 1.3× 525 1.8× 147 0.8× 254 1.5× 18 1.6k
Toshihiko Momiyama Japan 20 667 1.2× 693 1.4× 129 0.5× 196 1.1× 77 0.4× 55 1.3k

Countries citing papers authored by David A. Rempe

Since Specialization
Citations

This map shows the geographic impact of David A. Rempe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Rempe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Rempe more than expected).

Fields of papers citing papers by David A. Rempe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Rempe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Rempe. The network helps show where David A. Rempe may publish in the future.

Co-authorship network of co-authors of David A. Rempe

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Rempe. A scholar is included among the top collaborators of David A. Rempe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Rempe. David A. Rempe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rempe, David A.. (2014). Predicting Outcomes After Transient Ischemic Attack and Stroke. CONTINUUM Lifelong Learning in Neurology. 20(2). 412–428. 7 indexed citations
2.
Verkhratsky, Alexei, Michael V. Sofroniew, Albee Messing, et al.. (2012). Neurological Diseases as Primary Gliopathies: A Reassessment of Neurocentrism. ASN NEURO. 4(3). 206 indexed citations
3.
Rempe, David A., et al.. (2011). Cocultures of Neurons and Astrocytes as a Model for Examining Hypoxia-Induced Neuronal Death. Methods in molecular biology. 814. 353–366. 1 indexed citations
4.
Zhao, Yanxin & David A. Rempe. (2011). Prophylactic neuroprotection against stroke: Low-dose, prolonged treatment with deferoxamine or deferasirox establishes prolonged neuroprotection independent of HIF-1 function. Journal of Cerebral Blood Flow & Metabolism. 31(6). 1412–1423. 44 indexed citations
5.
Zhao, Yanxin & David A. Rempe. (2010). Targeting Astrocytes for Stroke Therapy. Neurotherapeutics. 7(4). 439–451. 141 indexed citations
6.
Li, Yan & David A. Rempe. (2010). During hypoxia, HUMMR joins the mitochondrial dance. Cell Cycle. 9(1). 50–57. 13 indexed citations
7.
Rempe, David A., Takahiro Takano, & Maiken Nedergaard. (2009). TR(I)Pping towards treatment for ischemia. Nature Neuroscience. 12(10). 1215–1216. 10 indexed citations
8.
Rempe, David A., et al.. (2009). The Janus-Faced Effects of Hypoxia on Astrocyte Function. The Neuroscientist. 15(6). 579–588. 39 indexed citations
9.
Li, Yan, et al.. (2009). HUMMR, a hypoxia- and HIF-1α–inducible protein, alters mitochondrial distribution and transport. The Journal of Cell Biology. 185(6). 1065–1081. 82 indexed citations
10.
Halterman, Marc W., et al.. (2008). Loss of c/EBP-β activity promotes the adaptive to apoptotic switch in hypoxic cortical neurons. Molecular and Cellular Neuroscience. 38(2). 125–137. 27 indexed citations
11.
Federoff, Howard J., et al.. (2008). The Good, the Bad, and the Cell Type-Specific Roles of Hypoxia Inducible Factor-1α in Neurons and Astrocytes. Journal of Neuroscience. 28(8). 1988–1993. 146 indexed citations
12.
Rempe, David A., et al.. (2007). In Cultured Astrocytes, p53 and MDM2 Do Not Alter Hypoxia-inducible Factor-1α Function Regardless of the Presence of DNA Damage. Journal of Biological Chemistry. 282(22). 16187–16201. 21 indexed citations
13.
Kyrozis, Andreas, David A. Rempe, Daniela Uziel, et al.. (2005). Blockade of Gap Junctions In Vivo Provides Neuroprotection After Perinatal Global Ischemia. Stroke. 36(10). 2232–2237. 112 indexed citations
14.
Bertram, Edward H., et al.. (1998). Functional anatomy of limbic epilepsy: a proposal for central synchronization of a diffusely hyperexcitable network. Epilepsy Research. 32(1-2). 194–205. 145 indexed citations
15.
Bánkfalvi, Ágnes, et al.. (1998). Gains and losses of CD44 expression during breast carcinogenesis and tumour progression. Histopathology. 33(2). 107–116. 57 indexed citations
16.
Rempe, David A., Edward H. Bertram, John Williamson, & Eric W. Lothman. (1997). Interneurons in Area CA1 Stratum Radiatum and Stratum Oriens Remain Functionally Connected to Excitatory Synaptic Input in Chronically Epileptic Animals. Journal of Neurophysiology. 78(3). 1504–1515. 42 indexed citations
17.
Rempe, David A., Patrick S. Mangan, & Eric W. Lothman. (1995). Regional heterogeneity of pathophysiological alterations in CA1 and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of Neurophysiology. 74(2). 816–828. 20 indexed citations
18.
Mangan, Patrick S., David A. Rempe, & Eric W. Lothman. (1995). Changes in inhibitory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of Neurophysiology. 74(2). 829–840. 60 indexed citations
19.
Lothman, Eric W., David A. Rempe, & Patrick S. Mangan. (1995). Changes in excitatory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of Neurophysiology. 74(2). 841–848. 42 indexed citations
20.
Bekenstein, Jonathan W., David A. Rempe, & Eric W. Lothman. (1993). Decreased heterosynaptic and homosynaptic paired pulse inhibition in the rat hippocampus as a chronic sequela to limbic status epilepticus. Brain Research. 601(1-2). 111–120. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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