David A. Ingram

13.7k total citations · 4 hit papers
119 papers, 10.2k citations indexed

About

David A. Ingram is a scholar working on Molecular Biology, Neurology and Oncology. According to data from OpenAlex, David A. Ingram has authored 119 papers receiving a total of 10.2k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Molecular Biology, 28 papers in Neurology and 20 papers in Oncology. Recurrent topics in David A. Ingram's work include Angiogenesis and VEGF in Cancer (38 papers), Neurofibromatosis and Schwannoma Cases (23 papers) and Congenital heart defects research (12 papers). David A. Ingram is often cited by papers focused on Angiogenesis and VEGF in Cancer (38 papers), Neurofibromatosis and Schwannoma Cases (23 papers) and Congenital heart defects research (12 papers). David A. Ingram collaborates with scholars based in United States, United Kingdom and India. David A. Ingram's co-authors include Mervin C. Yöder, Laura E. Mead, Daniel N. Prater, Jamie Case, Karen K. Hirschi, Fang Li, Karen E. Pollok, Hiromi Tanaka, David Gilley and Kelly E. Mortell and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Circulation.

In The Last Decade

David A. Ingram

119 papers receiving 10.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification of a novel hierarchy of endothelial progenitor cells using human peripheral and umbilical cord blood

2004 1.2k citations David A. Ingram, Laura E. Mead et al. profile →
Redefining endothelial progenitor cells via clonal analysis and hematopoietic stem/progenitor cell principals

2006 1.2k citations Mervin C. Yöder, Laura E. Mead et al. profile →
Assessing Identity, Phenotype, and Fate of Endothelial Progenitor Cells

2008 585 citations David A. Ingram, Mervin C. Yöder et al. profile →
Early Pulmonary Vascular Disease in Preterm Infants at Risk for Bronchopulmonary Dysplasia

2014 316 citations Peter M. Mourani, Marci K. Sontag et al. American Journal of Respiratory and Critical Care Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David A. Ingram United States	46	5.4k	2.0k	1.9k	1.9k	1.5k	119	10.2k
Nazneen Aziz United States	9	9.4k 1.7×	1.6k 0.8×	1.5k 0.8×	1.4k 0.7×	1.2k 0.8×	9	19.7k
Elaine Spector United States	26	10.1k 1.9×	1.7k 0.8×	1.5k 0.8×	1.4k 0.7×	1.2k 0.8×	71	20.6k
Julie M. Gastier‐Foster United States	36	10.4k 1.9×	1.8k 0.9×	2.1k 1.1×	1.7k 0.9×	1.9k 1.3×	122	22.8k
Dean Y. Li United States	51	4.1k 0.8×	1.9k 1.0×	1.2k 0.7×	516 0.3×	595 0.4×	119	9.9k
Beatrice Nico Italy	59	6.0k 1.1×	886 0.4×	869 0.5×	870 0.5×	2.0k 1.3×	236	10.8k
Karl H. Plate Germany	68	10.8k 2.0×	1.1k 0.5×	1.8k 1.0×	2.6k 1.4×	3.1k 2.0×	159	17.1k
Annika Armulik Sweden	19	3.9k 0.7×	822 0.4×	677 0.4×	873 0.5×	1.2k 0.8×	20	8.7k
S. Paul Oh United States	43	4.5k 0.8×	1.0k 0.5×	1.3k 0.7×	1.3k 0.7×	599 0.4×	97	7.5k
Daniel F. Bowen‐Pope United States	54	6.9k 1.3×	2.0k 1.0×	1.3k 0.7×	909 0.5×	1.3k 0.9×	105	13.1k
Charles E. Jackson United States	51	4.8k 0.9×	2.0k 1.0×	2.0k 1.1×	1.8k 1.0×	2.3k 1.5×	139	11.6k

Countries citing papers authored by David A. Ingram

Since Specialization

Citations

This map shows the geographic impact of David A. Ingram's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Ingram with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Ingram more than expected).

Fields of papers citing papers by David A. Ingram

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Ingram. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Ingram. The network helps show where David A. Ingram may publish in the future.

Co-authorship network of co-authors of David A. Ingram

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Ingram. A scholar is included among the top collaborators of David A. Ingram based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Ingram. David A. Ingram is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Ssentongo, Paddy, et al.. (2023). Optimal Duration of Systemic Corticosteroids in Coronavirus Disease 2019 Treatment: A Systematic Review and Meta-analysis. Open Forum Infectious Diseases. 10(3). ofad105–ofad105. 5 indexed citations

2.

Wagner, Brandie D., David A. Ingram, Brenda B. Poindexter, et al.. (2017). Antenatal Determinants of Bronchopulmonary Dysplasia and Late Respiratory Disease in Preterm Infants. American Journal of Respiratory and Critical Care Medicine. 196(3). 364–374. 124 indexed citations

3.

Bessler, Waylan, Farlyn Z. Hudson, Hanfang Zhang, et al.. (2016). Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans. Free Radical Biology and Medicine. 97. 212–222. 22 indexed citations

4.

Mourani, Peter M., Marci K. Sontag, Adel K. Younoszai, et al.. (2015). Early pulmonary vascular disease in preterm infants at risk for bronchopulmonary dysplasia. PMC. 5 indexed citations

5.

Stansfield, Brian K. & David A. Ingram. (2015). Clinical significance of monocyte heterogeneity. PMC. 2 indexed citations

6.

Maria, Michael V. Di, Adel K. Younoszai, Marci K. Sontag, et al.. (2015). Maturational Changes in Diastolic Longitudinal Myocardial Velocity in Preterm Infants. Journal of the American Society of Echocardiography. 28(9). 1045–1052. 28 indexed citations

7.

Mourani, Peter M., Marci K. Sontag, Adel K. Younoszai, et al.. (2014). Early Pulmonary Vascular Disease in Preterm Infants at Risk for Bronchopulmonary Dysplasia. American Journal of Respiratory and Critical Care Medicine. 191(1). 87–95. 316 indexed citations breakdown →

8.

Mendonca, Marc S., et al.. (2011). Differential Mechanisms of X-Ray-Induced Cell Death in Human Endothelial Progenitor Cells Isolated from Cord Blood and Adults. Radiation Research. 176(2). 208–216. 27 indexed citations

9.

Baker, Christopher D., Sharon Ryan, David A. Ingram, et al.. (2009). Endothelial Colony-forming Cells from Preterm Infants Are Increased and More Susceptible to Hyperoxia. American Journal of Respiratory and Critical Care Medicine. 180(5). 454–461. 91 indexed citations

10.

Timmermans, Frank, Jean Plum, Mervin C. Yöder, et al.. (2009). Endothelial progenitor cells: identity defined?. PMC. 6 indexed citations

11.

Snider, Paige, Robert B. Hinton, Ricardo A. Moreno‐Rodriguez, et al.. (2008). Periostin Is Required for Maturation and Extracellular Matrix Stabilization of Noncardiomyocyte Lineages of the Heart. Circulation Research. 102(7). 752–760. 269 indexed citations

12.

Case, Jamie, David A. Ingram, & Laura S. Haneline. (2008). Oxidative Stress Impairs Endothelial Progenitor Cell Function. Antioxidants and Redox Signaling. 10(11). 1895–1907. 88 indexed citations

13.

Li, Fang, et al.. (2008). Rap1a Is a Key Regulator of Fibroblast Growth Factor 2-Induced Angiogenesis and Together with Rap1b Controls Human Endothelial Cell Functions. Molecular and Cellular Biology. 28(18). 5803–5810. 86 indexed citations

14.

Yang, Feng‐Chun, Shi Chen, Hilary White, et al.. (2007). K-ras Is Critical for Modulating Multiple c-kit-Mediated Cellular Functions in Wild-Type and Nf1 +/− Mast Cells. The Journal of Immunology. 178(4). 2527–2534. 28 indexed citations

15.

Yang, Feng‐Chun, Chen Shi, Xiaohong Li, et al.. (2006). Nf1+/− mast cells induce neurofibroma like phenotypes through secreted TGF-β signaling. Human Molecular Genetics. 15(16). 2421–2437. 123 indexed citations

16.

Yang, Feng‐Chun, Shi Chen, Alexander G. Robling, et al.. (2006). Hyperactivation of p21ras and PI3K cooperate to alter murine and human neurofibromatosis type 1–haploinsufficient osteoclast functions. Journal of Clinical Investigation. 116(11). 2880–2891. 99 indexed citations

17.

Hiatt, Kelly, et al.. (2004). Loss of the Nf1 Tumor Suppressor Gene Decreases Fas Antigen Expression in Myeloid Cells. American Journal Of Pathology. 164(4). 1471–1479. 21 indexed citations

18.

Ingram, David A., Shi Chen, Cynthia M. Hingtgen, et al.. (2003). Neurofibromin-deficient Schwann cells secrete a potent migratory stimulus for Nf1+/– mast cells. Journal of Clinical Investigation. 112(12). 1851–1861. 27 indexed citations

19.

Ingram, David A., Shi Chen, Cynthia M. Hingtgen, et al.. (2003). Neurofibromin-deficient Schwann cells secrete a potent migratory stimulus for Nf1+/– mast cells. Journal of Clinical Investigation. 112(12). 1851–1861. 170 indexed citations

20.

Farmer, Simon F., Linda M. Harrison, David A. Ingram, & John A. Stephens. (1991). Plasticity of central motor pathways in children with hemiplegic cerebral palsy. Neurology. 41(9). 1505–1505. 169 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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