Daphne Dubach

463 total citations
8 papers, 307 citations indexed

About

Daphne Dubach is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Cell Biology. According to data from OpenAlex, Daphne Dubach has authored 8 papers receiving a total of 307 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 2 papers in Public Health, Environmental and Occupational Health and 2 papers in Cell Biology. Recurrent topics in Daphne Dubach's work include Signaling Pathways in Disease (3 papers), Down syndrome and intellectual disability research (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Daphne Dubach is often cited by papers focused on Signaling Pathways in Disease (3 papers), Down syndrome and intellectual disability research (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Daphne Dubach collaborates with scholars based in Australia, Spain and United States. Daphne Dubach's co-authors include Melanie Pritchard, Damien J. Keating, Yong Yu, David I. Finkelstein, Katherine Martin, María L. Arbonés, Sílvia Porta, David N. Bowser, Mark Zanin and Ian S. Harper and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and Human Molecular Genetics.

In The Last Decade

Daphne Dubach

8 papers receiving 303 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daphne Dubach Australia 7 192 73 52 49 48 8 307
M Cesnjaj United States 12 175 0.9× 41 0.6× 26 0.5× 39 0.8× 65 1.4× 14 401
Jong Hak Won United States 11 221 1.2× 37 0.5× 23 0.4× 50 1.0× 34 0.7× 26 366
Martin Krenn Austria 13 135 0.7× 24 0.3× 15 0.3× 86 1.8× 31 0.6× 36 394
Takeshi Imao Japan 4 235 1.2× 43 0.6× 10 0.2× 47 1.0× 108 2.3× 5 374
Ken Matsuura Japan 8 257 1.3× 72 1.0× 11 0.2× 65 1.3× 16 0.3× 11 383
Kathleen J. Krentz United States 9 145 0.8× 20 0.3× 50 1.0× 19 0.4× 67 1.4× 13 396
Paolo Marchi United Kingdom 10 223 1.2× 57 0.8× 10 0.2× 102 2.1× 49 1.0× 15 333
Vickram Bissonauth Canada 5 171 0.9× 40 0.5× 15 0.3× 21 0.4× 17 0.4× 9 343
Valeria Y. Vasileva Russia 11 187 1.0× 52 0.7× 10 0.2× 29 0.6× 125 2.6× 32 306

Countries citing papers authored by Daphne Dubach

Since Specialization
Citations

This map shows the geographic impact of Daphne Dubach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daphne Dubach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daphne Dubach more than expected).

Fields of papers citing papers by Daphne Dubach

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daphne Dubach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daphne Dubach. The network helps show where Daphne Dubach may publish in the future.

Co-authorship network of co-authors of Daphne Dubach

This figure shows the co-authorship network connecting the top 25 collaborators of Daphne Dubach. A scholar is included among the top collaborators of Daphne Dubach based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daphne Dubach. Daphne Dubach is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Peiris, Heshan, Daphne Dubach, Claire F. Jessup, et al.. (2014). RCAN1 Regulates Mitochondrial Function and Increases Susceptibility to Oxidative Stress in Mammalian Cells. Oxidative Medicine and Cellular Longevity. 2014. 1–12. 27 indexed citations
2.
Samarajiwa, Shamith, Niamh E. Mangan, Matthew P. Hardy, et al.. (2014). Soluble IFN Receptor Potentiates In Vivo Type I IFN Signaling and Exacerbates TLR4-Mediated Septic Shock. The Journal of Immunology. 192(9). 4425–4435. 23 indexed citations
3.
Ahmed, Md. Mahiuddin, et al.. (2012). Expression of trisomic proteins in Down syndrome model systems. Gene. 512(2). 219–225. 19 indexed citations
4.
Martin, Katherine, Alicia Corlett, Daphne Dubach, et al.. (2012). Over-expression of RCAN1 causes Down syndrome-like hippocampal deficits that alter learning and memory. Human Molecular Genetics. 21(13). 3025–3041. 68 indexed citations
5.
Keating, Damien J., Daphne Dubach, Mark Zanin, et al.. (2008). DSCR1/RCAN1 regulates vesicle exocytosis and fusion pore kinetics: implications for Down syndrome and Alzheimer's disease. Human Molecular Genetics. 17(7). 1020–1030. 65 indexed citations
6.
Yu, Yong, Po‐Yin Chu, David N. Bowser, et al.. (2008). Mice deficient for the chromosome 21 ortholog Itsn1 exhibit vesicle-trafficking abnormalities. Human Molecular Genetics. 17(21). 3281–3290. 75 indexed citations
7.
Yu, Yong, Pei‐Yu Chu, David N. Bowser, et al.. (2008). Mice deficient for the chromosome 21 ortholog Itsn1 exhibit vesicle-trafficking abnormalities. Human Molecular Genetics. 18(2). 389–389. 3 indexed citations
8.
Stephenson, Sarah, et al.. (2005). A Single PDZ Domain Protein Interacts with the Menkes Copper ATPase, ATP7A. Journal of Biological Chemistry. 280(39). 33270–33279. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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