Daniel Fau

1.7k total citations
31 papers, 1.3k citations indexed

About

Daniel Fau is a scholar working on Molecular Biology, Pharmacology and Physiology. According to data from OpenAlex, Daniel Fau has authored 31 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 11 papers in Pharmacology and 10 papers in Physiology. Recurrent topics in Daniel Fau's work include Drug-Induced Hepatotoxicity and Protection (10 papers), Muscle metabolism and nutrition (8 papers) and Metabolism and Genetic Disorders (7 papers). Daniel Fau is often cited by papers focused on Drug-Induced Hepatotoxicity and Protection (10 papers), Muscle metabolism and nutrition (8 papers) and Metabolism and Genetic Disorders (7 papers). Daniel Fau collaborates with scholars based in France, United States and Russia. Daniel Fau's co-authors include Dominique Pessayre, Alain Berson, Gérard Feldmann, D. Haouzi, Bernard Fromenty, Philippe Lettéron, C Fisch, Alain Moreau, Daniel Eugène and Véronique Descatoire and has published in prestigious journals such as Gastroenterology, Hepatology and Journal of Nutrition.

In The Last Decade

Daniel Fau

31 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel Fau France 19 519 457 159 158 135 31 1.3k
Robert L. Waller United States 12 831 1.6× 331 0.7× 168 1.1× 246 1.6× 243 1.8× 25 1.4k
C.‐P. Siegers Germany 26 589 1.1× 492 1.1× 280 1.8× 128 0.8× 324 2.4× 73 2.0k
C.R. de Castro Argentina 16 488 0.9× 180 0.4× 174 1.1× 177 1.1× 114 0.8× 43 899
Yuchen Sheng China 25 604 1.2× 776 1.7× 121 0.8× 267 1.7× 206 1.5× 46 1.7k
Mohamed M. Sayed‐Ahmed Saudi Arabia 29 334 0.6× 784 1.7× 212 1.3× 193 1.2× 144 1.1× 48 2.3k
Hassan Farghali Czechia 22 378 0.7× 533 1.2× 97 0.6× 275 1.7× 95 0.7× 98 1.7k
Sang Mi Shin South Korea 23 193 0.4× 932 2.0× 127 0.8× 320 2.0× 90 0.7× 35 1.6k
A. Esteller Spain 16 172 0.3× 271 0.6× 324 2.0× 168 1.1× 106 0.8× 66 1.2k
Limei Shan China 17 235 0.5× 497 1.1× 90 0.6× 130 0.8× 121 0.9× 24 1.1k
Mahmoud Mansour Saudi Arabia 20 391 0.8× 302 0.7× 194 1.2× 98 0.6× 181 1.3× 62 1.8k

Countries citing papers authored by Daniel Fau

Since Specialization
Citations

This map shows the geographic impact of Daniel Fau's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Fau with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Fau more than expected).

Fields of papers citing papers by Daniel Fau

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Fau. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Fau. The network helps show where Daniel Fau may publish in the future.

Co-authorship network of co-authors of Daniel Fau

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel Fau. A scholar is included among the top collaborators of Daniel Fau based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel Fau. Daniel Fau is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Haouzi, D., I. Cohen, Helena L.A. Vieira, et al.. (2002). Mitochondrial permeability transition as a novel principle of hepatorenal toxicity in vivo. APOPTOSIS. 7(5). 395–405. 48 indexed citations
2.
Haouzi, D., Marina Tinel, Nathalie Vadrot, et al.. (2001). Prolonged, But Not Acute, Glutathione Depletion Promotes Fas–Mediated Mitochondrial Permeability Transition and Apoptosis in Mice. Hepatology. 33(5). 1181–1188. 57 indexed citations
3.
Berson, Alain, Daniel Fau, Angéla Sutton, et al.. (2001). Mechanisms for experimental buprenorphine hepatotoxicity: major role of mitochondrial dysfunction versus metabolic activation. Journal of Hepatology. 34(2). 261–269. 74 indexed citations
4.
Haouzi, D., Alain Moreau, Claude Moulis, et al.. (2000). Cytochrome P450-generated reactive metabolites cause mitochondrial permeability transition, caspase activation, and apoptosis in rat hepatocytes. Hepatology. 32(2). 303–311. 84 indexed citations
5.
Feldmann, Gérard, D. Haouzi, Alain Moreau, et al.. (2000). Opening of the mitochondrial permeability transition pore causes matrix expansion and outer membrane rupture in fas-mediated hepatic apoptosis in mice. Hepatology. 31(3). 674–683. 111 indexed citations
6.
Fau, Daniel, Thomas J. Farrell, A. Moreau, et al.. (1997). Diterpenoids from germander, an herbal medicine, induce apoptosis in isolated rat hepatocytes. Gastroenterology. 113(4). 1334–1346. 88 indexed citations
7.
Berson, Alain, Sylvaine Renault, P Lettéron, et al.. (1996). Uncoupling of rat and human mitochondria: A possible explanation for tacrine-induced liver dysfunction. Gastroenterology. 110(6). 1878–1890. 60 indexed citations
8.
9.
Kalopissis, Athina-Despina, et al.. (1995). Inhibition of hepatic very—low-density lipoprotein secretion in obese zucker rats adapted to a high-protein diet. Metabolism. 44(1). 19–29. 13 indexed citations
10.
Loeper, Jacqueline, Véronique Descatoire, Philippe Lettéron, et al.. (1994). Hepatotoxicity of germander in mice. Gastroenterology. 106(2). 464–472. 86 indexed citations
11.
Fau, Daniel, Daniel Eugène, Alain Berson, et al.. (1994). Toxicity of the antiandrogen flutamide in isolated rat hepatocytes.. Journal of Pharmacology and Experimental Therapeutics. 269(3). 954–962. 101 indexed citations
12.
Berson, Alain, Claude Wolf, C. Chachaty, et al.. (1992). Generation of free radicals during the reductive metabolism of nilutamide by lung microsomes: Possible role in the development of lung lesions in patients treated with this anti-androgen. Biochemical Pharmacology. 43(3). 654–657. 14 indexed citations
13.
Fau, Daniel, Alain Berson, Daniel Eugène, et al.. (1992). Mechanism for the hepatotoxicity of the antiandrogen, nilutamide. Evidence suggesting that redox cycling of this nitroaromatic drug leads to oxidative stress in isolated hepatocytes.. Journal of Pharmacology and Experimental Therapeutics. 263(1). 69–77. 70 indexed citations
14.
Delatour, P, et al.. (1991). Enantioselective N‐demethylation of ketamine in the horse. Journal of Veterinary Pharmacology and Therapeutics. 14(2). 209–212. 24 indexed citations
15.
Berson, Alain, Claude Wolf, Virginie Berger, et al.. (1991). Generation of free radicals during the reductive metabolism of the nitroaromatic compound, nilutamide.. Journal of Pharmacology and Experimental Therapeutics. 257(2). 714–719. 31 indexed citations
16.
Fau, Daniel, et al.. (1988). Effects of Ingestion of High Protein or Excess Methionine Diets by Rats for Two Years. Journal of Nutrition. 118(1). 128–133. 33 indexed citations
17.
Fau, Daniel, et al.. (1987). Effect of Excess Dietary Methionine on Weight Gain and Plasma Amino Acids in Kittens. Journal of Nutrition. 117(11). 1838–1843. 14 indexed citations
18.
Fau, Daniel, James Morris, & Quinton R. Rogers. (1987). Effects of high dietary methionine on activities of selected enzymes in the liver of kittens (felis domesticus). Comparative Biochemistry and Physiology Part B Comparative Biochemistry. 88(2). 551–555. 11 indexed citations
19.
Fau, Daniel, et al.. (1980). Long term effects of methionine excess on rat metabolism.. Nutrition reports international. 21(4). 577–585. 6 indexed citations
20.
Fau, Daniel, et al.. (1980). Methionine excess and energy requirement of the rat.. Nutrition reports international. 22(5). 751–758. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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