Daniel Eliyahu

3.5k total citations · 3 hit papers
12 papers, 3.0k citations indexed

About

Daniel Eliyahu is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Daniel Eliyahu has authored 12 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Genetics. Recurrent topics in Daniel Eliyahu's work include Virus-based gene therapy research (5 papers), Cancer Research and Treatments (4 papers) and Cancer-related Molecular Pathways (4 papers). Daniel Eliyahu is often cited by papers focused on Virus-based gene therapy research (5 papers), Cancer Research and Treatments (4 papers) and Cancer-related Molecular Pathways (4 papers). Daniel Eliyahu collaborates with scholars based in Israel, United States and Germany. Daniel Eliyahu's co-authors include Moshe Oren, Tse‐Hua Tan, Arnold J. Levine, Cathy A. Finlay, Philip W. Hinds, Dan Michalovitz, Avraham Raz, David Givol, Peter Gruß and S Eliyahu and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Molecular and Cellular Biology.

In The Last Decade

Daniel Eliyahu

12 papers receiving 2.9k citations

Hit Papers

Activating mutations for transformation by p53 produce a ... 1984 2026 1998 2012 1988 1984 1989 250 500 750 1000
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Activating mutations for transformation by p53 produce a gene product that forms an hsc70-p53 complex with an altered half-life.
    1988 1.0k citations Cathy A. Finlay, Philip W. Hinds et al. Molecular and Cellular Biology profile →
  2. Participation of p53 cellular tumour antigen in transformation of normal embryonic cells
    1984 617 citations Daniel Eliyahu, Avraham Raz et al. Nature profile →
  3. Wild-type p53 can inhibit oncogene-mediated focus formation.
    1989 608 citations Daniel Eliyahu, Dan Michalovitz et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel Eliyahu Israel 10 2.3k 1.5k 761 633 521 12 3.0k
D. Pim Italy 20 2.2k 1.0× 1.6k 1.1× 550 0.7× 1.1k 1.8× 438 0.8× 23 3.8k
Jane Weaver-Feldhaus United States 10 1.8k 0.8× 1.9k 1.2× 214 0.3× 294 0.5× 757 1.5× 10 3.1k
Dan Michalovitz Israel 9 1.7k 0.7× 1.4k 0.9× 589 0.8× 390 0.6× 358 0.7× 10 2.2k
A J Levine United States 15 2.1k 0.9× 2.2k 1.5× 595 0.8× 399 0.6× 357 0.7× 19 3.0k
Karen Buchkovich United States 11 2.2k 1.0× 1.9k 1.2× 203 0.3× 1.2k 1.8× 304 0.6× 14 3.5k
Patricia Horan Hand United States 29 1.2k 0.5× 1.6k 1.0× 168 0.2× 369 0.6× 254 0.5× 52 3.3k
Lora Hedrick United States 24 1.9k 0.8× 2.0k 1.4× 196 0.3× 410 0.6× 771 1.5× 30 4.1k
Berthold Henglein France 22 1.7k 0.8× 2.1k 1.4× 130 0.2× 443 0.7× 321 0.6× 27 3.2k
Manijeh Daneshmand Canada 18 2.3k 1.0× 1.2k 0.8× 294 0.4× 1.1k 1.8× 474 0.9× 34 3.6k
Peter Scully United States 7 1.4k 0.6× 1.1k 0.8× 116 0.2× 443 0.7× 266 0.5× 8 2.0k

Countries citing papers authored by Daniel Eliyahu

Since Specialization
Citations

This map shows the geographic impact of Daniel Eliyahu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Eliyahu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Eliyahu more than expected).

Fields of papers citing papers by Daniel Eliyahu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Eliyahu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Eliyahu. The network helps show where Daniel Eliyahu may publish in the future.

Co-authorship network of co-authors of Daniel Eliyahu

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel Eliyahu. A scholar is included among the top collaborators of Daniel Eliyahu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel Eliyahu. Daniel Eliyahu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Eliyahu, Daniel, Stephen O. Evans, Neal Rosen, et al.. (1994). p53Val135 temperature sensitive mutant suppresses growth of human breast cancer cells. Breast Cancer Research and Treatment. 30(2). 167–177. 5 indexed citations
2.
Eliyahu, Daniel, et al.. (1989). Wild-type p53 can inhibit oncogene-mediated focus formation.. Proceedings of the National Academy of Sciences. 86(22). 8763–8767. 608 indexed citations breakdown →
3.
Finlay, Cathy A., Philip W. Hinds, Tse‐Hua Tan, et al.. (1988). Activating Mutations for Transformation by p53 Produce a Gene Product That Forms an hsc70-p53 Complex with an Altered Half-Life. Molecular and Cellular Biology. 8(2). 531–539. 283 indexed citations
4.
Finlay, Cathy A., Philip W. Hinds, Tse‐Hua Tan, et al.. (1988). Activating mutations for transformation by p53 produce a gene product that forms an hsc70-p53 complex with an altered half-life.. Molecular and Cellular Biology. 8(2). 531–539. 1015 indexed citations breakdown →
5.
Eliyahu, Daniel, Naomi Goldfinger, Gad Shaulsky, et al.. (1988). Meth A fibrosarcoma cells express two transforming mutant p53 species.. PubMed. 3(3). 313–21. 189 indexed citations
6.
Michalovitz, Dan, Daniel Eliyahu, & Moshe Oren. (1986). Overproduction of Protein p53 Contributes to Simian Virus 40-Mediated Transformation. Molecular and Cellular Biology. 6(10). 3531–3536. 20 indexed citations
7.
Michalovitz, Dan, Daniel Eliyahu, & Moshe Oren. (1986). Overproduction of protein p53 contributes to simian virus 40-mediated transformation.. Molecular and Cellular Biology. 6(10). 3531–3536. 42 indexed citations
8.
Eliyahu, Daniel, Dan Michalovitz, & Moshe Oren. (1985). Overproduction of p53 antigen makes established cells highly tumorigenic. Nature. 316(6024). 158–160. 171 indexed citations
9.
Eliyahu, Daniel, Avraham Raz, Peter Gruß, David Givol, & Moshe Oren. (1984). Participation of p53 cellular tumour antigen in transformation of normal embryonic cells. Nature. 312(5995). 646–649. 617 indexed citations breakdown →
10.
Soreq, Hermona, Anat Safran, & Daniel Eliyahu. (1983). Modified composition of major ontogenetically regulated mRNAs and proteins in the cerebellum of old and of staggerer mice. Developmental Brain Research. 10(1). 73–82. 8 indexed citations
11.
Soreq, Hermona, David Gurwitz, Daniel Eliyahu, & Mordechai Sokolovsky. (1982). Altered Ontogenesis of Muscarinic Receptors in Agranular Cerebellar Cortex. Journal of Neurochemistry. 39(3). 756–763. 15 indexed citations
12.
Eliyahu, Daniel & Hermona Soreq. (1982). Degranulation of Rat Cerebellum Induces Selective Variations in Gene Expression. Journal of Neurochemistry. 38(2). 313–321. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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