Daniel D. Brown

710 total citations
22 papers, 498 citations indexed

About

Daniel D. Brown is a scholar working on Molecular Biology, Genetics and Pollution. According to data from OpenAlex, Daniel D. Brown has authored 22 papers receiving a total of 498 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 5 papers in Genetics and 3 papers in Pollution. Recurrent topics in Daniel D. Brown's work include Congenital heart defects research (4 papers), Estrogen and related hormone effects (3 papers) and Selenium in Biological Systems (2 papers). Daniel D. Brown is often cited by papers focused on Congenital heart defects research (4 papers), Estrogen and related hormone effects (3 papers) and Selenium in Biological Systems (2 papers). Daniel D. Brown collaborates with scholars based in United States, United Kingdom and China. Daniel D. Brown's co-authors include Frank L. Conlon, Sarah C. Goetz, James C. Smith, Kathleen S. Christine, Yuriy Fedorov, Robert L. Metcalf, Loren Hansen, Chad M. Thompson, Laurie C. Haws and Mina Suh and has published in prestigious journals such as PLoS ONE, Analytical Chemistry and Development.

In The Last Decade

Daniel D. Brown

21 papers receiving 485 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel D. Brown United States 12 321 97 65 52 52 22 498
Vinesh Vinayachandran United States 16 856 2.7× 92 0.9× 32 0.5× 34 0.7× 41 0.8× 24 1.1k
Jingjing Cao China 8 494 1.5× 46 0.5× 33 0.5× 20 0.4× 101 1.9× 10 592
Wenrong Xia China 10 162 0.5× 100 1.0× 42 0.6× 17 0.3× 26 0.5× 20 364
Weichao Zhao China 12 136 0.4× 39 0.4× 20 0.3× 42 0.8× 65 1.3× 41 463
Gabriele E. Ackermann Switzerland 11 218 0.7× 220 2.3× 19 0.3× 37 0.7× 46 0.9× 16 575
Chuntao Li China 14 159 0.5× 40 0.4× 21 0.3× 16 0.3× 72 1.4× 46 500
James B. Hughes United States 13 232 0.7× 81 0.8× 36 0.6× 9 0.2× 41 0.8× 30 601
Anastassiia Vertii United States 12 325 1.0× 21 0.2× 31 0.5× 20 0.4× 34 0.7× 19 601
Bum-Ho Bin Japan 10 172 0.5× 233 2.4× 31 0.5× 24 0.5× 28 0.5× 11 642

Countries citing papers authored by Daniel D. Brown

Since Specialization
Citations

This map shows the geographic impact of Daniel D. Brown's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel D. Brown with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel D. Brown more than expected).

Fields of papers citing papers by Daniel D. Brown

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel D. Brown. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel D. Brown. The network helps show where Daniel D. Brown may publish in the future.

Co-authorship network of co-authors of Daniel D. Brown

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel D. Brown. A scholar is included among the top collaborators of Daniel D. Brown based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel D. Brown. Daniel D. Brown is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Li‐Ju, Yuan Zhang, Daniel D. Brown, et al.. (2025). High-Throughput Empirical and Virtual Screening To Discover Novel Inhibitors of Polyploid Giant Cancer Cells in Breast Cancer. Analytical Chemistry. 97(10). 5498–5506. 3 indexed citations
2.
Narayanan, Susrutha Puthanmadhom, et al.. (2024). Transcriptomic analysis identifies enrichment of cAMP/PKA/CREB signaling in invasive lobular breast cancer. Breast Cancer Research. 26(1). 149–149. 2 indexed citations
3.
Li, Zheqi, Daniel D. Brown, Soleilmane Omarjee, et al.. (2023). FOXA1 Reprogramming Dictates Retinoid X Receptor Response in ESR1 -Mutant Breast Cancer. Molecular Cancer Research. 21(6). 591–604. 9 indexed citations
4.
Li, Zheqi, Daniel D. Brown, Jason S. Carroll, et al.. (2023). The EstroGene Database Reveals Diverse Temporal, Context-Dependent, and Bidirectional Estrogen Receptor Regulomes in Breast Cancer. Cancer Research. 83(16). 2656–2674. 7 indexed citations
5.
Hooda, Jagmohan, Jian Chen, Daniel D. Brown, et al.. (2022). Loss of E-cadherin Induces IGF1R Activation and Reveals a Targetable Pathway in Invasive Lobular Breast Carcinoma. Molecular Cancer Research. 20(9). 1405–1419. 11 indexed citations
6.
Brown, Daniel D., David J. Dabbs, Adrian V. Lee, et al.. (2015). Developing in vitro models of human ductal carcinoma in situ from primary tissue explants. Breast Cancer Research and Treatment. 153(2). 311–321. 18 indexed citations
7.
Dabbs, David J., Peter C. Lucas, Rachel C. Jankowitz, et al.. (2015). Molecular drivers of lobular carcinoma in situ. Breast Cancer Research. 17(1). 76–76. 24 indexed citations
8.
Thompson, Chad M., Yuriy Fedorov, Daniel D. Brown, et al.. (2012). Assessment of Cr(VI)-Induced Cytotoxicity and Genotoxicity Using High Content Analysis. PLoS ONE. 7(8). e42720–e42720. 60 indexed citations
9.
10.
Ellis, J. Christopher, Daniel D. Brown, & James W. Brown. (2010). The small nucleolar ribonucleoprotein (snoRNP) database. RNA. 16(4). 664–666. 16 indexed citations
11.
Brown, Daniel D., et al.. (2007). Small heat shock protein Hsp27 is required for proper heart tube formation. genesis. 45(11). 667–678. 50 indexed citations
12.
Sarang, Satinder S., et al.. (2007). Identification, Coassembly, and Activity of γ-Aminobutyric Acid Receptor Subunits in Renal Proximal Tubular Cells. Journal of Pharmacology and Experimental Therapeutics. 324(1). 376–382. 9 indexed citations
13.
Brown, Daniel D., A. Davis, & Frank L. Conlon. (2006). Xtn3 is a developmentally expressed cardiac and skeletal muscle-specific novex-3 titin isoform. Gene Expression Patterns. 6(8). 913–918. 4 indexed citations
14.
Goetz, Sarah C., Daniel D. Brown, & Frank L. Conlon. (2006). TBX5 is required for embryonic cardiac cell cycle progression. Development. 133(13). 2575–2584. 57 indexed citations
15.
Brown, Daniel D., et al.. (2005). Tbx5 and Tbx20 act synergistically to control vertebrate heart morphogenesis. Development. 132(3). 553–563. 110 indexed citations
16.
Brown, Daniel D., et al.. (2003). Developmental expression of the Xenopus laevis Tbx20 orthologue. Development Genes and Evolution. 212(12). 604–607. 36 indexed citations
17.
18.
Metcalf, Robert L., et al.. (1980). Model ecosystem toxicity and uptake evaluation of selenium 75 selenite. Bulletin of Environmental Contamination and Toxicology. 24(5). 752–758. 1 indexed citations
19.
Coats, Joel R., et al.. (1976). Model ecosystem evaluation of the environmental impacts of the veterinary drugs phenothiazine, sulfamethazine, clopidol, and diethylstilbestrol.. Environmental Health Perspectives. 18. 167–179. 18 indexed citations
20.
Coats, Joel R., Robert L. Metcalf, Po‐Yung Lu, et al.. (1976). Model Ecosystem Evaluation of the Environmental Impacts of the Veterinary Drugs Phenothiazine, Sulfamethazine, Clopidol, and Diethylstilbestrol. Environmental Health Perspectives. 18. 167–167. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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