Dalila Ali-Hadji

535 citations
8 papers · 152 indexed · h-index 7
Topics
Virus-based gene therapy research (5 papers)Animal Genetics and Reproduction (4 papers)CRISPR and Genetic Engineering (2 papers)
Partner nations
France

In The Last Decade

Dalila Ali-Hadji

8 papers receiving 148 citations

Peers

Dalila Ali-Hadji
Comparison fields: 5 of 47
  • Molecular Biology 112
  • Genetics 103
  • Surgery 16
  • Hepatology 13
  • Oncology 11
Replace Irene Talón with:
Irene Talón Belgium
Leonor Rib Denmark
Rhonda Trousdale United States
J H Cho United States
Dawei Yu China
Damien Ulveling France
Patricia Stiedl Austria
Kaixiang Xu China
Yumiko Iba Japan
Chaiyaboot Ariyachet Thailand
Dalila Ali-Hadji relative to Irene Talón Belgium Irene Talón's profile →
Citations per field
00.5×1.6×
Irene Talón · 1×
Citations per year

Countries citing papers authored by Dalila Ali-Hadji

Since Specialization
Citations

This map shows the geographic impact of Dalila Ali-Hadji's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dalila Ali-Hadji with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dalila Ali-Hadji more than expected).

Fields of papers citing papers by Dalila Ali-Hadji

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dalila Ali-Hadji. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dalila Ali-Hadji. The network helps show where Dalila Ali-Hadji may publish in the future.

Co-authorship network of co-authors of Dalila Ali-Hadji

This figure shows the co-authorship network connecting the top 25 collaborators of Dalila Ali-Hadji. A scholar is included among the top collaborators of Dalila Ali-Hadji based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dalila Ali-Hadji. Dalila Ali-Hadji is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
#WorkIndexed citations
1 44
2 23
3 7
4 1
5 43
6
The promoter of the human cystic fibrosis transmembrane conductance regulator gene directing SV40 T antigen expression induces malignant proliferation of ependymal cells in transgenic mice.
10
7 18
8 6

About Dalila Ali-Hadji

Dalila Ali-Hadji is a scholar working on Genetics, Virology and Hepatology, having authored 8 papers that have together received 152 indexed citations. Recurring topics across this work include Virus-based gene therapy research (5 papers), Animal Genetics and Reproduction (4 papers) and CRISPR and Genetic Engineering (2 papers). The work is most often cited by research in Genetics (103 citations), Hepatology (13 citations) and Molecular Biology (112 citations). Dalila Ali-Hadji has collaborated with scholars based in France. Frequent co-authors include Andréa Pavirani, Wilfried Dalemans, Frédéric Perraud, F. Perraud, Serge Braun, Christine Thioudellet, Rainer Bischoff, Louis‐Marie Houdebine, Eric Degryse and Dominique Thépot. Their work appears in journals such as FEBS Letters, Experimental Cell Research and Gene Therapy.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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