Dagmar Nolte

1.5k total citations
35 papers, 1.0k citations indexed

About

Dagmar Nolte is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Dagmar Nolte has authored 35 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 21 papers in Cellular and Molecular Neuroscience and 8 papers in Neurology. Recurrent topics in Dagmar Nolte's work include Genetic Neurodegenerative Diseases (17 papers), Mitochondrial Function and Pathology (11 papers) and Neurological disorders and treatments (5 papers). Dagmar Nolte is often cited by papers focused on Genetic Neurodegenerative Diseases (17 papers), Mitochondrial Function and Pathology (11 papers) and Neurological disorders and treatments (5 papers). Dagmar Nolte collaborates with scholars based in Germany, United States and France. Dagmar Nolte's co-authors include Ulrich Müller, Stephan Niemann, Virgilio Gerald H. Evidente, Ulrich Müller, Michael F. Waters, Dominic B. Fee, Karla P. Figueroa, Stefan M. Pulst, Alexis Brice and Natali A. Minassian and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Genetics and Neurology.

In The Last Decade

Dagmar Nolte

35 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Dagmar Nolte Germany 19 634 619 408 115 90 35 1.0k
Jesse R. McLean Canada 16 346 0.5× 394 0.6× 602 1.5× 80 0.7× 138 1.5× 23 1.1k
Julia M. Schulze‐Hentrich Germany 21 237 0.4× 1.1k 1.8× 271 0.7× 92 0.8× 46 0.5× 46 1.5k
Philippe Sindou France 20 582 0.9× 308 0.5× 443 1.1× 16 0.1× 153 1.7× 34 1.0k
Karen Cleverley United Kingdom 8 302 0.5× 555 0.9× 408 1.0× 126 1.1× 53 0.6× 16 917
Anna Cartier United States 10 289 0.5× 318 0.5× 304 0.7× 96 0.8× 80 0.9× 13 868
Marie E. Jönsson Sweden 20 386 0.6× 919 1.5× 82 0.2× 161 1.4× 45 0.5× 27 1.3k
Christo Shalish United States 11 837 1.3× 258 0.4× 865 2.1× 89 0.8× 84 0.9× 13 1.2k
Jong‐Min Lee United States 21 1.1k 1.7× 1.2k 2.0× 379 0.9× 124 1.1× 32 0.4× 42 1.5k
Daniel Perrelet Switzerland 9 262 0.4× 357 0.6× 99 0.2× 27 0.2× 43 0.5× 9 620
Takuya Tamura Japan 17 360 0.6× 544 0.9× 102 0.3× 80 0.7× 97 1.1× 55 912

Countries citing papers authored by Dagmar Nolte

Since Specialization
Citations

This map shows the geographic impact of Dagmar Nolte's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dagmar Nolte with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dagmar Nolte more than expected).

Fields of papers citing papers by Dagmar Nolte

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dagmar Nolte. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dagmar Nolte. The network helps show where Dagmar Nolte may publish in the future.

Co-authorship network of co-authors of Dagmar Nolte

This figure shows the co-authorship network connecting the top 25 collaborators of Dagmar Nolte. A scholar is included among the top collaborators of Dagmar Nolte based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dagmar Nolte. Dagmar Nolte is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kang, Jun‐Suk, et al.. (2024). A novel KCND3 variant in the N‐terminus impairs the ionic current of Kv4.3 and is associated with SCA19/22. Journal of Cellular and Molecular Medicine. 28(16). e70039–e70039. 1 indexed citations
2.
Ackl, Nibal, et al.. (2022). A Severe Dementia Syndrome Caused by Intron Retention and Cryptic Splice Site Activation in STUB1 and Exacerbated by TBP Repeat Expansions. Frontiers in Molecular Neuroscience. 15. 878236–878236. 8 indexed citations
3.
Nolte, Dagmar, et al.. (2021). Mutations in MT-ATP6 are a frequent cause of adult-onset spinocerebellar ataxia. Journal of Neurology. 268(12). 4866–4873. 11 indexed citations
4.
Mazza, Davide, Franca Codazzi, Tyler Mark Pierson, et al.. (2019). Pathogenic variants in the AFG3L2 proteolytic domain cause SCA28 through haploinsufficiency and proteostatic stress-driven OMA1 activation. Journal of Medical Genetics. 56(8). 499–511. 19 indexed citations
5.
Kang, J.-S., Johannes Klein, Simon Baudrexel, et al.. (2013). White matter damage is related to ataxia severity in SCA3. Journal of Neurology. 261(2). 291–299. 50 indexed citations
6.
Kang, Jun‐Suk, et al.. (2013). A Novel Missense Mutation in AFG3L2 Associated with Late Onset and Slow Progression of Spinocerebellar Ataxia Type 28. Journal of Molecular Neuroscience. 52(4). 493–496. 18 indexed citations
7.
8.
Nolte, Dagmar, et al.. (2010). Spinocerebellar ataxia type 17 associated with an expansion of 42 glutamine residues in TATA-box binding protein gene. Journal of Neurology Neurosurgery & Psychiatry. 81(12). 1396–1399. 30 indexed citations
9.
Freund, Hans‐Joachim, Utako Birgit Barnikol, Dagmar Nolte, et al.. (2007). Subthalamic‐thalamic DBS in a case with spinocerebellar ataxia type 2 and severe tremor—A unusual clinical benefit. Movement Disorders. 22(5). 732–735. 53 indexed citations
10.
Evidente, Virgilio Gerald H., Mark Lyons, Froukje M. Beynen, et al.. (2007). First case of X‐linked dystonia‐parkinsonism (“Lubag”) to demonstrate a response to bilateral pallidal stimulation. Movement Disorders. 22(12). 1790–1793. 58 indexed citations
11.
Nolte, Dagmar, et al.. (2007). Structural and functional analysis of the human TAF1/DYT3 multiple transcript system. Mammalian Genome. 18(11). 787–795. 16 indexed citations
12.
Nolte, Dagmar, et al.. (2006). Spinocerebellar ataxia 14: Novel mutation in exon 2 of PRKCG in a German family. Movement Disorders. 22(2). 265–267. 14 indexed citations
13.
Voss, Ursula, Kirn R. Kessler, Udo Rüb, et al.. (2006). Stages of sleep pathology in spinocerebellar ataxia type 2 (SCA2). Neurology. 67(11). 1966–1972. 56 indexed citations
14.
Waters, Michael F., Natali A. Minassian, Giovanni Stévanin, et al.. (2006). Mutations in voltage-gated potassium channel KCNC3 cause degenerative and developmental central nervous system phenotypes. Nature Genetics. 38(4). 447–451. 220 indexed citations
15.
Waters, Michael F., Dominic B. Fee, Karla P. Figueroa, et al.. (2005). An autosomal dominant ataxia maps to 19q13: Allelic heterogeneity of SCA13 or novel locus?. Neurology. 65(7). 1111–1113. 28 indexed citations
16.
Nolte, Dagmar & Ulrich Müller. (2002). Human O-GlcNAc transferase (OGT): genomic structure, analysis of splice variants, fine mapping in Xq13.1. Mammalian Genome. 13(1). 62–64. 64 indexed citations
17.
Laccone, Franco, Ulrike Engel, Elke Holinski‐Feder, et al.. (1999). DNA analysis of Huntington's disease: five year experience in Germany, Austria and Switzerland (1993-1997). University of Regensburg Publication Server (University of Regensburg). 1 indexed citations
18.
Haberhausen, Gerd, et al.. (1997). AFX1 and p54 nrb : fine mapping, genomic structure, and exclusion as candidate genes of X-linked dystonia parkinsonism. Human Genetics. 100(5-6). 569–572. 18 indexed citations
19.
Rudolph, Bettina, Dagmar Nolte, & Bernhard Knapp. (1994). Isolation of a third member of the Plasmodium falciparum glycophorin-binding protein gene family. Molecular and Biochemical Parasitology. 68(1). 173–176. 2 indexed citations
20.
Nolte, Dagmar & Bernhard Knapp. (1991). Partial sequences of three new asparagine-rich blood stage proteins of Plasmodium falciparum. Molecular and Biochemical Parasitology. 46(2). 319–321. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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