D Kim

728 total citations
10 papers, 589 citations indexed

About

D Kim is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, D Kim has authored 10 papers receiving a total of 589 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Cardiology and Cardiovascular Medicine and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in D Kim's work include Ion channel regulation and function (9 papers), Cardiac electrophysiology and arrhythmias (8 papers) and Neuroscience and Neuropharmacology Research (5 papers). D Kim is often cited by papers focused on Ion channel regulation and function (9 papers), Cardiac electrophysiology and arrhythmias (8 papers) and Neuroscience and Neuropharmacology Research (5 papers). D Kim collaborates with scholars based in United States, Somalia and Bulgaria. D Kim's co-authors include C. D. Sladek, Joanne R. Mathiasen, Carmen Aguado‐Velasco, Michael J. Ackerman, David E. Clapham and Mark Wallert and has published in prestigious journals such as Circulation Research, The Journal of Physiology and The Journal of General Physiology.

In The Last Decade

D Kim

10 papers receiving 577 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
D Kim United States 10 477 371 245 81 59 10 589
Jérôme Petit-Jacques United States 10 540 1.1× 267 0.7× 354 1.4× 35 0.4× 27 0.5× 11 628
Toshihiko Kaku Japan 12 660 1.4× 603 1.6× 178 0.7× 63 0.8× 53 0.9× 22 841
Heinz G�gelein Germany 12 513 1.1× 201 0.5× 264 1.1× 36 0.4× 54 0.9× 13 608
I. Imanaga Japan 16 445 0.9× 215 0.6× 192 0.8× 55 0.7× 28 0.5× 32 591
A. Schwartz United States 9 522 1.1× 226 0.6× 331 1.4× 51 0.6× 20 0.3× 12 589
R P Xiao United States 5 630 1.3× 553 1.5× 219 0.9× 94 1.2× 55 0.9× 7 805
Allan J. Levi United Kingdom 15 616 1.3× 660 1.8× 319 1.3× 25 0.3× 35 0.6× 29 766
Withrow Gil Wier United States 12 363 0.8× 315 0.8× 169 0.7× 125 1.5× 18 0.3× 20 527
Valérie Leuranguer France 12 477 1.0× 272 0.7× 284 1.2× 75 0.9× 13 0.2× 14 548
Rie Schultz Hansen Denmark 16 895 1.9× 872 2.4× 296 1.2× 102 1.3× 83 1.4× 23 1.2k

Countries citing papers authored by D Kim

Since Specialization
Citations

This map shows the geographic impact of D Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D Kim more than expected).

Fields of papers citing papers by D Kim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D Kim. The network helps show where D Kim may publish in the future.

Co-authorship network of co-authors of D Kim

This figure shows the co-authorship network connecting the top 25 collaborators of D Kim. A scholar is included among the top collaborators of D Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D Kim. D Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Kim, D, C. D. Sladek, Carmen Aguado‐Velasco, & Joanne R. Mathiasen. (1995). Arachidonic acid activation of a new family of K+ channels in cultured rat neuronal cells.. The Journal of Physiology. 484(3). 643–660. 81 indexed citations
2.
Kim, D, et al.. (1995). Membrane stretch augments the cardiac muscarinic K+ channel activity. The Journal of Membrane Biology. 148(3). 287–97. 24 indexed citations
3.
Kim, D. (1993). Mechanism of rapid desensitization of muscarinic K+ current in adult rat and guinea pig atrial cells.. Circulation Research. 73(1). 89–97. 33 indexed citations
4.
Kim, D. (1993). Novel cation-selective mechanosensitive ion channel in the atrial cell membrane.. Circulation Research. 72(1). 225–231. 113 indexed citations
5.
Kim, D. (1992). A mechanosensitive K+ channel in heart cells. Activation by arachidonic acid.. The Journal of General Physiology. 100(6). 1021–1040. 105 indexed citations
6.
Wallert, Mark, Michael J. Ackerman, D Kim, & David E. Clapham. (1991). Two novel cardiac atrial K+ channels, IK.AA and IK.PC.. The Journal of General Physiology. 98(5). 921–939. 24 indexed citations
7.
Kim, D. (1991). Modulation of acetylcholine‐activated K+ channel function in rat atrial cells by phosphorylation.. The Journal of Physiology. 437(1). 133–155. 56 indexed citations
8.
Kim, D. (1991). Endothelin activation of an inwardly rectifying K+ current in atrial cells.. Circulation Research. 69(1). 250–255. 37 indexed citations
9.
Kim, D. (1990). Beta-adrenergic regulation of the muscarinic-gated K+ channel via cyclic AMP-dependent protein kinase in atrial cells.. Circulation Research. 67(5). 1292–1298. 23 indexed citations
10.
Kim, D, et al.. (1990). Regulation of K+ channels in cardiac myocytes by free fatty acids.. Circulation Research. 67(4). 1040–1046. 93 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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