D. De Sabata

449 total citations
9 papers, 95 citations indexed

About

D. De Sabata is a scholar working on Immunology, Genetics and Hematology. According to data from OpenAlex, D. De Sabata has authored 9 papers receiving a total of 95 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Immunology, 4 papers in Genetics and 4 papers in Hematology. Recurrent topics in D. De Sabata's work include Chronic Lymphocytic Leukemia Research (3 papers), Immune Cell Function and Interaction (3 papers) and Lymphoma Diagnosis and Treatment (3 papers). D. De Sabata is often cited by papers focused on Chronic Lymphocytic Leukemia Research (3 papers), Immune Cell Function and Interaction (3 papers) and Lymphoma Diagnosis and Treatment (3 papers). D. De Sabata collaborates with scholars based in Italy, France and Germany. D. De Sabata's co-authors include G. Perona, Fabio Benedetti, Gianpaolo Nadali, L. Morosato, Fabrizio Vinante, Gianpietro Semenzato, Dino Veneri, Achille Ambrosetti, Giovanni Pizzolo and Giuseppe Todeschini and has published in prestigious journals such as Cancer, Stem Cells and Leukemia & lymphoma.

In The Last Decade

D. De Sabata

9 papers receiving 94 citations

Peers

D. De Sabata

IMMUN

PFM

ONCOL

HEMAT

GENET

Marja J. Pongers-Willemse Netherlands

Marie V. Coignet United States

Moulod R. El-Agnaf United Kingdom

Bella Biderman Russia

Matteo Leoncin Italy

Jehane Fadlallah France

Naïs Prade France

Mika Kohri Japan

Emily C. Ayers United States

Audrey M. Sigmund United States

Marja J. Pongers-Willemse Netherlands

D. De Sabata

36 ×0.7

IMMUN

46 ×1.1

PFM

20 ×0.7

ONCOL

61 ×2.0

HEMAT

27 ×1.4

GENET

Citations per year, relative to D. De Sabata D. De Sabata (= 1×) peers Marja J. Pongers-Willemse

Countries citing papers authored by D. De Sabata

Since Specialization

Citations

This map shows the geographic impact of D. De Sabata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. De Sabata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. De Sabata more than expected).

Fields of papers citing papers by D. De Sabata

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D. De Sabata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. De Sabata. The network helps show where D. De Sabata may publish in the future.

Co-authorship network of co-authors of D. De Sabata

This figure shows the co-authorship network connecting the top 25 collaborators of D. De Sabata. A scholar is included among the top collaborators of D. De Sabata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. De Sabata. D. De Sabata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Tecchio, Cristina, A Andreini, Claudio Costantini, et al.. (2018). Haploidentical hematopoietic stem cell transplantation in a myelofibrosis patient with primary graft failure. Hematology Reports. 9(4). 7091–7091. 1 indexed citations

2.

Tecchio, Cristina, Federico Mosna, A Andreini, et al.. (2012). The National Institutes of Health criteria for classification and scoring of chronic graft versus host disease: long-term follow-up of a single center series. Leukemia & lymphoma. 54(5). 1020–1027. 2 indexed citations

3.

Veneri, Dino, Massimo Franchini, D. De Sabata, et al.. (2008). Peripheral blood CD5-positive B lymphocytes (B-1a cells) after allogeneic stem cell transplantation for acute myeloid leukaemia in humans.. PubMed. 6(4). 220–4. 6 indexed citations

4.

Vinante, Fabrizio, L. Morosato, Gianpaolo Nadali, et al.. (1995). Soluble forms of p55-IL-2R alpha, CD8, and CD30 molecules as markers of lymphoid cell activation in infectious mononucleosis.. PubMed. 79(5). 413–9. 13 indexed citations

5.

Benedetti, Fabio, D. De Sabata, & G. Perona. (1994). T suppressor activated lymphocytes (CD8⁺/DR⁺) inhibit megakaryocyte progenitor cell differentiation in a case of acquired amegakaryocytic thrombocytopenic purpura. Stem Cells. 12(2). 205–213. 22 indexed citations

6.

Ambrosetti, Achille, Gianpaolo Nadali, Fabrizio Vinante, et al.. (1993). Soluble interleukin-2 receptor in hairy-cell leukemia: a reliable marker of disease. International Journal of Clinical & Laboratory Research. 23(1-4). 34–37. 12 indexed citations

7.

Ambrosetti, Achille, Gianpaolo Nadali, Fabrizio Vinante, et al.. (1993). Serum levels of soluble interleukin-2 receptor in Hodgkin disease. Relationship with clinical stage, tumor burden, and treatment outcome. Cancer. 72(1). 201–206. 34 indexed citations

8.

Pizzolo, Giovanni, Friederike Dallenbach, Fabrizio Vinante, et al.. (1992). High Serum Levels of Soluble Interleukin-2 Receptor and Absence of Detectable Levels of Soluble CD30 Molecule: A Specific Diagnostic Combination for Hairy Cell Leukemia. Leukemia & lymphoma. 6(4-5). 385–388. 3 indexed citations

9.

Vinante, Fabrizio, L. Morosato, D. De Sabata, & Giovanni Pizzolo. (1991). Soluble molecules in lymphoproliferative disorders.. PubMed. 5 Suppl 1. 18–21. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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