Cornelia Scharler
Impact in
-
- Mesenchymal stem cell research
-
- Tendon Structure and Treatment
Papers in
- Genetics 4
- Mesenchymal stem cell research 3
-
- Peroxisome Proliferator-Activated Receptors 1
- Co-authors
- Dirk Strunk (6 shared papers)Lara Bieler (2 shared papers)Sébastien Couillard‐Després (2 shared papers)Elisabeth Russe (2 shared papers)Sarah Hochmann (2 shared papers)Katharina Schallmoser (3 shared papers)Heike Mrowetz (1 shared paper)Mario Gimona (1 shared paper)
- Journals
- Theranostics (2 papers)Scientific Reports (2 papers)Brain Pathology (1 paper)Molecular Neurobiology (1 paper)Frontiers in Neurology (1 paper)
- Partner nations
- AustriaUnited StatesJapan
In The Last Decade
Cornelia Scharler
10 papers receiving 298 citations
Peers
Comparison fields: 5 of 61
- Genetics 61
- Orthopedics and Sports Medicine 31
- Pathology and Forensic Medicine 55
- Rehabilitation 18
- Neurology 22
Countries citing papers authored by Cornelia Scharler
This map shows the geographic impact of Cornelia Scharler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cornelia Scharler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cornelia Scharler more than expected).
Fields of papers citing papers by Cornelia Scharler
This network shows the impact of papers produced by Cornelia Scharler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cornelia Scharler. The network helps show where Cornelia Scharler may publish in the future.
Co-authors
The 25 scholars most cited alongside Cornelia Scharler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 79 | |
| 2 | 2021 | 62 | |
| 3 | 2018 | 43 | |
| 4 | 2016 | 41 | |
| 5 | 2018 | 28 | |
| 6 | 2019 | 15 | |
| 7 | 2019 | 14 | |
| 8 | 2020 | 9 | |
| 9 | 2022 | 8 | |
| 10 | 2022 | 3 |
About Cornelia Scharler
Cornelia Scharler is a scholar working on Genetics, Molecular Biology, Neurology, Pathology and Forensic Medicine and Rehabilitation, having authored 10 papers that have together received 302 indexed citations. Recurring topics across this work include Mesenchymal stem cell research (3 papers), Wound Healing and Treatments (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Peroxisome Proliferator-Activated Receptors (1 paper), Skin and Cellular Biology Research (1 paper), Cancer, Hypoxia, and Metabolism (1 paper), Silk-based biomaterials and applications (1 paper) and Neurogenesis and neuroplasticity mechanisms (1 paper). The work is most often cited by research in Genetics (61 citations), Orthopedics and Sports Medicine (31 citations), Pathology and Forensic Medicine (55 citations), Rehabilitation (18 citations) and Neurology (22 citations). Cornelia Scharler has collaborated with scholars based in Austria, United States and Japan. Frequent co-authors include Dirk Strunk, Lara Bieler, Sébastien Couillard‐Després, Elisabeth Russe, Sarah Hochmann, Katharina Schallmoser, Heike Mrowetz, Mario Gimona, Francisco J. Rivera and Eva Rohde. Their work appears in journals such as Theranostics, Scientific Reports, Brain Pathology, Molecular Neurobiology and Frontiers in Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.