Conghua Lu

739 total citations
39 papers, 528 citations indexed

About

Conghua Lu is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Oncology. According to data from OpenAlex, Conghua Lu has authored 39 papers receiving a total of 528 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Pulmonary and Respiratory Medicine, 22 papers in Molecular Biology and 15 papers in Oncology. Recurrent topics in Conghua Lu's work include Lung Cancer Treatments and Mutations (22 papers), PI3K/AKT/mTOR signaling in cancer (11 papers) and Cancer Mechanisms and Therapy (6 papers). Conghua Lu is often cited by papers focused on Lung Cancer Treatments and Mutations (22 papers), PI3K/AKT/mTOR signaling in cancer (11 papers) and Cancer Mechanisms and Therapy (6 papers). Conghua Lu collaborates with scholars based in China, United States and Australia. Conghua Lu's co-authors include Yong He, Caiyu Lin, Yubo Wang, Rui Han, Li Li, Hengyi Chen, Kejun Zhang, Chen Hu, Mingxia Feng and Fenfen Sun and has published in prestigious journals such as SHILAP Revista de lepidopterología, Developmental Cell and Carcinogenesis.

In The Last Decade

Conghua Lu

37 papers receiving 525 citations

Peers

Conghua Lu
Miaolin Zhu China
Shumin Ouyang China
Changqie Pan China
Gaël S. Roth France
Qing-Rong Chen United States
EunGi Kim South Korea
Caterina Aversa Italy
G. Hu China
Hongjing Zang China
Miaolin Zhu China
Conghua Lu
Citations per year, relative to Conghua Lu Conghua Lu (= 1×) peers Miaolin Zhu

Countries citing papers authored by Conghua Lu

Since Specialization
Citations

This map shows the geographic impact of Conghua Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Conghua Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Conghua Lu more than expected).

Fields of papers citing papers by Conghua Lu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Conghua Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Conghua Lu. The network helps show where Conghua Lu may publish in the future.

Co-authorship network of co-authors of Conghua Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Conghua Lu. A scholar is included among the top collaborators of Conghua Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Conghua Lu. Conghua Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Qin, Jilong, Xiaohan Zhang, Linyu Fan, et al.. (2025). Facile fabrication and characterization of double network starch/PVA/NaCl composite hydrogel for flexible strain sensor. Reactive and Functional Polymers. 208. 106163–106163. 5 indexed citations
2.
Liu, Zhenhua, Huihui Zhou, Siwei Zhang, et al.. (2025). Characteristic components from Rehmannia radix and their effects on insulin resistance through PI-3K/AKT signaling pathway in HepG2 Cells. SHILAP Revista de lepidopterología. 2(4). 9420073–9420073. 2 indexed citations
3.
Zheng, Jie, Han She, Rui Han, et al.. (2025). Dapk2 dysfunction leads to Mic60 lactylation and mitochondrial metabolic reprogramming, promoting lung cancer EGFR-TKI resistance and metastasis. Developmental Cell. 60(23). 3267–3284.e7. 1 indexed citations
4.
Liu, Yihui, Rui Han, Jie Zheng, et al.. (2025). Multieffect Specific Nanovesicles for Homing Resistant Tumors and Overcoming Osimertinib‐Acquired Resistance in NSCLC. Advanced Healthcare Materials. 14(9). e2404087–e2404087. 2 indexed citations
5.
Wang, Zhiguo, Yan Dong, Li Li, et al.. (2024). AXIN1/MYC Axis Mediated the Osimertinib Resistance in EGFR Mutant Non-Small Cell Lung Cancer Cells. The Tohoku Journal of Experimental Medicine. 262(4). 269–276.
6.
Zheng, Jie, Jun Kang, Liping Wang, et al.. (2024). Mesoporous manganese nanocarrier target delivery metformin for the co-activation STING pathway to overcome immunotherapy resistance. iScience. 27(7). 110150–110150. 6 indexed citations
7.
Lu, Conghua, Chen Hu, Yihui Liu, et al.. (2024). Understanding the dynamics of TKI-induced changes in the tumor immune microenvironment for improved therapeutic effect. Journal for ImmunoTherapy of Cancer. 12(6). e009165–e009165. 9 indexed citations
8.
Zheng, Nan, Yimin Zhang, Qiang Ma, et al.. (2023). Pathological Response and Tumor Immune Microenvironment Remodeling Upon Neoadjuvant ALK-TKI Treatment in ALK-Rearranged Non-Small Cell Lung Cancer. Targeted Oncology. 18(4). 625–636. 5 indexed citations
9.
Wang, Yubo, Rui Han, Chen Hu, et al.. (2023). Overall signature of acquired KRAS gene changes in advanced non-small cell lung cancer patient with EGFR-TKI resistance. Japanese Journal of Clinical Oncology. 54(1). 89–96. 1 indexed citations
10.
Li, Li, Zhujun Li, Conghua Lu, et al.. (2022). Ibrutinib reverses IL-6-induced osimertinib resistance through inhibition of Laminin α5/FAK signaling. Communications Biology. 5(1). 155–155. 26 indexed citations
11.
Lu, Conghua, Rui Yu, Chong Zhang, et al.. (2022). Protective autophagy decreases lorlatinib cytotoxicity through Foxo3a-dependent inhibition of apoptosis in NSCLC. Cell Death Discovery. 8(1). 221–221. 9 indexed citations
12.
Wang, Yubo, Chen Hu, Mengxiao Zhu, et al.. (2022). Predictive value of early kinetics of ctDNA combined with cfDNA and serum CEA for EGFR‐TKI treatment in advanced non‐small cell lung cancer. Thoracic Cancer. 13(22). 3162–3173. 9 indexed citations
13.
Chen, Hengyi, Conghua Lu, Caiyu Lin, et al.. (2021). VPS34 suppression reverses osimertinib resistance via simultaneously inhibiting glycolysis and autophagy. Carcinogenesis. 42(6). 880–890. 9 indexed citations
14.
Wang, Yubo, Rui Han, Qiushi Wang, et al.. (2021). Biological Significance of 18F-FDG PET/CT Maximum Standard Uptake Value for Predicting EGFR Mutation Status in Non-Small Cell Lung Cancer Patients. International Journal of General Medicine. Volume 14. 347–356. 8 indexed citations
15.
Lin, Caiyu, Hengyi Chen, Rui Han, et al.. (2021). Hexokinases II‐mediated glycolysis governs susceptibility to crizotinib in ALK‐positive non‐small cell lung cancer. Thoracic Cancer. 12(23). 3184–3193. 16 indexed citations
16.
Chen, Hengyi, Caiyu Lin, Tao Peng, et al.. (2020). Metformin reduces HGF-induced resistance to alectinib via the inhibition of Gab1. Cell Death and Disease. 11(2). 111–111. 20 indexed citations
17.
Wang, Yubo, Caiyu Lin, Rui Han, et al.. (2020). Metformin attenuates TGF-β1-induced pulmonary fibrosis through inhibition of transglutaminase 2 and subsequent TGF-β pathways. 3 Biotech. 10(6). 287–287. 12 indexed citations
18.
Han, Rui, Shuai Hao, Conghua Lu, et al.. (2020). Aspirin sensitizes osimertinib‐resistant NSCLC cells in vitro and in vivo via Bim‐dependent apoptosis induction. Molecular Oncology. 14(6). 1152–1169. 26 indexed citations
19.
20.
Lin, Caiyu, Conghua Lu, Li Li, et al.. (2018). Metformin synergistically enhances the antitumor activity of the third‐generation EGFR‐TKI CO‐1686 in lung cancer cells through suppressing NF‐κB signaling. The Clinical Respiratory Journal. 12(12). 2642–2652. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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