Christopher T. Pappas

1.6k total citations
25 papers, 1.0k citations indexed

About

Christopher T. Pappas is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cell Biology. According to data from OpenAlex, Christopher T. Pappas has authored 25 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Cardiology and Cardiovascular Medicine, 16 papers in Molecular Biology and 8 papers in Cell Biology. Recurrent topics in Christopher T. Pappas's work include Cardiomyopathy and Myosin Studies (17 papers), Cardiovascular Effects of Exercise (9 papers) and Cellular Mechanics and Interactions (8 papers). Christopher T. Pappas is often cited by papers focused on Cardiomyopathy and Myosin Studies (17 papers), Cardiovascular Effects of Exercise (9 papers) and Cellular Mechanics and Interactions (8 papers). Christopher T. Pappas collaborates with scholars based in United States, Germany and Australia. Christopher T. Pappas's co-authors include Carol C. Gregorio, Paul A. Krieg, Anke Zieseniß, Henk Granzier, Miensheng Chu, John A. Cooper, Nandini Bhattacharya, Panagiotis Zaharias, Alla S. Kostyukova and Natalia Moroz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Genes & Development and The Journal of Cell Biology.

In The Last Decade

Christopher T. Pappas

24 papers receiving 1.0k citations

Peers

Christopher T. Pappas

CCM

GENET

M A Strehler-Page United States

Verena Arndt Germany

David H. Heeley Canada

Maki Yoshio Japan

Tushar Chakraborty India

Tomohiko Ishikawa Japan

Hiroyuki Kyushiki Japan

Thomas R. Roos Switzerland

Michael Perry United States

Luciano H. Apponi United States

M A Strehler-Page United States

Christopher T. Pappas

719 ×1.0

264 ×0.5

CCM

94 ×0.4

102 ×1.4

GENET

18 ×0.3

GENET

Citations per year, relative to Christopher T. Pappas Christopher T. Pappas (= 1×) peers M A Strehler-Page

Countries citing papers authored by Christopher T. Pappas

Since Specialization

Citations

This map shows the geographic impact of Christopher T. Pappas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher T. Pappas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher T. Pappas more than expected).

Fields of papers citing papers by Christopher T. Pappas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher T. Pappas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher T. Pappas. The network helps show where Christopher T. Pappas may publish in the future.

Co-authorship network of co-authors of Christopher T. Pappas

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher T. Pappas. A scholar is included among the top collaborators of Christopher T. Pappas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher T. Pappas. Christopher T. Pappas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pappas, Christopher T., et al.. (2024). Leiomodin 2 neonatal dilated cardiomyopathy mutation results in altered actin gene signatures and cardiomyocyte dysfunction. npj Regenerative Medicine. 9(1). 21–21.

Pappas, Christopher T., et al.. (2024). Human disease-causing mutations result in loss of leiomodin 2 through nonsense-mediated mRNA decay. PLoS Genetics. 20(5). e1011279–e1011279. 3 indexed citations

Farman, Gerrie P., Michaela Yuen, Rebecca C. Ahrens‐Nicklas, et al.. (2024). Lmod2 is necessary for effective skeletal muscle contraction. Science Advances. 10(11). eadk1890–eadk1890. 4 indexed citations

Yuen, Michaela, Lisa Worgan, Christopher T. Pappas, et al.. (2022). Neonatal-lethal dilated cardiomyopathy due to a homozygous LMOD2 donor splice-site variant. European Journal of Human Genetics. 30(4). 450–457. 11 indexed citations

Mi‐Mi, Lei, Gerrie P. Farman, Joshua Strom, et al.. (2020). In vivo elongation of thin filaments results in heart failure. PLoS ONE. 15(1). e0226138–e0226138. 15 indexed citations

Pappas, Christopher T., et al.. (2018). Cardiac-specific knockout of Lmod2 results in a severe reduction in myofilament force production and rapid cardiac failure. Journal of Molecular and Cellular Cardiology. 122. 88–97. 24 indexed citations

Eapen, Alex K., et al.. (2017). Acute and sub-chronic oral toxicity studies of erythritol in Beagle dogs. Food and Chemical Toxicology. 105. 448–455. 10 indexed citations

Moroz, Natalia, Christopher T. Pappas, Stefanie M. Novak, et al.. (2016). The N-terminal tropomyosin- and actin-binding sites are important for leiomodin 2’s function. Molecular Biology of the Cell. 27(16). 2565–2575. 21 indexed citations

Kolb, Justin, Frank Li, Mei Methawasin, et al.. (2016). Thin filament length in the cardiac sarcomere varies with sarcomere length but is independent of titin and nebulin. Journal of Molecular and Cellular Cardiology. 97. 286–294. 26 indexed citations

10.

Chu, Miensheng, Carol C. Gregorio, & Christopher T. Pappas. (2016). Nebulin, a multi-functional giant. Journal of Experimental Biology. 219(2). 146–152. 37 indexed citations

11.

Zaharias, Panagiotis & Christopher T. Pappas. (2016). Quality Management of Learning Management Systems: A User Experience Perspective. 3(1). 5. 36 indexed citations

12.

Pappas, Christopher T., Christine Henderson, Cathleen Cover, et al.. (2015). Knockout of Lmod2 results in shorter thin filaments followed by dilated cardiomyopathy and juvenile lethality. Proceedings of the National Academy of Sciences. 112(44). 13573–13578. 74 indexed citations

13.

Granzier, Henk, Kirk R. Hutchinson, Paola Tonino, et al.. (2014). Deleting titin’s I-band/A-band junction reveals critical roles for titin in biomechanical sensing and cardiac function. Proceedings of the National Academy of Sciences. 111(40). 14589–14594. 68 indexed citations

14.

Donlin, Laura T., Christian Andresen, Steffen Just, et al.. (2012). Smyd2 controls cytoplasmic lysine methylation of Hsp90 and myofilament organization. Genes & Development. 26(2). 114–119. 128 indexed citations

15.

Pappas, Christopher T., et al.. (2010). The Nebulin family: an actin support group. Trends in Cell Biology. 21(1). 29–37. 84 indexed citations

16.

Tsukada, Takehiro, Christopher T. Pappas, Natalia Moroz, et al.. (2010). Leiomodin-2 is an antagonist of tropomodulin-1 at the pointed end of the thin filaments in cardiac muscle. Journal of Cell Science. 123(18). 3136–3145. 86 indexed citations

17.

Pappas, Christopher T., Paul A. Krieg, & Carol C. Gregorio. (2010). Nebulin regulates actin filament lengths by a stabilization mechanism. The Journal of General Physiology. 136(1). i1–i1. 4 indexed citations

18.

Tonino, Paola, Christopher T. Pappas, Bryan D. Hudson, et al.. (2010). Reduced myofibrillar connectivity and increased Z-disk width in nebulin-deficient skeletal muscle. Journal of Cell Science. 123(3). 384–391. 52 indexed citations

19.

Pappas, Christopher T., Nandini Bhattacharya, John A. Cooper, & Carol C. Gregorio. (2008). Nebulin Interacts with CapZ and Regulates Thin Filament Architecture within the Z-Disc. Molecular Biology of the Cell. 19(5). 1837–1847. 74 indexed citations

20.

Cutter, Asher D., et al.. (2005). Transposable Element Orientation Bias in the Drosophila melanogaster Genome. Journal of Molecular Evolution. 61(6). 733–41. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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