Christopher Piper

1.0k total citations · 1 hit paper
7 papers, 791 citations indexed

About

Christopher Piper is a scholar working on Immunology, Epidemiology and Hematology. According to data from OpenAlex, Christopher Piper has authored 7 papers receiving a total of 791 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 2 papers in Epidemiology and 2 papers in Hematology. Recurrent topics in Christopher Piper's work include T-cell and B-cell Immunology (5 papers), Immune Cell Function and Interaction (3 papers) and Inflammatory Myopathies and Dermatomyositis (2 papers). Christopher Piper is often cited by papers focused on T-cell and B-cell Immunology (5 papers), Immune Cell Function and Interaction (3 papers) and Inflammatory Myopathies and Dermatomyositis (2 papers). Christopher Piper collaborates with scholars based in United Kingdom, Austria and Germany. Christopher Piper's co-authors include Claudia Mauri, Elizabeth C. Rosser, Lucy R. Wedderburn, André F. Rendeiro, Christoph Bock, Ignat Drozdov, Thomas Krausgruber, Dagmar Alber, Paul A. Blair and Diego Catalán and has published in prestigious journals such as The Journal of Immunology, Cell Metabolism and Frontiers in Immunology.

In The Last Decade

Christopher Piper

7 papers receiving 780 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Microbiota-Derived Metabolites Suppress Arthritis by Amplifying Aryl-Hydrocarbon Receptor Activation in Regulatory B Cells

2020 412 citations Elizabeth C. Rosser, Christopher Piper et al. Cell Metabolism profile →

Peers

Christopher Piper

MB

IMMUN

RHEUM

PHYSI

EPIDE

Renê Donizeti Ribeiro de Oliveira Brazil

Verena Brucklacher-Waldert United Kingdom

Yael Alippe United States

Rafael S. Grajewski Germany

Xuyan Yang China

Paula B. Donate Brazil

Denise Lasigliè Italy

Wanwan Huai China

Viola Klück Netherlands

Hiroko Kushiyama Japan

Renê Donizeti Ribeiro de Oliveira Brazil

Christopher Piper

254 ×0.7

MB

310 ×0.9

IMMUN

208 ×1.2

RHEUM

44 ×0.4

PHYSI

50 ×0.5

EPIDE

Citations per year, relative to Christopher Piper Christopher Piper (= 1×) peers Renê Donizeti Ribeiro de Oliveira

Countries citing papers authored by Christopher Piper

Since Specialization

Citations

This map shows the geographic impact of Christopher Piper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher Piper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher Piper more than expected).

Fields of papers citing papers by Christopher Piper

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher Piper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher Piper. The network helps show where Christopher Piper may publish in the future.

Co-authorship network of co-authors of Christopher Piper

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher Piper. A scholar is included among the top collaborators of Christopher Piper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher Piper. Christopher Piper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Rosser, Elizabeth C., Christopher Piper, Diana E. Matei, et al.. (2020). Microbiota-Derived Metabolites Suppress Arthritis by Amplifying Aryl-Hydrocarbon Receptor Activation in Regulatory B Cells. Cell Metabolism. 31(4). 837–851.e10. 412 indexed citations breakdown →

2.

Piper, Christopher, Elizabeth C. Rosser, Kristīne Oļeiņika, et al.. (2019). Aryl Hydrocarbon Receptor Contributes to the Transcriptional Program of IL-10-Producing Regulatory B Cells. Cell Reports. 29(7). 1878–1892.e7. 137 indexed citations

3.

Piper, Christopher, Meredyth Wilkinson, Claire T. Deakin, et al.. (2018). CD19+CD24hiCD38hi B Cells Are Expanded in Juvenile Dermatomyositis and Exhibit a Pro-Inflammatory Phenotype After Activation Through Toll-Like Receptor 7 and Interferon-α. Frontiers in Immunology. 9. 1372–1372. 75 indexed citations

4.

Piper, Christopher, David Bending, Hemlata Varsani, et al.. (2014). Regulatory B cell Il-10 production is diminished in juvenile dermatomyositis. Pediatric Rheumatology. 12(S1). 1 indexed citations

5.

Piper, Christopher, Anne M. Pesenacker, David Bending, et al.. (2014). Brief Report: T Cell Expression of Granulocyte–Macrophage Colony‐Stimulating Factor in Juvenile Arthritis Is Contingent Upon Th17 Plasticity. Arthritis & Rheumatology. 66(7). 1955–1960. 56 indexed citations

6.

Wong‐Baeza, Isabel, Anna Ridley, Hiroko Hatano, et al.. (2013). KIR3DL2 Binds to HLA-B27 Dimers and Free H Chains More Strongly than Other HLA Class I and Promotes the Expansion of T Cells in Ankylosing Spondylitis. The Journal of Immunology. 190(7). 3216–3224. 69 indexed citations

7.

Giles, Joanna L., Christopher Piper, Isabel Wong‐Baeza, et al.. (2012). HLA-B27 Homodimers and Free H Chains Are Stronger Ligands for Leukocyte Ig-like Receptor B2 than Classical HLA Class I. The Journal of Immunology. 188(12). 6184–6193. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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