Christopher J. Hille

931 citations
16 papers · 738 indexed · h-index 13

Christopher J. Hille

15 papers receiving 722 citations

Peers

Christopher J. Hille
Comparison fields: 5 of 68
  • Cellular and Molecular Neuroscience 479
  • Neurology 164
  • Cognitive Neuroscience 120
  • Physiology 159
  • Neurology 48
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Anne Michel Belgium
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Citations per year

Countries citing papers authored by Christopher J. Hille

Since Specialization
Citations

This map shows the geographic impact of Christopher J. Hille's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Hille with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Hille more than expected).

Fields of papers citing papers by Christopher J. Hille

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher J. Hille. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Hille. The network helps show where Christopher J. Hille may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Christopher J. Hille, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Christopher J. Hille Line = papers co-authored together Christopher J. Hille links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 201122
2 20107
3 200834
4 200812
5 20081
6 2006133
7 200251
8 200155
9
Dopamine release and uptake kinetics in the putamen of non-human primate striatum in vitro: Species and region differences
20004
10 2000100
11 200082
12 200090
13
Opioid peptide precursor expression in animal models of dystonia secondary to dopamine-replacement therapy in Parkinson's disease.
199822
14 199627
15 198842
16 198756

About Christopher J. Hille

Christopher J. Hille is a scholar working on Cellular and Molecular Neuroscience, Neurology, Physiology, Pharmacology and Physiology, having authored 16 papers that have together received 738 indexed citations. Recurring topics across this work include Neurotransmitter Receptor Influence on Behavior (7 papers), Neuroscience and Neuropharmacology Research (6 papers), Parkinson's Disease Mechanisms and Treatments (5 papers), Alzheimer's disease research and treatments (3 papers), Pharmacological Receptor Mechanisms and Effects (3 papers), Cholinesterase and Neurodegenerative Diseases (3 papers), Receptor Mechanisms and Signaling (2 papers) and Neuropeptides and Animal Physiology (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (479 citations), Neurology (164 citations), Cognitive Neuroscience (120 citations), Physiology (159 citations) and Neurology (48 citations). Christopher J. Hille has collaborated with scholars based in United Kingdom, Russia and Canada. Frequent co-authors include Stephanie J. Cragg, Susan A. Greenfield, Jonathan M. Brotchie, S.A. Greenfield, A.J. Cross, P. Slater, Yannick P. Maneuf, Alan R. Crossman, Susan H. Fox and Bill Henry. Their work appears in journals such as Experimental Neurology, Journal of Neuroscience, Alzheimer s & Dementia, Journal of Pain and Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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