Christophe Alberti

1.2k total citations
8 papers, 275 citations indexed

About

Christophe Alberti is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Oncology. According to data from OpenAlex, Christophe Alberti has authored 8 papers receiving a total of 275 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Pediatrics, Perinatology and Child Health and 2 papers in Oncology. Recurrent topics in Christophe Alberti's work include DNA Repair Mechanisms (2 papers), Childhood Cancer Survivors' Quality of Life (2 papers) and Adolescent and Pediatric Healthcare (2 papers). Christophe Alberti is often cited by papers focused on DNA Repair Mechanisms (2 papers), Childhood Cancer Survivors' Quality of Life (2 papers) and Adolescent and Pediatric Healthcare (2 papers). Christophe Alberti collaborates with scholars based in France, United States and Canada. Christophe Alberti's co-authors include Lionel Larue, Patrick Pla, Takahiro Kunisada, Domenico Albino, Anna Marie Skalka, Valeria Masciullo, René Daniel, Neal J. Meropol, Alfonso Bellacosa and Filippo Schepis and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Clinical Cancer Research and Pigment Cell & Melanoma Research.

In The Last Decade

Christophe Alberti

8 papers receiving 273 citations

Peers

Christophe Alberti

MB

ONCOL

CB

CR

PFM

Job de Lange Netherlands

Nikhil R. Bhagwat United States

Liat Goldberg United States

Bjoern Seidel Germany

Alessia Galasso Italy

Stéphanie Vinot France

Xiafang Zhang United States

Letícia Koch Lerner Brazil

Somadri Ghosh Belgium

Christel Boutonnet France

Job de Lange Netherlands

Christophe Alberti

316 ×1.4

MB

147 ×2.3

ONCOL

57 ×1.4

CB

51 ×1.2

CR

14 ×0.4

PFM

Citations per year, relative to Christophe Alberti Christophe Alberti (= 1×) peers Job de Lange

Countries citing papers authored by Christophe Alberti

Since Specialization

Citations

This map shows the geographic impact of Christophe Alberti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christophe Alberti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christophe Alberti more than expected).

Fields of papers citing papers by Christophe Alberti

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christophe Alberti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christophe Alberti. The network helps show where Christophe Alberti may publish in the future.

Co-authorship network of co-authors of Christophe Alberti

This figure shows the co-authorship network connecting the top 25 collaborators of Christophe Alberti. A scholar is included among the top collaborators of Christophe Alberti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christophe Alberti. Christophe Alberti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Petit, Valérie, Christophe Alberti, Stuart Gallagher, et al.. (2019). C57BL/6 congenic mouse NRAS^Q61K melanoma cell lines are highly sensitive to the combination of Mek and Akt inhibitors in vitro and in vivo. Pigment Cell & Melanoma Research. 32(6). 829–841. 24 indexed citations

2.

Guilmin‐Crépon, Sophie, et al.. (2016). Propriétés psychométriques du Good 2 Go, premier questionnaire en langue française d’aptitude à la transition pédiatrie – médecine d’adultes pour adolescents malades chroniques. Revue d Épidémiologie et de Santé Publique. 64. S190–S191. 1 indexed citations

3.

Guilmin‐Crépon, Sophie, et al.. (2016). Propriétés psychométriques du Good 2 Go, premier questionnaire en langue française d’aptitude à la transition pédiatrie–médecine d’adultes pour adolescents malades chroniques. Revue d Épidémiologie et de Santé Publique. 64. S160–S160. 1 indexed citations

4.

Quanz, Maria, Nathalie Berthault, Maryline Roy, et al.. (2009). Small-Molecule Drugs Mimicking DNA Damage: A New Strategy for Sensitizing Tumors to Radiotherapy. Clinical Cancer Research. 15(4). 1308–1316. 76 indexed citations

5.

Pla, Patrick, et al.. (2005). Ednrb2 orients cell migration towards the dorsolateral neural crest pathway and promotes melanocyte differentiation. Pigment Cell Research. 18(3). 181–187. 49 indexed citations

6.

Pla, Patrick, et al.. (2004). Dct::lacZ ES Cells: A Novel Cellular Model to Study Melanocyte Determination and Differentiation. Pigment Cell Research. 17(2). 142–149. 20 indexed citations

7.

Alberti, Christophe, et al.. (2003). Tyrosinase Gene Correction Using Fluorescent Oligonucleotides. Pigment Cell Research. 16(2). 133–138. 5 indexed citations

8.

Cortellino, Salvatore, David P. Turner, Valeria Masciullo, et al.. (2003). The base excision repair enzyme MED1 mediates DNA damage response to antitumor drugs and is associated with mismatch repair system integrity. Proceedings of the National Academy of Sciences. 100(25). 15071–15076. 99 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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