Chiaki Iwamura

2.2k total citations
43 papers, 1.7k citations indexed

About

Chiaki Iwamura is a scholar working on Immunology, Physiology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Chiaki Iwamura has authored 43 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Immunology, 15 papers in Physiology and 11 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Chiaki Iwamura's work include Immune Cell Function and Interaction (20 papers), Asthma and respiratory diseases (14 papers) and IL-33, ST2, and ILC Pathways (12 papers). Chiaki Iwamura is often cited by papers focused on Immune Cell Function and Interaction (20 papers), Asthma and respiratory diseases (14 papers) and IL-33, ST2, and ILC Pathways (12 papers). Chiaki Iwamura collaborates with scholars based in Japan, United States and Australia. Chiaki Iwamura's co-authors include Toshinori Nakayama, Kenta Shinoda, Hiroyuki Hosokawa, Damon J. Tumes, Shinichiro Motohashi, Masakatsu Yamashita, Atsushi Onodera, Koji Tokoyoda, Yusuke Endo and Haruhiko Koseki and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and SHILAP Revista de lepidopterología.

In The Last Decade

Chiaki Iwamura

43 papers receiving 1.6k citations

Peers

Chiaki Iwamura
Kenta Shinoda Japan
Louis M. Renzetti United States
Yves Laumonnier Germany
Mohini Gray United Kingdom
Anupriya Khare United States
Tracy L. Burcin United States
Cláudia Zuany‐Amorim France
Elena Izquierdo Spain
Sébastien Fleury France
Takeshi Otsuka Japan
Kenta Shinoda Japan
Chiaki Iwamura
Citations per year, relative to Chiaki Iwamura Chiaki Iwamura (= 1×) peers Kenta Shinoda

Countries citing papers authored by Chiaki Iwamura

Since Specialization
Citations

This map shows the geographic impact of Chiaki Iwamura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chiaki Iwamura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chiaki Iwamura more than expected).

Fields of papers citing papers by Chiaki Iwamura

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chiaki Iwamura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chiaki Iwamura. The network helps show where Chiaki Iwamura may publish in the future.

Co-authorship network of co-authors of Chiaki Iwamura

This figure shows the co-authorship network connecting the top 25 collaborators of Chiaki Iwamura. A scholar is included among the top collaborators of Chiaki Iwamura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chiaki Iwamura. Chiaki Iwamura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kaneko, Tatsuya, Chiaki Iwamura, Masahiro Kiuchi, et al.. (2024). Amphiregulin-producing TH2 cells facilitate esophageal fibrosis of eosinophilic esophagitis. SHILAP Revista de lepidopterología. 3(3). 100287–100287. 2 indexed citations
2.
Onodera, Atsushi, Kota Kokubo, Masahiro Kiuchi, et al.. (2023). Pathogenic helper T cells as the novel therapeutic targets for immune-mediated intractable diseases. Pharmacology & Therapeutics. 247. 108445–108445. 5 indexed citations
3.
Iwamura, Chiaki & Toshinori Nakayama. (2018). Role of CD1d- and MR1-Restricted T Cells in Asthma. Frontiers in Immunology. 9. 1942–1942. 22 indexed citations
4.
5.
Sugamata, Ryuichi, Chiaki Iwamura, Tomokazu Nagao, et al.. (2013). Effect of invariant natural killer T cells with IL-5 and activated IL-6 receptor in ventilator-associated lung injury in mice. Experimental Lung Research. 40(1). 1–11. 5 indexed citations
6.
Hasegawa, Akihiro, Chiaki Iwamura, Masayuki Kitajima, et al.. (2013). Crucial Role for CD69 in the Pathogenesis of Dextran Sulphate Sodium-Induced Colitis. PLoS ONE. 8(6). e65494–e65494. 18 indexed citations
7.
Kasuya, Yoshitoshi, Kensuke Tanaka, Keita Yamauchi, et al.. (2012). Role of CD69 in acute lung injury. Life Sciences. 90(17-18). 657–665. 12 indexed citations
8.
Yamashita, Junji, Chiaki Iwamura, Kunitoshi Mitsumori, et al.. (2011). Murine Schnurri-2 controls natural killer cell function and lymphoma development. Leukemia & lymphoma. 53(3). 479–486. 4 indexed citations
9.
Kasuya, Yoshitoshi, Keita Yamauchi, Hiromi Sugimoto, et al.. (2011). Cigarette smoke-induced pulmonary inflammation is attenuated in CD69-deficient mice. Journal of Receptors and Signal Transduction. 31(6). 434–439. 12 indexed citations
10.
Yamauchi, Keita, Yoshitoshi Kasuya, Kensuke Tanaka, et al.. (2011). Attenuation of lung inflammation and fibrosis in CD69-deficient mice after intratracheal bleomycin. Respiratory Research. 12(1). 131–131. 32 indexed citations
11.
Suzuki, Akane, Chiaki Iwamura, Kenta Shinoda, et al.. (2010). Polycomb Group Gene Product Ring1B Regulates Th2-Driven Airway Inflammation through the Inhibition of Bim-Mediated Apoptosis of Effector Th2 Cells in the Lung. The Journal of Immunology. 184(8). 4510–4520. 17 indexed citations
12.
Hasegawa, Akihiro, Chiaki Iwamura, Kenta Shinoda, et al.. (2009). CD69 Controls the Pathogenesis of Allergic Airway Inflammation. The Journal of Immunology. 183(12). 8203–8215. 70 indexed citations
13.
Kitajima, Masayuki, Chiaki Iwamura, Kenta Shinoda, et al.. (2009). Enhanced Th2 Cell Differentiation and Allergen-Induced Airway Inflammation in Zfp35 -Deficient Mice. The Journal of Immunology. 183(8). 5388–5396. 9 indexed citations
14.
Terashima, Asuka, Hiroshi Watarai, Sayo Inoue, et al.. (2008). A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity. The Journal of Experimental Medicine. 205(12). 2727–2733. 190 indexed citations
15.
Hirahara, Kiyoshi, Masakatsu Yamashita, Chiaki Iwamura, et al.. (2008). Repressor of GATA regulates TH2-driven allergic airway inflammation and airway hyperresponsiveness. Journal of Allergy and Clinical Immunology. 122(3). 512–520.e11. 42 indexed citations
16.
Iwamura, Chiaki & Toshinori Nakayama. (2008). Toll-like receptors in the respiratory system: Their roles in inflammation. Current Allergy and Asthma Reports. 8(1). 7–13. 57 indexed citations
17.
Kimura, Motoko Y., Chiaki Iwamura, Akane Suzuki, et al.. (2007). Schnurri-2 Controls Memory Th1 and Th2 Cell Numbers In Vivo. The Journal of Immunology. 178(8). 4926–4936. 17 indexed citations
18.
Iwamura, Chiaki, Minoru Kimura, Kenta Shinoda, et al.. (2007). Schnurri-2 regulates Th2-dependent airway inflammation and airway hyperresponsiveness. International Immunology. 19(6). 755–762. 10 indexed citations
19.
Iwamura, Chiaki & Toshinori Nakayama. (2007). Role of α-Galactosylceramide-activated Vα14 Natural Killer T Cells in the Regulation of Allergic Diseases. Allergology International. 56(1). 1–6. 18 indexed citations
20.
Iwamura, Chiaki, Michiko Aihara, Masatoshi Nakazawa, et al.. (2006). Effects of Tributyltin on the Development of Atopic Dermatitis-Like Eruptions in DS-<i>Nh</i> Mice. International Archives of Allergy and Immunology. 141(4). 337–345. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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