Chi He

825 total citations
11 papers, 644 citations indexed

About

Chi He is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, Chi He has authored 11 papers receiving a total of 644 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 10 papers in Cancer Research and 2 papers in Epidemiology. Recurrent topics in Chi He's work include MicroRNA in disease regulation (6 papers), Cancer, Hypoxia, and Metabolism (5 papers) and RNA modifications and cancer (4 papers). Chi He is often cited by papers focused on MicroRNA in disease regulation (6 papers), Cancer, Hypoxia, and Metabolism (5 papers) and RNA modifications and cancer (4 papers). Chi He collaborates with scholars based in China. Chi He's co-authors include Shi-Jiang Deng, Shichang Deng, Gang Zhao, Chunyou Wang, Shuai Zhu, Mingliang Liu, Zhu Zeng, Xiang Li, Yan Jin and Ye Zeng and has published in prestigious journals such as Cancer Research, Oncogene and The Journal of Pathology.

In The Last Decade

Chi He

11 papers receiving 640 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chi He China 10 533 493 108 41 39 11 644
Shi-Jiang Deng China 12 696 1.3× 654 1.3× 168 1.6× 61 1.5× 69 1.8× 14 874
Hezhao Zhao China 11 284 0.5× 175 0.4× 82 0.8× 35 0.9× 36 0.9× 18 413
Ya‐Ping Tsai Taiwan 6 371 0.7× 243 0.5× 120 1.1× 22 0.5× 19 0.5× 6 515
Xinhua Xie China 10 420 0.8× 338 0.7× 99 0.9× 14 0.3× 23 0.6× 12 551
Liang Chang China 13 399 0.7× 346 0.7× 42 0.4× 33 0.8× 27 0.7× 21 478
Liangfeng Han United States 7 434 0.8× 310 0.6× 80 0.7× 11 0.3× 29 0.7× 8 556
Thomas H. Y. Leung Hong Kong 7 392 0.7× 197 0.4× 93 0.9× 17 0.4× 36 0.9× 7 469

Countries citing papers authored by Chi He

Since Specialization
Citations

This map shows the geographic impact of Chi He's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chi He with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chi He more than expected).

Fields of papers citing papers by Chi He

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chi He. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chi He. The network helps show where Chi He may publish in the future.

Co-authorship network of co-authors of Chi He

This figure shows the co-authorship network connecting the top 25 collaborators of Chi He. A scholar is included among the top collaborators of Chi He based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chi He. Chi He is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Wang, Qi, et al.. (2022). CircRNF121 knockdown suppresses the progression of cervical cancer by regulating miR ‐153‐3p/ ATF2 axis and wnt/β‐catenin pathway. Drug Development Research. 83(3). 755–768. 3 indexed citations
2.
He, Chi, Yi Fu, Yan Chen, & Xiquan Li. (2021). Long non‑coding RNA SNHG8 promotes autophagy as a ceRNA to upregulate ATG7 by sponging microRNA‑588 in colorectal cancer. Oncology Letters. 22(2). 577–577. 20 indexed citations
3.
Deng, Shi-Jiang, Hengyu Chen, Zhu Zeng, et al.. (2018). Nutrient Stress–Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Progression. Cancer Research. 79(7). 1398–1412. 103 indexed citations
4.
Niu, Yi, Yan Jin, Shichang Deng, et al.. (2018). MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. Oncogene. 37(13). 1743–1758. 46 indexed citations
5.
Li, Xiang, Shichang Deng, Mingliang Liu, et al.. (2018). The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. Cell Death and Disease. 9(2). 133–133. 32 indexed citations
6.
Deng, Shi-Jiang, Heng‐Yu Chen, Ye Zeng, et al.. (2018). Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcription. Oncogene. 37(44). 5811–5828. 162 indexed citations
8.
Zhu, Shuai, Hai-Yun Zhou, Shichang Deng, et al.. (2017). ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway. Cell Death and Disease. 8(5). e2806–e2806. 86 indexed citations
9.
Zhu, Shuai, Chi He, Shi-Jiang Deng, et al.. (2016). MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting Vimentin Expression. Molecular Cancer Therapeutics. 15(9). 2209–2219. 53 indexed citations
10.
Li, Xiang, Shi-Jiang Deng, Shuai Zhu, et al.. (2016). Hypoxia-induced lncRNA-NUTF2P3-001 contributes to tumorigenesis of pancreatic cancer by derepressing the miR-3923/KRAS pathway. Oncotarget. 7(5). 6000–6014. 80 indexed citations
11.
Deng, Shichang, Xiang Li, Yi Niu, et al.. (2015). MiR-652 inhibits acidic microenvironment-induced epithelial-mesenchymal transition of pancreatic cancer cells by targeting ZEB1. Oncotarget. 6(37). 39661–39675. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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