Chengchun Min

479 total citations
15 papers, 402 citations indexed

About

Chengchun Min is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Chengchun Min has authored 15 papers receiving a total of 402 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 4 papers in Cell Biology. Recurrent topics in Chengchun Min's work include Receptor Mechanisms and Signaling (12 papers), Neuropeptides and Animal Physiology (6 papers) and Protein Kinase Regulation and GTPase Signaling (4 papers). Chengchun Min is often cited by papers focused on Receptor Mechanisms and Signaling (12 papers), Neuropeptides and Animal Physiology (6 papers) and Protein Kinase Regulation and GTPase Signaling (4 papers). Chengchun Min collaborates with scholars based in South Korea, Japan and United States. Chengchun Min's co-authors include Kyeong‐Man Kim, Mei Zheng, Xiaohan Zhang, Seung Hoon Cheon, Kyeong-Man Kim, Jae H. Park, Min–Ho Oak, Lan Ma, Hitoshi Kurose and Chan Young Shin and has published in prestigious journals such as Molecular and Cellular Biology, Biochimica et Biophysica Acta (BBA) - Biomembranes and Molecular Pharmacology.

In The Last Decade

Chengchun Min

15 papers receiving 394 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chengchun Min South Korea 13 320 174 68 31 24 15 402
Daniel M. Williams United States 10 398 1.2× 86 0.5× 82 1.2× 57 1.8× 31 1.3× 14 525
Paul Tewson United States 12 432 1.4× 162 0.9× 43 0.6× 20 0.6× 17 0.7× 14 552
Claire Didszun Germany 5 201 0.6× 104 0.6× 70 1.0× 28 0.9× 12 0.5× 6 306
Jessica A. Hennessey United States 10 463 1.4× 96 0.6× 60 0.9× 29 0.9× 6 0.3× 16 565
Sajag Bhattarai United States 11 461 1.4× 118 0.7× 100 1.5× 18 0.6× 44 1.8× 24 547
Samantha L. Yuen United States 14 289 0.9× 72 0.4× 57 0.8× 54 1.7× 9 0.4× 25 428
Yuka Sugiyama Japan 10 254 0.8× 318 1.8× 53 0.8× 42 1.4× 10 0.4× 23 621
Fen Nie China 11 465 1.5× 71 0.4× 41 0.6× 11 0.4× 29 1.2× 16 597
Pei Zhuang United States 10 267 0.8× 71 0.4× 29 0.4× 17 0.5× 50 2.1× 17 425
Kissaou Tchedre United States 10 298 0.9× 137 0.8× 26 0.4× 28 0.9× 27 1.1× 19 372

Countries citing papers authored by Chengchun Min

Since Specialization
Citations

This map shows the geographic impact of Chengchun Min's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chengchun Min with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chengchun Min more than expected).

Fields of papers citing papers by Chengchun Min

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chengchun Min. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chengchun Min. The network helps show where Chengchun Min may publish in the future.

Co-authorship network of co-authors of Chengchun Min

This figure shows the co-authorship network connecting the top 25 collaborators of Chengchun Min. A scholar is included among the top collaborators of Chengchun Min based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chengchun Min. Chengchun Min is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Lee, Sunmi, Joo Young Lee, Eun‐Woo Lee, et al.. (2019). Absence of Cytosolic 2-Cys Prx Subtypes I and II Exacerbates TNF-α-Induced Apoptosis via Different Routes. Cell Reports. 26(8). 2194–2211.e6. 16 indexed citations
2.
Min, Chengchun, Yukyung Jun, Doo Jae Lee, et al.. (2018). Silencing of peroxiredoxin II by promoter methylation is necessary for the survival and migration of gastric cancer cells. Experimental & Molecular Medicine. 50(2). e443–e443. 14 indexed citations
3.
Zheng, Mei, Xiaohan Zhang, Ningning Sun, et al.. (2016). RalA employs GRK2 and β-arrestins for the filamin A-mediated regulation of trafficking and signaling of dopamine D 2 and D 3 receptor. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(8). 2072–2083. 14 indexed citations
4.
Min, Chengchun, Xiaohan Zhang, Mei Zheng, et al.. (2016). Molecular Signature That Determines the Acute Tolerance of G Protein-Coupled Receptors. Biomolecules & Therapeutics. 25(3). 239–248. 11 indexed citations
5.
Zheng, Mei, et al.. (2016). Functional Regulation of Dopamine D3 Receptor through Interaction with PICK1. Biomolecules & Therapeutics. 24(5). 475–481. 6 indexed citations
6.
Zheng, Mei, Xiaohan Zhang, Xiaowei Zhang, et al.. (2015). Agonist-induced changes in RalA activities allows the prediction of the endocytosis of G protein-coupled receptors. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(1). 77–90. 17 indexed citations
7.
Zhang, Xiaohan, Mei Zheng, Xiaowei Zhang, et al.. (2015). Selectivity of commonly used inhibitors of clathrin-mediated and caveolae-dependent endocytosis of G protein–coupled receptors. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1848(10). 2101–2110. 92 indexed citations
8.
Cao, Yongkai, Chengchun Min, Srijan Acharya, Kyeong‐Man Kim, & Seung Hoon Cheon. (2015). Design, synthesis and evaluation of bitopic arylpiperazinephenyl-1,2,4-oxadiazoles as preferential dopamine D3 receptor ligands. Bioorganic & Medicinal Chemistry. 24(2). 191–200. 15 indexed citations
9.
Min, Chengchun, Mei Zheng, Xiaohan Zhang, et al.. (2014). N-linked Glycosylation on the N-terminus of the dopamine D2 and D3 receptors determines receptor association with specific microdomains in the plasma membrane. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1853(1). 41–51. 40 indexed citations
10.
Min, Chengchun, et al.. (2012). Multiple signaling routes involved in the regulation of adenylyl cyclase and extracellular regulated kinase by dopamine D2 and D3 receptors. Pharmacological Research. 67(1). 31–41. 19 indexed citations
11.
Min, Chengchun, et al.. (2011). RGS4 exerts inhibitory activities on the signaling of dopamine D2 receptor and D3 receptor through the N-terminal region. Pharmacological Research. 65(2). 213–220. 16 indexed citations
12.
Min, Chengchun, et al.. (2011). Novel Regulatory Mechanism of Canonical Wnt Signaling by Dopamine D2 Receptor through Direct Interaction with β-Catenin. Molecular Pharmacology. 80(1). 68–78. 28 indexed citations
13.
Zheng, Mei, et al.. (2011). β-Arrestin2 Plays Permissive Roles in the Inhibitory Activities of RGS9-2 on G Protein-Coupled Receptors by Maintaining RGS9-2 in the Open Conformation. Molecular and Cellular Biology. 31(24). 4887–4901. 29 indexed citations
14.
Zheng, Mei, Chengchun Min, Lan Ma, et al.. (2010). Agonist-Induced Endocytosis and Receptor Phosphorylation Mediate Resensitization of Dopamine D2 Receptors. Molecular Endocrinology. 24(3). 574–586. 71 indexed citations
15.
Cho, Eunyoung, et al.. (2008). Characterization of functional roles of DRY motif in the 2nd intracellular loop of dopamine D2 and D3 receptors. Archives of Pharmacal Research. 31(4). 474–481. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026