Cheng-Chin Kuo

1.4k total citations
45 papers, 1.1k citations indexed

About

Cheng-Chin Kuo is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Cheng-Chin Kuo has authored 45 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 20 papers in Immunology and 7 papers in Cancer Research. Recurrent topics in Cheng-Chin Kuo's work include Immune Response and Inflammation (8 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (6 papers) and Cell Adhesion Molecules Research (6 papers). Cheng-Chin Kuo is often cited by papers focused on Immune Response and Inflammation (8 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (6 papers) and Cell Adhesion Molecules Research (6 papers). Cheng-Chin Kuo collaborates with scholars based in Taiwan, United States and Iran. Cheng-Chin Kuo's co-authors include Jing-Yiing Wu, Kenneth K. Wu, Guan-Lin Lee, Shaw‐Fang Yet, Shu‐Mei Liang, Chi‐Ming Liang, Yi‐Fu Wang, Yu‐Juei Hsu, Liang‐Tung Yang and Pei‐Hsuan Lin and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Applied Physics Letters.

In The Last Decade

Cheng-Chin Kuo

43 papers receiving 1.1k citations

Peers

Cheng-Chin Kuo
Hongtao Lu China
Sandhya Xavier United States
Yan Cui China
Guido Orlandini Italy
Begoña Muñoz‐García Spain
Yu-Chuen Huang Taiwan
James R. Springstead United States
Ting‐Ting Chang Taiwan
An Chen China
Hideki Kimura Japan
Hongtao Lu China
Cheng-Chin Kuo
Citations per year, relative to Cheng-Chin Kuo Cheng-Chin Kuo (= 1×) peers Hongtao Lu

Countries citing papers authored by Cheng-Chin Kuo

Since Specialization
Citations

This map shows the geographic impact of Cheng-Chin Kuo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cheng-Chin Kuo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cheng-Chin Kuo more than expected).

Fields of papers citing papers by Cheng-Chin Kuo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cheng-Chin Kuo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cheng-Chin Kuo. The network helps show where Cheng-Chin Kuo may publish in the future.

Co-authorship network of co-authors of Cheng-Chin Kuo

This figure shows the co-authorship network connecting the top 25 collaborators of Cheng-Chin Kuo. A scholar is included among the top collaborators of Cheng-Chin Kuo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cheng-Chin Kuo. Cheng-Chin Kuo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Wu, Ping‐Hsun, Po‐Yu Liu, Cheng-Chin Kuo, et al.. (2024). Identification of Gut Microbiome Signatures Associated with Indole Pathway in Tryptophan Metabolism in Patients Undergoing Hemodialysis. Biomolecules. 14(6). 623–623. 6 indexed citations
3.
Chang, Tzu-Ching, Yi-Jhu Lu, Shu-Man Liang, et al.. (2024). The effects of acetylated cordycepin derivatives on promoting vascular angiogenesis and attenuating myocardial ischemic injury. Heliyon. 10(21). e40026–e40026.
4.
Chiang, Kuang-Mao, Chia‐Min Chung, Chi-Jen Lo, et al.. (2022). Pharmacometabolomic study of drug response to antihypertensive medications for hypertension marker identification in Han Chinese individuals in Taiwan. Computational and Structural Biotechnology Journal. 20. 6458–6466. 3 indexed citations
5.
Kuo, Cheng-Chin, Jing-Yiing Wu, & Kenneth K. Wu. (2022). Cancer-derived extracellular succinate: a driver of cancer metastasis. Journal of Biomedical Science. 29(1). 93–93. 60 indexed citations
6.
Liang, Shu-Man, Yi‐Ju Wu, Yi-Jhu Lu, et al.. (2019). Cordycepin Suppresses Endothelial Cell Proliferation, Migration, Angiogenesis, and Tumor Growth by Regulating Focal Adhesion Kinase and p53. Cancers. 11(2). 168–168. 24 indexed citations
7.
Yeh, Chang-Ching, et al.. (2019). Vanadium Derivative Exposure Promotes Functional Alterations of VSMCs and Consequent Atherosclerosis via ROS/p38/NF-κB-Mediated IL-6 Production. International Journal of Molecular Sciences. 20(24). 6115–6115. 28 indexed citations
8.
Wang, Yi‐Fu, Yu‐Juei Hsu, Guan-Lin Lee, et al.. (2016). Endothelium-Derived 5-Methoxytryptophan Is a Circulating Anti-Inflammatory Molecule That Blocks Systemic Inflammation. Circulation Research. 119(2). 222–236. 69 indexed citations
9.
Wang, Yi‐Fu, et al.. (2016). Endothelium-Derived 5-Methoxytryptophan Protects Endothelial Barrier Function by Blocking p38 MAPK Activation. PLoS ONE. 11(3). e0152166–e0152166. 32 indexed citations
10.
Wang, Yi‐Fu, Guan-Lin Lee, Yuhua Huang, & Cheng-Chin Kuo. (2016). sn-1,2-diacylglycerols protect against lethal endotoxemia by controlling systemic inflammation. Immunobiology. 221(11). 1309–1318. 14 indexed citations
11.
Lee, Guan-Lin, Jing-Yiing Wu, Chang-Ching Yeh, & Cheng-Chin Kuo. (2016). TLR4 induces CREB-mediated IL-6 production via upregulation of F-spondin to promote vascular smooth muscle cell migration. Biochemical and Biophysical Research Communications. 473(4). 1205–1210. 16 indexed citations
12.
Lin, Pei‐Hsuan, et al.. (2015). N-terminal functional domain of Gasdermin A3 regulates mitochondrial homeostasis via mitochondrial targeting. Journal of Biomedical Science. 22(1). 44–44. 84 indexed citations
13.
Chang, Tzu-Ching, et al.. (2014). Quiescent and Proliferative Fibroblasts Exhibit Differential p300 HAT Activation through Control of 5-Methoxytryptophan Production. PLoS ONE. 9(2). e88507–e88507. 14 indexed citations
14.
Kuo, Cheng-Chin, et al.. (2012). Lobe-Specific Calcium Binding in Calmodulin Regulates Endothelial Nitric Oxide Synthase Activation. PLoS ONE. 7(6). e39851–e39851. 7 indexed citations
15.
Chen, Yiwen, Szu‐Ting Lin, Jimin Li, et al.. (2011). Proteomic analysis of gemcitabine-induced drug resistance in pancreatic cancer cells. Molecular BioSystems. 7(11). 3065–3074. 37 indexed citations
16.
Wu, Jing-Yiing & Cheng-Chin Kuo. (2011). Pivotal Role of ADP-ribosylation Factor 6 in Toll-like Receptor 9-mediated Immune Signaling. Journal of Biological Chemistry. 287(6). 4323–4334. 25 indexed citations
17.
Ko, Bor‐Sheng, Tzu-Ching Chang, Chien‐Hung Chen, et al.. (2009). Bortezomib suppresses focal adhesion kinase expression via interrupting nuclear factor-kappa B. Life Sciences. 86(5-6). 199–206. 31 indexed citations
18.
Kuo, Cheng-Chin, et al.. (2007). Effects of CpG-B ODN on the protein expression profile of swine PBMC. Veterinary Research. 38(6). 795–808. 3 indexed citations
19.
Kuo, Cheng-Chin, Shu‐Mei Liang, & Chi‐Ming Liang. (2006). CpG-B Oligodeoxynucleotide Promotes Cell Survival via Up-regulation of Hsp70 to Increase Bcl-xL and to Decrease Apoptosis-inducing Factor Translocation. Journal of Biological Chemistry. 281(50). 38200–38207. 33 indexed citations
20.
Kuo, Cheng-Chin, Wenting Lin, Chi‐Ming Liang, & Shu‐Mei Liang. (2006). Class I and III Phosphatidylinositol 3′-Kinase Play Distinct Roles in TLR Signaling Pathway. The Journal of Immunology. 176(10). 5943–5949. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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