Charles Schwartz

443 total citations
8 papers, 317 citations indexed

About

Charles Schwartz is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Charles Schwartz has authored 8 papers receiving a total of 317 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Genetics and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Charles Schwartz's work include Congenital heart defects research (3 papers), Genetics and Neurodevelopmental Disorders (2 papers) and RNA modifications and cancer (1 paper). Charles Schwartz is often cited by papers focused on Congenital heart defects research (3 papers), Genetics and Neurodevelopmental Disorders (2 papers) and RNA modifications and cancer (1 paper). Charles Schwartz collaborates with scholars based in United States, Belgium and Norway. Charles Schwartz's co-authors include Lisbeth Tranebjærg, Giovanni Neri, J. Fernando Arena, Pietro Chiurazzi, Patrick J. Willems, Kristel De Boulle, Connie Schrander‐Stumpel, Robin M. Winter, Lieve Vits and Bernhard Korn and has published in prestigious journals such as Nature Genetics, Genomics and American Journal of Medical Genetics.

In The Last Decade

Charles Schwartz

8 papers receiving 315 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Charles Schwartz United States 6 185 150 83 41 32 8 317
Kendall Jensen United States 5 233 1.3× 71 0.5× 91 1.1× 10 0.2× 19 0.6× 7 333
Fernando Arena United States 7 352 1.9× 171 1.1× 57 0.7× 60 1.5× 34 1.1× 8 477
Emma Bedoukian United States 13 238 1.3× 119 0.8× 29 0.3× 61 1.5× 16 0.5× 31 375
Daniel Franjic United States 5 233 1.3× 103 0.7× 37 0.4× 11 0.3× 17 0.5× 5 358
Antonio Vitobello France 13 446 2.4× 212 1.4× 79 1.0× 10 0.2× 18 0.6× 36 595
Jaime L. Sabel United States 6 317 1.7× 57 0.4× 90 1.1× 55 1.3× 13 0.4× 6 474
Leanne Moynihan United Kingdom 7 111 0.6× 71 0.5× 20 0.2× 34 0.8× 5 0.2× 7 226
Margaret R. Starostik United States 7 340 1.8× 102 0.7× 66 0.8× 17 0.4× 48 1.5× 10 502
J. N. Beech United Kingdom 9 186 1.0× 40 0.3× 158 1.9× 36 0.9× 52 1.6× 9 371
Abigail L. D. Tadenev United States 11 284 1.5× 98 0.7× 76 0.9× 120 2.9× 7 0.2× 21 426

Countries citing papers authored by Charles Schwartz

Since Specialization
Citations

This map shows the geographic impact of Charles Schwartz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles Schwartz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles Schwartz more than expected).

Fields of papers citing papers by Charles Schwartz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles Schwartz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles Schwartz. The network helps show where Charles Schwartz may publish in the future.

Co-authorship network of co-authors of Charles Schwartz

This figure shows the co-authorship network connecting the top 25 collaborators of Charles Schwartz. A scholar is included among the top collaborators of Charles Schwartz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Charles Schwartz. Charles Schwartz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Hansen, Gwenn M., et al.. (1999). A Mouse Chromosome 19 Genetic Map Including theLvis1Viral Insertion Site. Genomics. 56(2). 228–231. 3 indexed citations
2.
Sossey‐Alaoui, Khalid, Jennifer A. Lyon, Fatima Abidi, et al.. (1999). Molecular Cloning and Characterization of TRPC5 (HTRP5), the Human Homologue of a Mouse Brain Receptor-Activated Capacitative Ca2+ Entry Channel. Genomics. 60(3). 330–340. 61 indexed citations
3.
Chiurazzi, Pietro, et al.. (1996). XLMRgenes: Update 1996. American Journal of Medical Genetics. 64(1). 147–157. 78 indexed citations
4.
Tranebjærg, Lisbeth, Herbert A. Lubs, Martine Borghgraef, et al.. (1996). Seventh international workshop on the fragile X and X-linked mental retardation. American Journal of Medical Genetics. 64(1). 1–14. 8 indexed citations
5.
Stevenson, Roger E., et al.. (1995). Split foot and developmental retardation associated with a deletion of three microsatellite markers in 7q21.2‐q22.1. Clinical Genetics. 47(2). 90–95. 26 indexed citations
6.
Vits, Lieve, Guy Van Camp, Paul Coucke, et al.. (1994). MASA syndrome is due to mutations in the neural cell adhesion gene L1CAM. Nature Genetics. 7(3). 408–413. 110 indexed citations
7.
Kaufman, Kenneth M., James Snider, Nigel K. Spurr, Charles Schwartz, & James M. Sodetz. (1989). Chromosomal assignment of genes encoding the α, β, and γ subunits of human complement protein C8: Identification of a close physical linkage between the α and the β loci. Genomics. 5(3). 475–480. 30 indexed citations
8.
Schwartz, Charles. (1957). Theory of Hyperfine Structure. Physical Review. 106(6). 1371–1371. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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