Charles Mackay

452 citations
16 papers · 333 · h-index 10

Impact in

    • Ion Channels and Receptors
    • Nitric Oxide and Endothelin Effects
    • Asthma and respiratory diseases
    • Erythrocyte Function and Pathophysiology

Papers in

    • Protein Kinase Regulation and GTPase Signaling 3
    • Ion channel regulation and function 2
    • Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2

Charles Mackay

16 papers receiving 330 citations

Peers

Charles Mackay
Comparison fields: 5 of 75
  • Sensory Systems 23
  • Physiology 95
  • Immunology and Allergy 21
  • Biochemistry 25
  • Immunology 56
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Citations per field
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Citations per year

Countries citing papers authored by Charles Mackay

Since Specialization
Citations

This map shows the geographic impact of Charles Mackay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles Mackay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles Mackay more than expected).

Fields of papers citing papers by Charles Mackay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles Mackay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles Mackay. The network helps show where Charles Mackay may publish in the future.

Co-authors

The 25 scholars most cited alongside Charles Mackay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Charles Mackay Line = papers co-authored together Charles Mackay links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 201467
2 202042
3 201741
4 201838
5 199534
6 201926
7 202424
8 201723
9 201711
10 202311
11 20159
12 20233
13 20241
14 20061
15 20211
16 19931

About Charles Mackay

Charles Mackay is a scholar working on Molecular Biology, Immunology, Physiology, Cardiology and Cardiovascular Medicine and Sensory Systems, having authored 16 papers that have together received 333 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (3 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Genetic and Kidney Cyst Diseases (2 papers), Heart Failure Treatment and Management (2 papers), Cell Adhesion Molecules Research (2 papers), Ion channel regulation and function (2 papers), Eicosanoids and Hypertension Pharmacology (2 papers) and Nitric Oxide and Endothelin Effects (2 papers). The work is most often cited by research in Sensory Systems (23 citations), Physiology (95 citations), Immunology and Allergy (21 citations), Biochemistry (25 citations) and Immunology (56 citations). Charles Mackay has collaborated with scholars based in United States, United Kingdom and Australia. Frequent co-authors include Greg A. Knock, Yasin Shaifta, Jeremy Ward, Jonathan H. Jaggar, M. Dennis Leo, Alejandro Mata‐Daboin, Raquibul Hasan, Simon Bulley, Carlos Fernández‐Peña and Vladimir Snetkov. Their work appears in journals such as The Journal of Physiology, eLife, Proceedings of the National Academy of Sciences, Journal of Hypertension and American Journal of Physiology-Lung Cellular and Molecular Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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