Celina Genschel

928 total citations · 1 hit paper
7 papers, 450 citations indexed

About

Celina Genschel is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Celina Genschel has authored 7 papers receiving a total of 450 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Cancer Research and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Celina Genschel's work include MicroRNA in disease regulation (5 papers), RNA Interference and Gene Delivery (3 papers) and Signaling Pathways in Disease (2 papers). Celina Genschel is often cited by papers focused on MicroRNA in disease regulation (5 papers), RNA Interference and Gene Delivery (3 papers) and Signaling Pathways in Disease (2 papers). Celina Genschel collaborates with scholars based in Germany, Austria and United Kingdom. Celina Genschel's co-authors include Thomas Thum, Sándor Bátkai, Steffen Rump, Janika Viereck, Carmen Theek, Jörg Täubel, Arthur A. Levin, Ulrike Lorch, Wilfried Hauke and Johann Bauersachs and has published in prestigious journals such as Circulation, Circulation Research and Scientific Reports.

In The Last Decade

Celina Genschel

6 papers receiving 449 citations

Hit Papers

Novel antisense therapy targeting microRNA-132 in patient... 2020 2026 2022 2024 2020 50 100 150 200 250

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Celina Genschel Germany 3 328 266 116 35 26 7 450
Yuting Zhuang China 9 336 1.0× 224 0.8× 112 1.0× 66 1.9× 20 0.8× 10 450
Shenjian Luo China 10 311 0.9× 341 1.3× 149 1.3× 41 1.2× 18 0.7× 14 522
Annette Just Germany 11 330 1.0× 265 1.0× 112 1.0× 61 1.7× 35 1.3× 22 499
Ping Pang China 9 310 0.9× 133 0.5× 34 0.3× 53 1.5× 25 1.0× 18 377
Meredith Whitehead United Kingdom 5 212 0.6× 175 0.7× 43 0.4× 32 0.9× 57 2.2× 11 352
Ge Mang China 8 289 0.9× 157 0.6× 80 0.7× 34 1.0× 85 3.3× 8 432
Yujia Yue United States 5 265 0.8× 131 0.5× 115 1.0× 56 1.6× 41 1.6× 7 383
Chunyu Zeng China 7 473 1.4× 407 1.5× 58 0.5× 36 1.0× 104 4.0× 10 638
Steffen Rump Germany 4 303 0.9× 227 0.9× 119 1.0× 29 0.8× 25 1.0× 8 420
Xiaodong Pan China 8 161 0.5× 82 0.3× 70 0.6× 69 2.0× 28 1.1× 14 298

Countries citing papers authored by Celina Genschel

Since Specialization
Citations

This map shows the geographic impact of Celina Genschel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Celina Genschel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Celina Genschel more than expected).

Fields of papers citing papers by Celina Genschel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Celina Genschel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Celina Genschel. The network helps show where Celina Genschel may publish in the future.

Co-authorship network of co-authors of Celina Genschel

This figure shows the co-authorship network connecting the top 25 collaborators of Celina Genschel. A scholar is included among the top collaborators of Celina Genschel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Celina Genschel. Celina Genschel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Bátkai, Sándor, Andreas Spannbauer, Janika Viereck, et al.. (2025). MiR-132 inhibition improves myocardial strain in a large animal model of chronic left ventricular adverse remodelling. PubMed. 3(2). qyaf088–qyaf088.
2.
Bátkai, Sándor, Celina Genschel, Janika Viereck, et al.. (2023). Abstract 15057: miR-132 Inhibitor CDR 132L Attenuates Cardiac Remodeling by Exhibiting Anti-Fibrotic Effects. Circulation. 148(Suppl_1). 1 indexed citations
3.
Täubel, Jörg, Wilfried Hauke, Steffen Rump, et al.. (2021). Novel antisense therapy targeting microRNA-132 in patients with heart failure. European Heart Journal. 42(Supplement_1). 1 indexed citations
4.
Täubel, Jörg, Wilfried Hauke, Steffen Rump, et al.. (2021). Abstract 114: Safety And Efficacy Of CDR132L, A Novel Antisense Therapeutic Which Targets MicroRNA-132 In Heart Failure Patients. Circulation Research. 129(Suppl_1). 1 indexed citations
5.
Täubel, Jörg, Wilfried Hauke, Steffen Rump, et al.. (2020). Novel antisense therapy targeting microRNA-132 in patients with heart failure: results of a first-in-human Phase 1b randomized, double-blind, placebo-controlled study. European Heart Journal. 42(2). 178–188. 266 indexed citations breakdown →
6.
Bátkai, Sándor, Celina Genschel, Janika Viereck, et al.. (2020). CDR132L improves systolic and diastolic function in a large animal model of chronic heart failure. European Heart Journal. 42(2). 192–201. 108 indexed citations
7.
Kölling, Malte, Celina Genschel, Tamás Kaucsár, et al.. (2018). Hypoxia-induced long non-coding RNA Malat1 is dispensable for renal ischemia/reperfusion-injury. Scientific Reports. 8(1). 3438–3438. 73 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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