Cécile Otten

846 total citations
18 papers, 438 citations indexed

About

Cécile Otten is a scholar working on Molecular Biology, Neurology and Cell Biology. According to data from OpenAlex, Cécile Otten has authored 18 papers receiving a total of 438 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 4 papers in Neurology and 4 papers in Cell Biology. Recurrent topics in Cécile Otten's work include Congenital heart defects research (6 papers), Vascular Malformations Diagnosis and Treatment (4 papers) and Intracranial Aneurysms: Treatment and Complications (3 papers). Cécile Otten is often cited by papers focused on Congenital heart defects research (6 papers), Vascular Malformations Diagnosis and Treatment (4 papers) and Intracranial Aneurysms: Treatment and Complications (3 papers). Cécile Otten collaborates with scholars based in Germany, Croatia and United States. Cécile Otten's co-authors include Salim Abdelilah‐Seyfried, Stefan Rohr, Brian A. Link, Jingjing Zhang, Shuang Cui, Christian Piehl, Annabel Christ, Thomas E. Willnow, Ingolf E. Blasig and Jörg Piontek and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Circulation Research.

In The Last Decade

Cécile Otten

16 papers receiving 435 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Cécile Otten Germany	12	315	153	50	44	39	18	438
Filomena Ricciardi Germany	7	238 0.8×	68 0.4×	56 1.1×	16 0.4×	92 2.4×	8	364
Amrita Mandal United States	9	349 1.1×	73 0.5×	41 0.8×	24 0.5×	28 0.7×	13	464
Andy Yang United States	9	195 0.6×	71 0.5×	25 0.5×	13 0.3×	15 0.4×	13	323
Stephanie J. Munger United States	9	299 0.9×	72 0.5×	12 0.2×	29 0.7×	18 0.5×	13	544
C.M. Chen United Kingdom	13	613 1.9×	122 0.8×	74 1.5×	22 0.5×	59 1.5×	18	730
José Wojnacki Argentina	11	233 0.7×	209 1.4×	30 0.6×	18 0.4×	25 0.6×	19	429
AM González United Kingdom	6	227 0.7×	61 0.4×	57 1.1×	33 0.8×	52 1.3×	10	431
Jennifer Lévy United States	10	415 1.3×	269 1.8×	37 0.7×	32 0.7×	142 3.6×	11	619
Anna‐Kaisa Anttonen Finland	11	280 0.9×	142 0.9×	24 0.5×	17 0.4×	31 0.8×	18	457
Kamron N. Khan United Kingdom	17	603 1.9×	87 0.6×	36 0.7×	27 0.6×	36 0.9×	36	884

Countries citing papers authored by Cécile Otten

Since Specialization

Citations

This map shows the geographic impact of Cécile Otten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cécile Otten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cécile Otten more than expected).

Fields of papers citing papers by Cécile Otten

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cécile Otten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cécile Otten. The network helps show where Cécile Otten may publish in the future.

Co-authorship network of co-authors of Cécile Otten

This figure shows the co-authorship network connecting the top 25 collaborators of Cécile Otten. A scholar is included among the top collaborators of Cécile Otten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cécile Otten. Cécile Otten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

1.

Otten, Cécile, et al.. (2024). Tyrosyl-DNA phosphodiesterase 2 (Tdp2) repairs DNA-protein crosslinks and protects against double strand breaks in vivo. Frontiers in Cell and Developmental Biology. 12. 1394531–1394531.

2.

Boulday, Gwénola, et al.. (2023). Impaired retinoic acid signaling in cerebral cavernous malformations. Scientific Reports. 13(1). 5572–5572.

3.

Otten, Cécile, et al.. (2023). Tyrosyl-DNA phosphodiesterase 1 (TDP1) and SPRTN protease repair histone 3 and topoisomerase 1 DNA–protein crosslinks in vivo. Open Biology. 13(10). 230113–230113. 6 indexed citations

4.

Mihaljević, Ivan, et al.. (2022). Stage‐dependent localization of F‐actin and Na⁺/K⁺‐ATPase in zebrafish embryos detected using optimized cryosectioning immunostaining protocol. Microscopy Research and Technique. 86(3). 294–310. 1 indexed citations

5.

Arnould, Minh, Cécile Otten, Salim Abdelilah‐Seyfried, et al.. (2020). Novel Chronic Mouse Model of Cerebral Cavernous Malformations. Stroke. 51(4). 1272–1278. 18 indexed citations

6.

Otten, Cécile, Tina C. Wan, John A. Auchampach, et al.. (2020). Llgl1 regulates zebrafish cardiac development by mediating Yap stability in cardiomyocytes. Development. 147(16). 8 indexed citations

7.

Otten, Cécile, et al.. (2019). Blood Flow Suppresses Vascular Anomalies in a Zebrafish Model of Cerebral Cavernous Malformations. Circulation Research. 125(10). e43–e54. 19 indexed citations

8.

Otten, Cécile, et al.. (2018). Heg1 and Ccm1/2 proteins control endocardial mechanosensitivity during zebrafish valvulogenesis. eLife. 7. 49 indexed citations

9.

Lombardo, Verónica A., Cécile Otten, & Salim Abdelilah‐Seyfried. (2015). Large-scale Zebrafish Embryonic Heart Dissection for Transcriptional Analysis. Journal of Visualized Experiments. 52087–52087. 15 indexed citations

10.

Lombardo, Verónica A., Cécile Otten, & Salim Abdelilah‐Seyfried. (2015). Large-scale Zebrafish Embryonic Heart Dissection for Transcriptional Analysis. Journal of Visualized Experiments. 1 indexed citations

11.

Wieffer, Marnix, Elena Cibrián–Uhalte, York Posor, et al.. (2013). PI4K2β/AP-1-Based TGN-Endosomal Sorting Regulates Wnt Signaling. Current Biology. 23(21). 2185–2190. 31 indexed citations

12.

Otten, Cécile & Salim Abdelilah‐Seyfried. (2013). Laser-inflicted Injury of Zebrafish Embryonic Skeletal Muscle. Journal of Visualized Experiments. e4351–e4351. 11 indexed citations

13.

Veerkamp, Justus, Franziska Rudolph, Zoltán Cseresnyés, et al.. (2013). Unilateral Dampening of Bmp Activity by Nodal Generates Cardiac Left-Right Asymmetry. Developmental Cell. 24(6). 660–667. 43 indexed citations

14.

Otten, Cécile & Salim Abdelilah‐Seyfried. (2013). Laser-inflicted Injury of Zebrafish Embryonic Skeletal Muscle. Journal of Visualized Experiments. 11 indexed citations

15.

Otten, Cécile, Peter F. M. van der Ven, Sandeep Paul, et al.. (2012). Xirp Proteins Mark Injured Skeletal Muscle in Zebrafish. PLoS ONE. 7(2). e31041–e31041. 31 indexed citations

16.

Zhang, Jingjing, Jörg Piontek, Hartwig Wolburg, et al.. (2010). Establishment of a neuroepithelial barrier by Claudin5a is essential for zebrafish brain ventricular lumen expansion. Proceedings of the National Academy of Sciences. 107(4). 1425–1430. 77 indexed citations

17.

Rohr, Stefan, Cécile Otten, & Salim Abdelilah‐Seyfried. (2008). Asymmetric Involution of the Myocardial Field Drives Heart Tube Formation in Zebrafish. Circulation Research. 102(2). e12–9. 77 indexed citations

18.

Cui, Shuang, Cécile Otten, Stefan Rohr, Salim Abdelilah‐Seyfried, & Brian A. Link. (2007). Analysis of aPKCλ and aPKCζ reveals multiple and redundant functions during vertebrate retinogenesis. Molecular and Cellular Neuroscience. 34(3). 431–444. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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