Caterina Barresi

1.2k total citations
16 papers, 841 citations indexed

About

Caterina Barresi is a scholar working on Epidemiology, Molecular Biology and Dermatology. According to data from OpenAlex, Caterina Barresi has authored 16 papers receiving a total of 841 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Epidemiology, 5 papers in Molecular Biology and 4 papers in Dermatology. Recurrent topics in Caterina Barresi's work include Autophagy in Disease and Therapy (5 papers), Wound Healing and Treatments (3 papers) and HIV Research and Treatment (2 papers). Caterina Barresi is often cited by papers focused on Autophagy in Disease and Therapy (5 papers), Wound Healing and Treatments (3 papers) and HIV Research and Treatment (2 papers). Caterina Barresi collaborates with scholars based in Austria, United Kingdom and Italy. Caterina Barresi's co-authors include Erwin Tschachler, Leopold Eckhart, Heidemarie Rossiter, Michael Mildner, Флориан Грубер, Veronika Mlitz, Dagmar Födinger, Minoo Ghannadan, Caroline Stremnitzer and Sanja Kežić and has published in prestigious journals such as PLoS ONE, Cancer Research and Journal of Virology.

In The Last Decade

Caterina Barresi

16 papers receiving 819 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Caterina Barresi Austria	13	306	268	210	175	109	16	841
Ryan F.L. O’Shaughnessy United Kingdom	19	281 0.9×	452 1.7×	277 1.3×	161 0.9×	184 1.7×	39	964
Petra Ovaere Belgium	5	304 1.0×	345 1.3×	222 1.1×	82 0.5×	122 1.1×	6	796
Marco Averbeck Germany	18	203 0.7×	337 1.3×	295 1.4×	55 0.3×	253 2.3×	33	1.1k
Patrick A.M. Jansen Netherlands	16	270 0.9×	357 1.3×	115 0.5×	60 0.3×	230 2.1×	33	904
Lisa E. Shaw Austria	12	234 0.8×	284 1.1×	96 0.5×	179 1.0×	241 2.2×	22	863
Jun Muto Japan	17	337 1.1×	499 1.9×	237 1.1×	122 0.7×	525 4.8×	53	1.4k
Seung‐Chul Lee South Korea	19	524 1.7×	175 0.7×	119 0.6×	156 0.9×	86 0.8×	85	1.1k
Mieke Bergers Netherlands	21	305 1.0×	398 1.5×	278 1.3×	68 0.4×	337 3.1×	34	1.2k
Norihiro Fujimoto Japan	15	195 0.6×	290 1.1×	216 1.0×	117 0.7×	49 0.4×	35	915
Dagmar Födinger Austria	16	321 1.0×	323 1.2×	135 0.6×	87 0.5×	359 3.3×	28	1.2k

Countries citing papers authored by Caterina Barresi

Since Specialization

Citations

This map shows the geographic impact of Caterina Barresi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Caterina Barresi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Caterina Barresi more than expected).

Fields of papers citing papers by Caterina Barresi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Caterina Barresi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Caterina Barresi. The network helps show where Caterina Barresi may publish in the future.

Co-authorship network of co-authors of Caterina Barresi

This figure shows the co-authorship network connecting the top 25 collaborators of Caterina Barresi. A scholar is included among the top collaborators of Caterina Barresi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Caterina Barresi. Caterina Barresi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Barresi, Caterina, Heidemarie Rossiter, Maria Buchberger, et al.. (2022). Inactivation of Autophagy in Keratinocytes Reduces Tumor Growth in Mouse Models of Epithelial Skin Cancer. Cells. 11(22). 3691–3691. 5 indexed citations

Koenig, Ulrich, Horst Robenek, Caterina Barresi, et al.. (2019). Cell death induced autophagy contributes to terminal differentiation of skin and skin appendages. Autophagy. 16(5). 932–945. 44 indexed citations

Rothbauer, Mario, et al.. (2015). Automated, Miniaturized, and Integrated Quality Control-on-Chip (QC-on-a-Chip) for Cell-Based Cancer Therapy Applications. Frontiers in Materials. 2. 24 indexed citations

Koenig, Ulrich, Manfred Fobker, Barbara Lengauer, et al.. (2014). Autophagy facilitates secretion and protects against degeneration of the Harderian gland. Autophagy. 11(2). 298–313. 11 indexed citations

Zhang, Chengfeng, Флориан Грубер, Michael Mildner, et al.. (2014). Suppression of Autophagy Dysregulates the Antioxidant Response and Causes Premature Senescence of Melanocytes. Journal of Investigative Dermatology. 135(5). 1348–1357. 94 indexed citations

Rossiter, Heidemarie, Ulrich König, Caterina Barresi, et al.. (2013). Epidermal keratinocytes form a functional skin barrier in the absence of Atg7 dependent autophagy. Journal of Dermatological Science. 71(1). 67–75. 53 indexed citations

Mildner, Michael, Stefan Hacker, Thomas Haider, et al.. (2013). Secretome of Peripheral Blood Mononuclear Cells Enhances Wound Healing. PLoS ONE. 8(3). e60103–e60103. 49 indexed citations

Barresi, Caterina, Caroline Stremnitzer, Veronika Mlitz, et al.. (2010). Increased Sensitivity of Histidinemic Mice to UVB Radiation Suggests a Crucial Role of Endogenous Urocanic Acid in Photoprotection. Journal of Investigative Dermatology. 131(1). 188–194. 89 indexed citations

Mildner, Michael, Jiang Jin, Leopold Eckhart, et al.. (2010). Knockdown of Filaggrin Impairs Diffusion Barrier Function and Increases UV Sensitivity in a Human Skin Model. Journal of Investigative Dermatology. 130(9). 2286–2294. 213 indexed citations

10.

Rossiter, Heidemarie, Caterina Barresi, Minoo Ghannadan, et al.. (2007). Inactivation of VEGF in mammary gland epithelium severely compromises mammary gland development and function. The FASEB Journal. 21(14). 3994–4004. 56 indexed citations

11.

Fischer, Heinz, Heidemarie Rossiter, Minoo Ghannadan, et al.. (2005). Caspase-14 but not caspase-3 is processed during the development of fetal mouse epidermis. Differentiation. 73(8). 406–413. 42 indexed citations

12.

Rossiter, Heidemarie, Caterina Barresi, Johannes Pammer, et al.. (2004). Loss of Vascular Endothelial Growth Factor A Activity in Murine Epidermal Keratinocytes Delays Wound Healing and Inhibits Tumor Formation. Cancer Research. 64(10). 3508–3516. 102 indexed citations

13.

Borgia, Maria Clotilde, et al.. (2001). Further evidence against the implication of active cytomegalovirus infection in vascular atherosclerotic diseases. Atherosclerosis. 157(2). 457–462. 13 indexed citations

14.

Rossiter, Heidemarie, et al.. (2001). Lack of vascular endothelial cell growth factor (VEGF) in murine epidermal keratinocytes results in retardation of angiogenesis-dependent processes in the skin. 1 indexed citations

15.

Capobianchi, Maria Rosaria, Caterina Barresi, Paola Borghi, et al.. (1997). Human immunodeficiency virus type 1 gp120 stimulates cytomegalovirus replication in monocytes: possible role of endogenous interleukin-8. Journal of Virology. 71(2). 1591–1597. 32 indexed citations

16.

Dianzani, F., et al.. (1996). Interferon Gamma Stimulates Cell-Mediated Transmission of HIV Type 1 from Abortively Infected Endothelial Cells. AIDS Research and Human Retroviruses. 12(7). 621–627. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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