Catena Kresbach

461 total citations
13 papers, 188 citations indexed

About

Catena Kresbach is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Catena Kresbach has authored 13 papers receiving a total of 188 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Genetics and 3 papers in Surgery. Recurrent topics in Catena Kresbach's work include Glioma Diagnosis and Treatment (6 papers), Hedgehog Signaling Pathway Studies (4 papers) and Testicular diseases and treatments (3 papers). Catena Kresbach is often cited by papers focused on Glioma Diagnosis and Treatment (6 papers), Hedgehog Signaling Pathway Studies (4 papers) and Testicular diseases and treatments (3 papers). Catena Kresbach collaborates with scholars based in Germany, Austria and United States. Catena Kresbach's co-authors include Ulrich Schüller, Oliver Kretz, Soeren S. Lienkamp, Tobias B. Huber, Sebastian J. Arnold, Gerd Walz, Hannes Engel, Michael M. Kaminski, Florian Grahammer and Stefan Rutkowski and has published in prestigious journals such as Nature Cell Biology, Development and British Journal of Cancer.

In The Last Decade

Catena Kresbach

11 papers receiving 186 citations

Peers

Catena Kresbach
Miguel A. Jiménez United States
Jeremy Klein United States
Deanne King United States
Mao Takei Japan
Abigail Knecht United States
Subreena Simrick United Kingdom
Catena Kresbach
Citations per year, relative to Catena Kresbach Catena Kresbach (= 1×) peers Irati Romero‐Garmendia

Countries citing papers authored by Catena Kresbach

Since Specialization
Citations

This map shows the geographic impact of Catena Kresbach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Catena Kresbach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Catena Kresbach more than expected).

Fields of papers citing papers by Catena Kresbach

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Catena Kresbach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Catena Kresbach. The network helps show where Catena Kresbach may publish in the future.

Co-authorship network of co-authors of Catena Kresbach

This figure shows the co-authorship network connecting the top 25 collaborators of Catena Kresbach. A scholar is included among the top collaborators of Catena Kresbach based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Catena Kresbach. Catena Kresbach is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Bremmer, Felix, Gereon Poschmann, Catena Kresbach, et al.. (2024). Assessing the risk to develop a growing teratoma syndrome based on molecular and epigenetic subtyping as well as novel secreted biomarkers. Cancer Letters. 585. 216673–216673. 2 indexed citations
2.
Skowron, Margaretha A., Gereon Poschmann, Patrick Petzsch, et al.. (2024). Molecular and epigenetic ex vivo profiling of testis cancer-associated fibroblasts and their interaction with germ cell tumor cells and macrophages. Matrix Biology. 132. 10–23. 3 indexed citations
3.
Kresbach, Catena, Denise Obrecht, Katja von Hoff, et al.. (2024). Clinically relevant molecular hallmarks of PFA ependymomas display intratumoral heterogeneity and correlate with tumor morphology. Acta Neuropathologica. 147(1). 23–23. 1 indexed citations
5.
Schoof, Melanie, et al.. (2023). MYC overexpression and SMARCA4 loss cooperate to drive medulloblastoma formation in mice. Acta Neuropathologica Communications. 11(1). 174–174. 5 indexed citations
6.
Kresbach, Catena, Mario M. Dorostkar, Patrick N. Harter, et al.. (2023). Molecular refinement of pilocytic astrocytoma in adult patients. Neuropathology and Applied Neurobiology. 50(1).
7.
Kresbach, Catena, et al.. (2023). Intraventricular SHH inhibition proves efficient in SHH medulloblastoma mouse model and prevents systemic side effects. Neuro-Oncology. 26(4). 609–622. 3 indexed citations
8.
Kresbach, Catena, et al.. (2022). MODL-03. Establishment of intraventricular Shh inhibition as a therapeutic option for young patients with medulloblastoma. Neuro-Oncology. 24(Supplement_1). i168–i168.
9.
Kresbach, Catena, et al.. (2022). Updates in the classification of ependymal neoplasms: The 2021 WHO Classification and beyond. Brain Pathology. 32(4). e13068–e13068. 40 indexed citations
10.
Kresbach, Catena, et al.. (2021). EXTH-70. ESTABLISHMENT OF INTRAVENTRICULAR SHH INHIBITION AS A THERAPEUTIC OPTION IN YOUNG PATIENTS WITH MEDULLOBLASTOMA. Neuro-Oncology. 23(Supplement_6). vi179–vi179. 1 indexed citations
12.
Schoof, Melanie, Michael Spohn, Catena Kresbach, et al.. (2021). Brahma-related gene 1 has time-specific roles during brain and eye development. Development. 148(10). 11 indexed citations
13.
Kaminski, Michael M., Catena Kresbach, Hannes Engel, et al.. (2016). Direct reprogramming of fibroblasts into renal tubular epithelial cells by defined transcription factors. Nature Cell Biology. 18(12). 1269–1280. 99 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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