This map shows the geographic impact of Carlo Rivolta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carlo Rivolta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carlo Rivolta more than expected).
This network shows the impact of papers produced by Carlo Rivolta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carlo Rivolta. The network helps show where Carlo Rivolta may publish in the future.
Co-authorship network of co-authors of Carlo Rivolta
This figure shows the co-authorship network connecting the top 25 collaborators of Carlo Rivolta.
A scholar is included among the top collaborators of Carlo Rivolta based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Carlo Rivolta. Carlo Rivolta is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Peter, Virginie G., et al.. (2020). Exploring the genetic landscape of inherited retinal diseases in North-Western Pakistan reveals a high degree of autozygosity and prevalent founder mutations. Investigative Ophthalmology & Visual Science. 61(7). 2385–2385.1 indexed citations
9.
Nikopoulos, Konstantinos, Pietro Farinelli, Béryl Royer‐Bertrand, et al.. (2016). Whole exome sequencing reveals CEP78 as a novel disease gene for cone-rod dystrophy. Investigative Ophthalmology & Visual Science. 57(12).1 indexed citations
Saqib, Muhammad Arif Nadeem, Ehsan Ullah, Falak Sher Khan, et al.. (2014). Homozygosity Mapping and Disease Genes Screening in Pakistani Families with Inherited Retinal Dystrophies. Investigative Ophthalmology & Visual Science. 55(13). 3292–3292.1 indexed citations
Ransijn, Adriana, Giulia Venturini, Silvio Alessandro Di Gioia, et al.. (2012). FAM161A Mutations In Patients With Early-onset Retinitis Pigmentosa In The United States. Investigative Ophthalmology & Visual Science. 53(14). 4545–4545.1 indexed citations
Benaglio, Paola, et al.. (2011). Next Generation Sequencing Of Pooled DNA From A Large Cohort Of Patients Reveals New SNRNP200 Mutations Associated With Retinitis Pigmentosa. Investigative Ophthalmology & Visual Science. 52(14). 3316–3316.1 indexed citations
16.
Tanačković, Goranka, Adriana Ransijn, P Thibault, et al.. (2011). Generalized Defects In Spliceosome Composition And Pre-mRNA Splicing Are Associated With Retinitis Pigmentosa In Humans. Investigative Ophthalmology & Visual Science. 52(14). 1639–1639.1 indexed citations
17.
Rivolta, Carlo, Terri L. McGee, Nicholas M. Wade, et al.. (2009). A Single-Base Substitution Within an Intronic Repetitive Element in PRPF31 Causes Dominant Retinitis Pigmentosa With Reduced Penetrance. Investigative Ophthalmology & Visual Science. 50(13). 2318–2318.6 indexed citations
18.
Rivolta, Carlo, et al.. (2003). All-Exon Screen of the Ush2a Gene in Recessive Nonsyndromic Retinitis Pigmentosa and Usher Syndrome Type II. Investigative Ophthalmology & Visual Science. 44(13). 2315–2315.1 indexed citations
Rivolta, Carlo, et al.. (2002). Mutations In The USH2A Gene Are A Frequent Cause Of Recessive Nonsyndromic Retinitis Pigmentosa As Well As Usher Syndrome Type II. Investigative Ophthalmology & Visual Science. 43(13). 795–795.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.