Carleton R. Sage

1.2k total citations
26 papers, 692 citations indexed

About

Carleton R. Sage is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Carleton R. Sage has authored 26 papers receiving a total of 692 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 6 papers in Cellular and Molecular Neuroscience and 6 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Carleton R. Sage's work include Receptor Mechanisms and Signaling (9 papers), Diabetes Treatment and Management (6 papers) and Neurotransmitter Receptor Influence on Behavior (5 papers). Carleton R. Sage is often cited by papers focused on Receptor Mechanisms and Signaling (9 papers), Diabetes Treatment and Management (6 papers) and Neurotransmitter Receptor Influence on Behavior (5 papers). Carleton R. Sage collaborates with scholars based in United States and China. Carleton R. Sage's co-authors include Ashley Davis, K Farrell, Thomas J. Stout, Robert M. Stroud, Graeme Semple, Jeremy G. Richman, Ruoping Chen, Nigel R. A. Beeley, Anwar Ghuloum and Ajay N. Jain and has published in prestigious journals such as Science, Nucleic Acids Research and Journal of Molecular Biology.

In The Last Decade

Carleton R. Sage

26 papers receiving 671 citations

Peers

Carleton R. Sage
Beth A. Fleck United States
Dahabada H. J. Lopes United States
James A. Hebda United States
Lydia Young United Kingdom
James Fossetta United States
Dev Trivedi United States
Carleton R. Sage
Citations per year, relative to Carleton R. Sage Carleton R. Sage (= 1×) peers Marianna Tatarek‐Nossol

Countries citing papers authored by Carleton R. Sage

Since Specialization
Citations

This map shows the geographic impact of Carleton R. Sage's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carleton R. Sage with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carleton R. Sage more than expected).

Fields of papers citing papers by Carleton R. Sage

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Carleton R. Sage. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carleton R. Sage. The network helps show where Carleton R. Sage may publish in the future.

Co-authorship network of co-authors of Carleton R. Sage

This figure shows the co-authorship network connecting the top 25 collaborators of Carleton R. Sage. A scholar is included among the top collaborators of Carleton R. Sage based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carleton R. Sage. Carleton R. Sage is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gaidarov, Ibragim, John Frazer, Xiaohua Chen, et al.. (2025). Mechanisms of constitutive and agonist-induced 5-HT2B internalization, persistent endosomal signaling and paradoxical regulation of agonist pharmacology. Cellular Signalling. 131. 111769–111769. 2 indexed citations
2.
Gonçalves, L.M., et al.. (2024). BioPrint Meets the AI Age: Development of Artificial Intelligence-Based ADMET Models for the Drug-Discovery Platform SAFIRE. Future Medicinal Chemistry. 16(7). 587–599. 2 indexed citations
3.
Ren, Albert, Xiuwen Zhu, Konrad Feichtinger, et al.. (2019). Discovery of a lead series of potent benzodiazepine 5-HT2C receptor agonists with high selectivity in functional and binding assays. Bioorganic & Medicinal Chemistry Letters. 30(5). 126929–126929. 3 indexed citations
4.
Schrader, Thomas O., Albert Ren, Konrad Feichtinger, et al.. (2016). Tetrahydroquinoline-based tricyclic amines as potent and selective agonists of the 5-HT2C receptor. Bioorganic & Medicinal Chemistry Letters. 26(24). 5877–5882. 6 indexed citations
5.
Grottick, Andrew J., et al.. (2014). Investigating interactions between phentermine, dexfenfluramine, and 5-HT2C agonists, on food intake in the rat. Psychopharmacology. 232(11). 1973–1982. 10 indexed citations
6.
Semple, Graeme, Albert Ren, Beatriz Fioravanti, et al.. (2011). Discovery of fused bicyclic agonists of the orphan G-protein coupled receptor GPR119 with in vivo activity in rodent models of glucose control. Bioorganic & Medicinal Chemistry Letters. 21(10). 3134–3141. 53 indexed citations
7.
Semple, Graeme, Juerg Lehmann, Albert Ren, et al.. (2011). Discovery of a second generation agonist of the orphan G-protein coupled receptor GPR119 with an improved profile. Bioorganic & Medicinal Chemistry Letters. 22(4). 1750–1755. 39 indexed citations
8.
Sage, Carleton R., et al.. (2011). G-Protein Coupled Receptors Virtual Screening Using Genetic Algorithm Focused Chemical Space. Journal of Chemical Information and Modeling. 51(8). 1754–1761. 5 indexed citations
9.
Boatman, P. Douglas, Thomas O. Schrader, Benjamin Johnson, et al.. (2010). Potent tricyclic pyrazole tetrazole agonists of the nicotinic acid receptor (GPR109a). Bioorganic & Medicinal Chemistry Letters. 20(9). 2797–2800. 23 indexed citations
10.
Skinner, Philip J., Peter J. Webb, Carleton R. Sage, et al.. (2009). 5-N,N-Disubstituted 5-aminopyrazole-3-carboxylic acids are highly potent agonists of GPR109b. Bioorganic & Medicinal Chemistry Letters. 19(15). 4207–4209. 20 indexed citations
11.
Skinner, Philip J., Peter J. Webb, Carleton R. Sage, et al.. (2007). 3-Nitro-4-amino benzoic acids and 6-amino nicotinic acids are highly selective agonists of GPR109b. Bioorganic & Medicinal Chemistry Letters. 17(23). 6619–6622. 24 indexed citations
12.
Beeley, Nigel R. A. & Carleton R. Sage. (2003). GPCRs: an update on structural approaches to drug discovery. 2(1). 19–25. 31 indexed citations
13.
Sage, Carleton R., et al.. (2001). Mutation in the β-Tubulin Signature Motif Suppresses Microtubule GTPase Activity and Dynamics, and Slows Mitosis. Biochemistry. 40(51). 15725–15732. 22 indexed citations
14.
Reyes, C., Carleton R. Sage, Earl Rutenber, et al.. (1998). Inactivity of N229A thymidylate synthase due to water-mediated effects: isolating a late stage in methyl transfer. Journal of Molecular Biology. 284(3). 699–712. 7 indexed citations
15.
Stout, Thomas J., Carleton R. Sage, & Robert M. Stroud. (1998). The additivity of substrate fragments in enzyme–ligand binding. Structure. 6(7). 839–848. 65 indexed citations
16.
Sage, Carleton R., et al.. (1998). D221 in Thymidylate Synthase Controls Conformation Change, and Thereby Opening of the Imidazolidine,. Biochemistry. 37(39). 13893–13901. 27 indexed citations
17.
Sage, Carleton R., Earl Rutenber, Thomas J. Stout, & R. M. Stroud. (1996). An Essential Role for Water in an Enzyme Reaction Mechanism:  The Crystal Structure of the Thymidylate Synthase Mutant E58Q,. Biochemistry. 35(50). 16270–16281. 23 indexed citations
18.
Sage, Carleton R., et al.. (1995). β‐Tubulin mutation suppresses microtubule dynamics in vitro and slows mitosis in vivo. Cell Motility and the Cytoskeleton. 30(4). 285–300. 20 indexed citations
19.
Xia, Ling, Joyce F. Liu, Carleton R. Sage, et al.. (1995). Intronic U14 snoRNAs ofXenopus laevisare located in two different parent genes and can be processed from their introns during early oogenesis. Nucleic Acids Research. 23(23). 4844–4849. 23 indexed citations
20.
Davis, Ashley, Carleton R. Sage, Leslie Wilson, & K Farrell. (1993). Purification and biochemical characterization of tubulin from the budding yeast Saccharomyces cerevisiae. Biochemistry. 32(34). 8823–8835. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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