C. Weiß

850 total citations
16 papers, 599 citations indexed

About

C. Weiß is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Surgery. According to data from OpenAlex, C. Weiß has authored 16 papers receiving a total of 599 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cardiology and Cardiovascular Medicine, 7 papers in Molecular Biology and 4 papers in Surgery. Recurrent topics in C. Weiß's work include Cardiovascular Function and Risk Factors (4 papers), Cardiac Fibrosis and Remodeling (3 papers) and Cardiovascular Disease and Adiposity (2 papers). C. Weiß is often cited by papers focused on Cardiovascular Function and Risk Factors (4 papers), Cardiac Fibrosis and Remodeling (3 papers) and Cardiovascular Disease and Adiposity (2 papers). C. Weiß collaborates with scholars based in Germany, United States and Switzerland. C. Weiß's co-authors include Sebastian J. Buss, Hugo A. Katus, Stefan E. Hardt, Pratima Malekar, Marco Hagenmueller, Johannes Riffel, David Wolf, Raffi Bekeredjian, Seigo Izumo and Florian Bea and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and FEBS Letters.

In The Last Decade

C. Weiß

16 papers receiving 588 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
C. Weiß Germany	12	242	198	112	89	68	16	599
Masatoshi Ishizaki Japan	15	287 1.2×	87 0.4×	72 0.6×	203 2.3×	91 1.3×	58	678
Young Kuk Cho South Korea	14	341 1.4×	149 0.8×	107 1.0×	132 1.5×	53 0.8×	50	736
Andrew J. Waclawik United States	11	232 1.0×	126 0.6×	61 0.5×	100 1.1×	49 0.7×	36	534
Moshe Swissa Israel	17	148 0.6×	448 2.3×	45 0.4×	88 1.0×	69 1.0×	52	696
Beibei Liu China	14	208 0.9×	124 0.6×	104 0.9×	77 0.9×	139 2.0×	45	684
Miguel Carracedo Sweden	18	197 0.8×	165 0.8×	131 1.2×	140 1.6×	86 1.3×	24	753
Barnabas Gellen France	16	231 1.0×	372 1.9×	39 0.3×	97 1.1×	71 1.0×	47	650
Jingquan Zhong China	11	138 0.6×	188 0.9×	41 0.4×	73 0.8×	78 1.1×	55	532
Asako Katsume Japan	11	373 1.5×	157 0.8×	93 0.8×	82 0.9×	262 3.9×	13	806

Countries citing papers authored by C. Weiß

Since Specialization

Citations

This map shows the geographic impact of C. Weiß's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. Weiß with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. Weiß more than expected).

Fields of papers citing papers by C. Weiß

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. Weiß. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. Weiß. The network helps show where C. Weiß may publish in the future.

Co-authorship network of co-authors of C. Weiß

This figure shows the co-authorship network connecting the top 25 collaborators of C. Weiß. A scholar is included among the top collaborators of C. Weiß based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C. Weiß. C. Weiß is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Streit, Marcus R., C. Weiß, Marco Ochs, et al.. (2016). Cardiac Effects of Attenuating Gsα - Dependent Signaling. PLoS ONE. 11(1). e0146988–e0146988. 3 indexed citations

Pinto, Fausto J., Syrus Karsai, C. Weiß, et al.. (2016). Neodymium‐doped yttrium aluminium garnet (Nd: YAG ) 1064‐nm picosecond laser vs. Nd: YAG 1064‐nm nanosecond laser in tattoo removal: a randomized controlled single‐blind clinical trial. British Journal of Dermatology. 176(2). 457–464. 39 indexed citations

Weiß, C., et al.. (2015). The bigger, the better? About the size of decompressive hemicraniectomies. Clinical Neurology and Neurosurgery. 135. 15–21. 30 indexed citations

Kahl, Kai G., Ulrich Schweiger, Matthias Gutberlet, et al.. (2014). Pericardial, intra‐abdominal, and subcutaneous adipose tissue in patients with major depressive disorder. Acta Psychiatrica Scandinavica. 130(2). 137–143. 22 indexed citations

Weiß, C., Marco Ochs, Marco Hagenmueller, et al.. (2013). DYRK2 Negatively Regulates Cardiomyocyte Growth by Mediating Repressor Function of GSK-3β on eIF2Bε. PLoS ONE. 8(9). e70848–e70848. 14 indexed citations

Weiß, C., Marco Ochs, Sebastian J. Buss, et al.. (2010). Activation of PPARγ by pioglitazone does not attenuate left ventricular hypertrophy following aortic banding in rats. Naunyn-Schmiedeberg s Archives of Pharmacology. 381(4). 285–295. 11 indexed citations

Malekar, Pratima, Marco Hagenmueller, Sebastian J. Buss, et al.. (2010). Wnt Signaling Is Critical for Maladaptive Cardiac Hypertrophy and Accelerates Myocardial Remodeling. Hypertension. 55(4). 939–945. 123 indexed citations

Buss, Sebastian J., Johannes Riffel, Pratima Malekar, et al.. (2009). Beneficial Effects of Mammalian Target of Rapamycin Inhibition on Left Ventricular Remodeling After Myocardial Infarction. Journal of the American College of Cardiology. 54(25). 2435–2446. 175 indexed citations

Hagenmueller, Marco, Pratima Malekar, C. Weiß, et al.. (2009). Depletion of mammalian target of rapamycin (mTOR) via siRNA mediated knockdown leads to stabilization of β‐catenin and elicits distinct features of cardiomyocyte hypertrophy. FEBS Letters. 584(1). 74–80. 8 indexed citations

10.

Buss, Sebastian J., David Wolf, Grigorios Korosoglou, et al.. (2009). Myocardial Left Ventricular Dysfunction in Patients with Systemic Lupus Erythematosus: New Insights from Tissue Doppler and Strain Imaging. The Journal of Rheumatology. 37(1). 79–86. 60 indexed citations

11.

Weiß, C., Marcus Egermann, & Peter Bärtsch. (2004). Exercise-induced activation of coagulation in subjects with activated protein C resistance. Blood Coagulation & Fibrinolysis. 15(4). 317–321. 5 indexed citations

12.

Weiss, Thomas G., Dieter Fischer, Dirk Hausmann, & C. Weiß. (2002). Endothelial function in patients with peripheral vascular disease: influence of prostaglandin E1. Prostaglandins Leukotrienes and Essential Fatty Acids. 67(5). 277–281. 18 indexed citations

13.

Weiß, C., et al.. (2000). Hemostasis and fibrinolysis in patients with intermittent claudication: effects of prostaglandin E1. Prostaglandins Leukotrienes and Essential Fatty Acids. 63(5). 271–277. 15 indexed citations

14.

Hack, Volker, C. Weiß, B Friedmann, et al.. (1997). Decreased plasma glutamine level and CD4+ T cell number in response to 8 wk of anaerobic training. American Journal of Physiology-Endocrinology and Metabolism. 272(5). E788–E795. 23 indexed citations

15.

Kinscherf, Ralf, Volker Hack, Thomas Fischbach, et al.. (1996). Low plasma glutamine in combination with high glutamate levels indicate risk for loss of body cell mass in healthy individuals: the effect of N-acetyl-cysteine. Journal of Molecular Medicine. 74(7). 393–400. 41 indexed citations

16.

Weiß, C., Ralf Kinscherf, Stephen J. Roth, et al.. (1995). Lymphocyte Subpopulations and Concentrations of Soluble CD8 and CD4 Antigen After Anaerobic Training. International Journal of Sports Medicine. 16(2). 117–121. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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