C J Epstein

1.8k total citations · 1 hit paper
16 papers, 1.5k citations indexed

About

C J Epstein is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, C J Epstein has authored 16 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Physiology and 4 papers in Genetics. Recurrent topics in C J Epstein's work include Mitochondrial Function and Pathology (4 papers), Down syndrome and intellectual disability research (2 papers) and BRCA gene mutations in cancer (2 papers). C J Epstein is often cited by papers focused on Mitochondrial Function and Pathology (4 papers), Down syndrome and intellectual disability research (2 papers) and BRCA gene mutations in cancer (2 papers). C J Epstein collaborates with scholars based in United States. C J Epstein's co-authors include Pak H. Chan, Hiroyuki Kinouchi, Erin E. Carlson, Takuji Mizui, Yoram Groner, Karen B. Avraham, Susan Smith, Michelle Lovett, Galit Rotman and Orna Elroy‐Stein and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Neurology.

In The Last Decade

C J Epstein

16 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Attenuation of focal cerebral ischemic injury in transgenic mice overexpressing CuZn superoxide dismutase.

1991 498 citations Hiroyuki Kinouchi, C J Epstein et al. Proceedings of the National Academy of Sciences profile →

Peers

C J Epstein

MB

NEURO

PHYSI

CMN

NEURO

M. Elizabeth Ross United States

Sylvia F. Chen United States

Laura J.S. Greenlund United States

Shao-Hua Yang United States

Eugene M. Johnson United States

Masayoshi Shibata Japan

Yutaka Nagata Japan

Yoshiko Yanagisawa Japan

Arrel D. Toews United States

Maria Paola Cerù Italy

M. Elizabeth Ross United States

C J Epstein

897 ×1.3

MB

222 ×0.7

NEURO

545 ×1.6

PHYSI

554 ×1.9

CMN

722 ×2.4

NEURO

Citations per year, relative to C J Epstein C J Epstein (= 1×) peers M. Elizabeth Ross

Countries citing papers authored by C J Epstein

Since Specialization

Citations

This map shows the geographic impact of C J Epstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C J Epstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C J Epstein more than expected).

Fields of papers citing papers by C J Epstein

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C J Epstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C J Epstein. The network helps show where C J Epstein may publish in the future.

Co-authorship network of co-authors of C J Epstein

This figure shows the co-authorship network connecting the top 25 collaborators of C J Epstein. A scholar is included among the top collaborators of C J Epstein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C J Epstein. C J Epstein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

1.

Kostić, Vladimir, Mark E. Gurney, Han‐Xiang Deng, et al.. (1997). Midbrain dopaminergic neuronal degeneration in a transgenic mouse model of familial amyotrophic lateral sclerosis. Annals of Neurology. 41(4). 497–504. 76 indexed citations

2.

Mikawa, Shigeki, Hiroyuki Kinouchi, Hideyuki Kamii, et al.. (1996). Attenuation of acute and chronic damage following traumatic brain injury in copper, zinc—superoxide dismutase transgenic mice. Journal of neurosurgery. 85(5). 885–891. 146 indexed citations

3.

Elmer, Gregory I., et al.. (1996). Transgenic superoxide dismutase mice. Behavioural Pharmacology. 7(7). 628???639–628???639. 9 indexed citations

4.

Holtzman, David M., et al.. (1993). Nerve growth factor reverses neuronal atrophy in a Down syndrome model of age‐related neurodegeneration. Neurology. 43(12). 2668–2668. 58 indexed citations

5.

Holtzman, David M., S. J. DeArmond, Michael P. McKinley, et al.. (1992). Mouse model of neurodegeneration: atrophy of basal forebrain cholinergic neurons in trisomy 16 transplants.. Proceedings of the National Academy of Sciences. 89(4). 1383–1387. 43 indexed citations

6.

Kinouchi, Hiroyuki, et al.. (1991). Attenuation of focal cerebral ischemic injury in transgenic mice overexpressing CuZn superoxide dismutase.. Proceedings of the National Academy of Sciences. 88(24). 11158–11162. 498 indexed citations breakdown →

7.

Chan, Pak H., et al.. (1990). Reduced neurotoxicity in transgenic mice overexpressing human copper-zinc-superoxide dismutase.. PubMed. 21(11 Suppl). III80–2. 80 indexed citations

8.

Epstein, C J, Karen B. Avraham, Michelle Lovett, et al.. (1987). Transgenic mice with increased Cu/Zn-superoxide dismutase activity: animal model of dosage effects in Down syndrome.. Proceedings of the National Academy of Sciences. 84(22). 8044–8048. 394 indexed citations

9.

Epstein, C J. (1986). Developmental genetics. Cellular and Molecular Life Sciences. 42(10). 1117–1128. 31 indexed citations

10.

Cox, David R., Lois B. Epstein, & C J Epstein. (1980). Genes coding for sensitivity to interferon (IfRec) and soluble superoxide dismutase (SOD-1) are linked in mouse and man and map to mouse chromosome 16.. Proceedings of the National Academy of Sciences. 77(4). 2168–2172. 82 indexed citations

11.

Neufeld, Elizabeth F., Ingeborg Liebaers, C J Epstein, et al.. (1977). The Hunter syndrome in females: is there an autosomal recessive form of iduronate sulfatase deficiency?. PubMed. 29(5). 455–61. 39 indexed citations

12.

Yatziv, S & C J Epstein. (1977). Hunter syndrome presenting as macrocephaly and hydrocephalus.. Journal of Medical Genetics. 14(6). 445–447. 12 indexed citations

13.

Epstein, C J, Robert P. Erickson, Bryan D. Hall, & Mitchell S. Golbus. (1975). The center-satellite system for the wide-scale distribution of genetic counseling services.. PubMed. 27(3). 322–32. 14 indexed citations

14.

Epstein, C J. (1973). Who should do genetic counseling, and under what circumstances?. PubMed. 9(4). 39–48. 7 indexed citations

15.

Epstein, C J & A. G. Motulsky. (1965). Evolutionary origins of human proteins.. PubMed. 4. 85–127. 3 indexed citations

16.

Epstein, C J. (1964). STRUCTURAL AND CONTROL GENE DEFECTS IN HEREDITARY DISEASES IN MAN. The Lancet. 284(7368). 1066–1067. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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