Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Cloning of the gene coding for a shared human melanoma antigen recognized by autologous T cells infiltrating into tumor.
1994886 citationsYutaka Kawakami, S Eliyahu et al.Proceedings of the National Academy of Sciencesprofile →
Identification of a human melanoma antigen recognized by tumor-infiltrating lymphocytes associated with in vivo tumor rejection.
1994742 citationsYutaka Kawakami, S Eliyahu et al.Proceedings of the National Academy of Sciencesprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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This map shows the geographic impact of C H Delgado's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C H Delgado with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C H Delgado more than expected).
This network shows the impact of papers produced by C H Delgado. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C H Delgado. The network helps show where C H Delgado may publish in the future.
Co-authorship network of co-authors of C H Delgado
This figure shows the co-authorship network connecting the top 25 collaborators of C H Delgado.
A scholar is included among the top collaborators of C H Delgado based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with C H Delgado. C H Delgado is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Steller, Michael A., et al.. (1996). Overexpression of the insulin-like growth factor-1 receptor and autocrine stimulation in human cervical cancer cells.. PubMed. 56(8). 1761–5.70 indexed citations
Kawakami, Yutaka, S Eliyahu, C H Delgado, et al.. (1994). Cloning of the gene coding for a shared human melanoma antigen recognized by autologous T cells infiltrating into tumor.. Proceedings of the National Academy of Sciences. 91(9). 3515–3519.886 indexed citations breakdown →
5.
Kawakami, Yutaka, S Eliyahu, C H Delgado, et al.. (1994). Identification of a human melanoma antigen recognized by tumor-infiltrating lymphocytes associated with in vivo tumor rejection.. Proceedings of the National Academy of Sciences. 91(14). 6458–6462.742 indexed citations breakdown →
Pérez, M., António Garrido, Ignacio Algarra, et al.. (1989). H-2 antigens and tumour-associated transplantation antigens in clones derived from a methylcholanthrene-induced BALB/c tumour: their influence on the generation in vitro and in vivo of the specific anti-tumour immune response.. PubMed. 6(3). 204–18.7 indexed citations
8.
Garrido, António, et al.. (1986). Influence of class I H-2 gene expression on local tumor growth. Description of a model obtained from clones derived from a solid BALB/c tumor.. PubMed. 3(2). 98–110.23 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.