C E Gillett

931 total citations
9 papers, 729 citations indexed

About

C E Gillett is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, C E Gillett has authored 9 papers receiving a total of 729 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Oncology and 2 papers in Pathology and Forensic Medicine. Recurrent topics in C E Gillett's work include Cancer-related Molecular Pathways (3 papers), Molecular Biology Techniques and Applications (2 papers) and HER2/EGFR in Cancer Research (2 papers). C E Gillett is often cited by papers focused on Cancer-related Molecular Pathways (3 papers), Molecular Biology Techniques and Applications (2 papers) and HER2/EGFR in Cancer Research (2 papers). C E Gillett collaborates with scholars based in United Kingdom, Belgium and United States. C E Gillett's co-authors include D Steele, Kay Savage, K W Ryder, Andrew Tutt, Anne‐Marie Russell, Alan Ashworth, R. J. Springall, J. S. Reis-Filho, Nicholas C. Turner and Fernando Schmitt and has published in prestigious journals such as The Lancet, Journal of Clinical Oncology and Oncogene.

In The Last Decade

C E Gillett

8 papers receiving 698 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
C E Gillett United Kingdom 6 449 369 304 163 110 9 729
Anita Langerød Norway 9 484 1.1× 568 1.5× 365 1.2× 111 0.7× 75 0.7× 17 898
Crispinita D. Arroyo United States 12 375 0.8× 224 0.6× 220 0.7× 108 0.7× 107 1.0× 12 663
Louis‐François Plassa France 12 549 1.2× 398 1.1× 377 1.2× 72 0.4× 81 0.7× 14 834
M Briffod France 12 312 0.7× 239 0.6× 261 0.9× 55 0.3× 136 1.2× 29 603
Tone Ikdahl Andersen Norway 17 521 1.2× 570 1.5× 353 1.2× 402 2.5× 227 2.1× 24 1.0k
G Merlo Italy 12 590 1.3× 516 1.4× 230 0.8× 94 0.6× 104 0.9× 13 856
Billie‐Jo M. Kerns United States 12 338 0.8× 228 0.6× 185 0.6× 46 0.3× 111 1.0× 18 592
Pinchas Osin United Kingdom 13 231 0.5× 298 0.8× 249 0.8× 283 1.7× 239 2.2× 16 715
Anne-Lise B√∏rresen-Dale Norway 9 483 1.1× 420 1.1× 301 1.0× 78 0.5× 90 0.8× 10 778
Daniela Kandioler-Eckersberger Austria 8 389 0.9× 220 0.6× 235 0.8× 70 0.4× 63 0.6× 11 583

Countries citing papers authored by C E Gillett

Since Specialization
Citations

This map shows the geographic impact of C E Gillett's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C E Gillett with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C E Gillett more than expected).

Fields of papers citing papers by C E Gillett

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C E Gillett. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C E Gillett. The network helps show where C E Gillett may publish in the future.

Co-authorship network of co-authors of C E Gillett

This figure shows the co-authorship network connecting the top 25 collaborators of C E Gillett. A scholar is included among the top collaborators of C E Gillett based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C E Gillett. C E Gillett is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Loi, Sherene, Benjamin Haibe‐Kains, Lajos Pusztai, et al.. (2009). PIK3CA, AKT1 MUTATION AND HER2 AMPLIFICATION GENE SIGNATURES (GS) SUGGEST PREDOMINANTLY NEGATIVE FEEDBACK INHIBITION OF PI3K/AKT PATHWAY IN HUMAN BREAST CANCER (BC).. Annals of Oncology. 20. 45–45. 1 indexed citations
2.
Loi, Sherene, Benjamin Haibe‐Kains, Lajos Pusztai, et al.. (2009). Correlation of PIK3CA mutation-associated gene expression signature (PIK3CA-GS) with deactivation of the PI3K pathway and with prognosis within the luminal-B ER+ breast cancers. Journal of Clinical Oncology. 27(15_suppl). 533–533. 2 indexed citations
3.
Turner, Nicholas C., J. S. Reis-Filho, Anne‐Marie Russell, et al.. (2006). BRCA1 dysfunction in sporadic basal-like breast cancer. Oncogene. 26(14). 2126–2132. 437 indexed citations
5.
Cooper, Lucienne, C E Gillett, A M Hanby, & D M Barnes. (1998). p53 immunoreactivity in breast cancer tissues does not deteriorate in stored paraffin sections. The Breast. 7(5). 260–264. 1 indexed citations
6.
Barnes, D M & C E Gillett. (1995). Determination of cell proliferation. Molecular Pathology. 48(1). M2–M5. 21 indexed citations
7.
Camplejohn, RS, C. M. Ash, C E Gillett, et al.. (1995). The prognostic significance of DNA flow cytometry in breast cancer: results from 881 patients treated in a single centre. British Journal of Cancer. 71(1). 140–145. 50 indexed citations
8.
Camplejohn, RS, et al.. (1993). Ki-S1, a novel proliferative marker: flow cytometric assessment of staining in human breast carcinoma cells. British Journal of Cancer. 67(4). 657–662. 23 indexed citations
9.
Barnes, D M, C E Gillett, L. G. Bobrow, et al.. (1992). Abnormal expression of wild type p53 protein in normal cells of a cancer family patient. The Lancet. 340(8814). 259–263. 170 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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