Bruno Amati

14.3k total citations · 1 hit paper
94 papers, 10.7k citations indexed

About

Bruno Amati is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Bruno Amati has authored 94 papers receiving a total of 10.7k indexed citations (citations by other indexed papers that have themselves been cited), including 78 papers in Molecular Biology, 30 papers in Oncology and 9 papers in Immunology. Recurrent topics in Bruno Amati's work include Genomics and Chromatin Dynamics (27 papers), Ubiquitin and proteasome pathways (25 papers) and Cancer-related Molecular Pathways (23 papers). Bruno Amati is often cited by papers focused on Genomics and Chromatin Dynamics (27 papers), Ubiquitin and proteasome pathways (25 papers) and Cancer-related Molecular Pathways (23 papers). Bruno Amati collaborates with scholars based in Italy, United States and United Kingdom. Bruno Amati's co-authors include Hartmut Land, Scott R. Frank, Gérard I. Evan, Arianna Sabò, Trevor D. Littlewood, Paula Fernández, Susan M. Gasser, Ernesto Guccione, Marianne Schroeder and Stefan Taubert and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Bruno Amati

93 papers receiving 10.5k citations

Hit Papers

Genomic targets of the human c-Myc protein 2003 2026 2010 2018 2003 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bruno Amati Italy 52 9.1k 2.9k 1.4k 940 902 94 10.7k
David T. Weaver United States 49 6.5k 0.7× 2.6k 0.9× 1.3k 1.0× 747 0.8× 1.4k 1.5× 150 9.0k
David O. Ferguson United States 40 6.2k 0.7× 2.4k 0.8× 1.4k 1.0× 572 0.6× 842 0.9× 67 7.6k
Antonio Di Cristofano United States 37 6.7k 0.7× 2.1k 0.7× 1.5k 1.1× 631 0.7× 1.2k 1.3× 75 9.1k
Ralf Janknecht United States 61 8.3k 0.9× 1.7k 0.6× 1.6k 1.2× 834 0.9× 898 1.0× 125 10.2k
Eugenio Santos Spain 45 5.8k 0.6× 2.2k 0.8× 1.1k 0.8× 873 0.9× 802 0.9× 154 8.3k
Douglas C. Dean United States 54 8.4k 0.9× 4.6k 1.6× 1.9k 1.4× 1.3k 1.4× 1.4k 1.5× 107 11.9k
Bruce E. Clurman United States 45 7.8k 0.9× 4.2k 1.5× 1.2k 0.9× 1.9k 2.0× 641 0.7× 77 9.6k
Nicholas B. La Thangue United Kingdom 53 7.6k 0.8× 3.8k 1.3× 1.0k 0.7× 715 0.8× 678 0.8× 103 9.4k
Davide Ruggero United States 48 9.9k 1.1× 1.6k 0.5× 1.7k 1.3× 1.3k 1.3× 1.1k 1.2× 91 11.9k
Ada Sacchi Italy 55 5.8k 0.6× 3.7k 1.3× 1.5k 1.1× 1.2k 1.3× 683 0.8× 162 8.4k

Countries citing papers authored by Bruno Amati

Since Specialization
Citations

This map shows the geographic impact of Bruno Amati's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruno Amati with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruno Amati more than expected).

Fields of papers citing papers by Bruno Amati

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruno Amati. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruno Amati. The network helps show where Bruno Amati may publish in the future.

Co-authorship network of co-authors of Bruno Amati

This figure shows the co-authorship network connecting the top 25 collaborators of Bruno Amati. A scholar is included among the top collaborators of Bruno Amati based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bruno Amati. Bruno Amati is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Donati, Giulio, Paola Nicoli, Alessandro Verrecchia, et al.. (2023). Oxidative stress enhances the therapeutic action of a respiratory inhibitor in MYC ‐driven lymphoma. EMBO Molecular Medicine. 15(6). e16910–e16910. 10 indexed citations
2.
Filipuzzi, M., Alessandro Verrecchia, Paola Nicoli, et al.. (2022). Polycomb group ring finger protein 6 suppresses Myc-induced lymphomagenesis. Life Science Alliance. 5(8). e202101344–e202101344. 4 indexed citations
3.
Pellanda, Paola, M. Filipuzzi, Alessia Loffreda, et al.. (2021). Integrated requirement of non‐specific and sequence‐specific DNA binding in Myc‐driven transcription. The EMBO Journal. 40(10). e105464–e105464. 23 indexed citations
4.
Santoro, Angela, Lucilla Luzi, Giorgio Melloni, et al.. (2019). p53 Loss in Breast Cancer Leads to Myc Activation, Increased Cell Plasticity, and Expression of a Mitotic Signature with Prognostic Value. Cell Reports. 26(3). 624–638.e8. 53 indexed citations
5.
Croci, Ottavio, Serena Fazio, Francesca Biagioni, et al.. (2017). Transcriptional integration of mitogenic and mechanical signals by Myc and YAP. Genes & Development. 31(20). 2017–2022. 67 indexed citations
6.
Pretis, Stefano de, Theresia R. Kress, Marco J. Morelli, et al.. (2017). Integrative analysis of RNA polymerase II and transcriptional dynamics upon MYC activation. Genome Research. 27(10). 1658–1664. 46 indexed citations
7.
Kress, Theresia R., Paola Pellanda, Luca Pellegrinet, et al.. (2016). Identification of MYC-Dependent Transcriptional Programs in Oncogene-Addicted Liver Tumors. Cancer Research. 76(12). 3463–3472. 44 indexed citations
8.
Bianchi, Valerio, Arnaud Céol, Stefano de Pretis, et al.. (2016). Integrated Systems for NGS Data Management and Analysis: Open Issues and Available Solutions. Frontiers in Genetics. 7. 75–75. 35 indexed citations
9.
Tonelli, Claudia, Bruno Amati, & Marco J. Morelli. (2015). p53 transcriptional programs in B cells upon exposure to genotoxic stress in vivo : Computational analysis of next-generation sequencing data. Genomics Data. 7. 29–31. 2 indexed citations
10.
Campaner, Stefano, Fabio Spreafico, Thomas Burgold, et al.. (2011). The Methyltransferase Set7/9 (Setd7) Is Dispensable for the p53-Mediated DNA Damage Response In Vivo. Molecular Cell. 43(4). 681–688. 71 indexed citations
11.
Campaner, Stefano, Mirko Doni, Per Hydbring, et al.. (2009). Cdk2 suppresses cellular senescence induced by the c-myc oncogene. Nature Cell Biology. 12(1). 54–59. 207 indexed citations
12.
Smith, Aaron P., Alessandro Verrecchia, Giovanni Fagà, et al.. (2008). A positive role for Myc in TGFβ-induced Snail transcription and epithelial-to-mesenchymal transition. Oncogene. 28(3). 422–430. 106 indexed citations
13.
Squatrito, Massimo, Chiara Gorrini, & Bruno Amati. (2006). Tip60 in DNA damage response and growth control: many tricks in one HAT. Trends in Cell Biology. 16(9). 433–442. 248 indexed citations
14.
Guccione, Ernesto, Giacomo Finocchiaro, Lucilla Luzi, et al.. (2006). Myc-binding-site recognition in the human genome is determined by chromatin context. Nature Cell Biology. 8(7). 764–770. 299 indexed citations
15.
Grandori, Carla, Kou-Juey Wu, Paula Fernández, et al.. (2003). Werner syndrome protein limits MYC-induced cellular senescence. Genes & Development. 17(13). 1569–1574. 146 indexed citations
16.
Beck, Andreas, Larry Richman, Kay Hofmann, et al.. (1998). Cyclin E2: a novel CDK2 partner in the late G1 and S phases of the mammalian cell cycle. Oncogene. 17(20). 2637–2643. 125 indexed citations
17.
Amati, Bruno, et al.. (1993). Distinct DNA binding preferences for the c-Myc/Max and Max/Max dimers. Nucleic Acids Research. 21(23). 5372–5376. 89 indexed citations
18.
Amati, Bruno, Stephen Dalton, Mary W. Brooks, et al.. (1992). Transcriptional activation by the human c-Myc oncoprotein in yeast requires interaction with Max. Nature. 359(6394). 423–426. 382 indexed citations
19.
Gasser, Susan M., et al.. (1990). Studies on Scaffold Attachment Sites and Their Relation to Genome Function. International review of cytology. 119. 57–96. 176 indexed citations
20.
Amati, Bruno & Susan M. Gasser. (1990). Drosophila Scaffold-Attached Regions Bind Nuclear Scaffolds and Can Function as ARS Elements in Both Budding and Fission Yeasts. Molecular and Cellular Biology. 10(10). 5442–5454. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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