Brooke M. McCartney
- Molecular Biology top 10%
- Cell Biology top 1%
- Cellular and Molecular Neuroscience top 10%
- Neurology top 10%
- Pathology and Forensic Medicine
- Co-authors
- Richard G. FehonMark PeiferDennis LaJeunesseInke NäthkeAmy BejsovecRima M. KulikauskasElizabeth E. GrevengoedKathryn Akong
- Topics
- Wnt/β-catenin signaling in development and cancer (15 papers)Hippo pathway signaling and YAP/TAZ (10 papers)Cancer-related gene regulation (10 papers)
- Cited by
- Cell BiologyMolecular BiologyAging
- Partner nations
- United StatesUnited KingdomSwitzerland
In The Last Decade
Brooke M. McCartney
32 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 82
- Molecular Biology 1.1k
- Cell Biology 783
- Cellular and Molecular Neuroscience 199
- Neurology 145
- Pathology and Forensic Medicine 92
Countries citing papers authored by Brooke M. McCartney
This map shows the geographic impact of Brooke M. McCartney's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brooke M. McCartney with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brooke M. McCartney more than expected).
Fields of papers citing papers by Brooke M. McCartney
This network shows the impact of papers produced by Brooke M. McCartney. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brooke M. McCartney. The network helps show where Brooke M. McCartney may publish in the future.
Co-authorship network of co-authors of Brooke M. McCartney
This figure shows the co-authorship network connecting the top 25 collaborators of Brooke M. McCartney. A scholar is included among the top collaborators of Brooke M. McCartney based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brooke M. McCartney. Brooke M. McCartney is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 2 | |
| 3 | 21 | |
| 4 | 0 | |
| 5 | 12 | |
| 6 | 7 | |
| 7 | 59 | |
| 8 | 37 | |
| 9 | 15 | |
| 10 | 23 | |
| 11 | 21 | |
| 12 | 17 | |
| 13 | 11 | |
| 14 | 110 | |
| 15 | 62 | |
| 16 | 2 | |
| 17 | 56 | |
| 18 | 56 | |
| 19 | 22 | |
| 20 | 122 |
About Brooke M. McCartney
Brooke M. McCartney is a scholar working on Cell Biology, Molecular Biology and Neurology, having authored 36 papers that have together received 1.5k indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (15 papers), Hippo pathway signaling and YAP/TAZ (10 papers) and Cancer-related gene regulation (10 papers). The work is most often cited by research in Cell Biology (783 citations), Molecular Biology (1.1k citations) and Aging (20 citations). Brooke M. McCartney has collaborated with scholars based in United States, United Kingdom and Switzerland. Frequent co-authors include Richard G. Fehon, Mark Peifer, Dennis LaJeunesse, Inke Näthke, Amy Bejsovec, Rima M. Kulikauskas, Elizabeth E. Grevengoed, Kathryn Akong, David M. Roberts and Donald G. McEwen. Their work appears in journals such as Journal of Biological Chemistry, Nature Communications and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.