Brenda Wallace‐Jones

666 citations
11 papers · 563 indexed · h-index 10
Topics
Wnt/β-catenin signaling in development and cancer (4 papers)HER2/EGFR in Cancer Research (3 papers)Cellular Mechanics and Interactions (3 papers)

In The Last Decade

Brenda Wallace‐Jones

11 papers receiving 552 citations

Peers

Brenda Wallace‐Jones
Comparison fields: 5 of 50
  • Molecular Biology 474
  • Oncology 241
  • Cell Biology 155
  • Cancer Research 61
  • Immunology and Allergy 53
Replace Maria Teresa Vento with:
Maria Teresa Vento Italy
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Citations per year

Countries citing papers authored by Brenda Wallace‐Jones

Since Specialization
Citations

This map shows the geographic impact of Brenda Wallace‐Jones's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brenda Wallace‐Jones with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brenda Wallace‐Jones more than expected).

Fields of papers citing papers by Brenda Wallace‐Jones

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Brenda Wallace‐Jones. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brenda Wallace‐Jones. The network helps show where Brenda Wallace‐Jones may publish in the future.

Co-authorship network of co-authors of Brenda Wallace‐Jones

This figure shows the co-authorship network connecting the top 25 collaborators of Brenda Wallace‐Jones. A scholar is included among the top collaborators of Brenda Wallace‐Jones based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brenda Wallace‐Jones. Brenda Wallace‐Jones is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
#WorkIndexed citations
1 65
2 62
3 110
4 9
5 75
6 46
7 30
8 11
9 65
10
Cripto-1 induces phosphatidylinositol 3'-kinase-dependent phosphorylation of AKT and glycogen synthase kinase 3beta in human cervical carcinoma cells.
52
11
Cripto-1 inhibits beta-casein expression in mammary epithelial cells through a p21ras-and phosphatidylinositol 3'-kinase-dependent pathway.
38

About Brenda Wallace‐Jones

Brenda Wallace‐Jones is a scholar working on Anatomy, Immunology and Allergy and Oncology, having authored 11 papers that have together received 563 indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (4 papers), HER2/EGFR in Cancer Research (3 papers) and Cellular Mechanics and Interactions (3 papers). The work is most often cited by research in Cell Biology (155 citations), Oncology (241 citations) and Immunology and Allergy (53 citations). Brenda Wallace‐Jones has collaborated with scholars based in United States, Germany and Italy. Frequent co-authors include Caterina Bianco, Christian Wechselberger, Andreas D. Ebert, David S. Salomon, Nadia Khan, Youping Sun, Masaharu Seno, Isabel Martínez-Lacaci, Luigi Strizzi and Nicola Normanno. Their work appears in journals such as Journal of Biological Chemistry, JNCI Journal of the National Cancer Institute and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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