Boris Bardot

839 total citations
19 papers, 623 citations indexed

About

Boris Bardot is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Boris Bardot has authored 19 papers receiving a total of 623 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 11 papers in Oncology and 3 papers in Cell Biology. Recurrent topics in Boris Bardot's work include Cancer-related Molecular Pathways (10 papers), Developmental Biology and Gene Regulation (5 papers) and RNA modifications and cancer (3 papers). Boris Bardot is often cited by papers focused on Cancer-related Molecular Pathways (10 papers), Developmental Biology and Gene Regulation (5 papers) and RNA modifications and cancer (3 papers). Boris Bardot collaborates with scholars based in France, United States and United Kingdom. Boris Bardot's co-authors include Franck Toledo, Cedric S. Wesley, François Ahimou, Maria Marx, Emmanuelle Huillard, Georges Calothy, Sara Jaber, Donald G. Blair, Qingyun Zhang and Lilia Topol and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and The Journal of Cell Biology.

In The Last Decade

Boris Bardot

18 papers receiving 617 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Boris Bardot France	14	430	161	95	70	61	19	623
May Chien United States	5	638 1.5×	173 1.1×	39 0.4×	53 0.8×	96 1.6×	9	807
Anita Olofsson Sweden	10	382 0.9×	61 0.4×	152 1.6×	79 1.1×	35 0.6×	10	615
Mateusz Kolanczyk Germany	15	315 0.7×	60 0.4×	197 2.1×	66 0.9×	82 1.3×	23	680
Aino Murtomäki United States	7	403 0.9×	215 1.3×	57 0.6×	60 0.9×	90 1.5×	8	611
Victoria A. Robb United States	12	518 1.2×	132 0.8×	78 0.8×	72 1.0×	129 2.1×	14	872
Dave N.T. Aryee Austria	17	614 1.4×	250 1.6×	52 0.5×	68 1.0×	74 1.2×	31	965
Marcel van der Weiden Netherlands	14	252 0.6×	133 0.8×	61 0.6×	161 2.3×	65 1.1×	25	625
John J. Halliday United States	7	309 0.7×	231 1.4×	69 0.7×	117 1.7×	97 1.6×	9	731
Marie E. Beckner United States	16	392 0.9×	146 0.9×	28 0.3×	32 0.5×	69 1.1×	32	764
Ludger Klewes Canada	15	437 1.0×	80 0.5×	139 1.5×	31 0.4×	85 1.4×	18	634

Countries citing papers authored by Boris Bardot

Since Specialization

Citations

This map shows the geographic impact of Boris Bardot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Bardot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Bardot more than expected).

Fields of papers citing papers by Boris Bardot

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boris Bardot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Bardot. The network helps show where Boris Bardot may publish in the future.

Co-authorship network of co-authors of Boris Bardot

This figure shows the co-authorship network connecting the top 25 collaborators of Boris Bardot. A scholar is included among the top collaborators of Boris Bardot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Boris Bardot. Boris Bardot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Jaber, Sara, Iva Simeonova, Marc Gabriel, et al.. (2025). Oncogenic and teratogenic effects of Trp53Y217C, an inflammation-prone mouse model of the human hotspot mutant TP53Y220C. eLife. 13.

Fajac, Anne, Iva Simeonova, Julia Leemput, et al.. (2024). Mutant mice lacking alternatively spliced p53 isoforms unveil Ackr4 as a male-specific prognostic factor in Myc-driven B-cell lymphomas. eLife. 13. 3 indexed citations

Jaber, Sara, Iva Simeonova, Marc Gabriel, et al.. (2024). Oncogenic and teratogenic effects of Trp53Y217C, an inflammation-prone mouse model of the human hotspot mutant TP53Y220C. eLife. 13. 1 indexed citations

Toufektchan, Eléonore, Neelam Giri, Irena Drašković, et al.. (2020). Germline mutation of MDM4 , a major p53 regulator, in a familial syndrome of defective telomere maintenance. Science Advances. 6(15). eaay3511–eaay3511. 27 indexed citations

Bardot, Boris & Franck Toledo. (2017). Targeting MDM4 Splicing in Cancers. Genes. 8(2). 82–82. 14 indexed citations

Jaber, Sara, et al.. (2016). p53 downregulates the Fanconi anaemia DNA repair pathway. Nature Communications. 7(1). 11091–11091. 43 indexed citations

Bardot, Boris, Julia Leemput, Annaïg Hamon, et al.. (2014). Mice engineered for an obligatory Mdm4 exon skipping express higher levels of the Mdm4-S isoform but exhibit increased p53 activity. Oncogene. 34(22). 2943–2948. 24 indexed citations

Fang, Ming, Iva Simeonova, Boris Bardot, et al.. (2013). Mdm4 loss in mice expressing a p53 hypomorph alters tumor spectrum without improving survival. Oncogene. 33(10). 1336–1339. 11 indexed citations

Simeonova, Iva, Sara Jaber, Irena Drašković, et al.. (2013). Mutant Mice Lacking the p53 C-Terminal Domain Model Telomere Syndromes. Cell Reports. 3(6). 2046–2058. 57 indexed citations

10.

Morin, Aurélie, et al.. (2013). Of mice and men. Transcription. 4(2). 67–71. 1 indexed citations

11.

Simeonova, Iva, et al.. (2012). Fuzzy Tandem Repeats Containing p53 Response Elements May Define Species-Specific p53 Target Genes. PLoS Genetics. 8(6). e1002731–e1002731. 20 indexed citations

12.

Bardot, Boris, B. Lemaire, Valérie Domenga, et al.. (2009). Distinct phenotypic and functional features of CADASIL mutations in the Notch3 ligand binding domain. Brain. 132(6). 1601–1612. 89 indexed citations

13.

Qin, Tielin, et al.. (2005). Delta activity independent of its activity as a ligand of Notch. BMC Developmental Biology. 5(1). 6–6. 17 indexed citations

14.

Bardot, Boris, Laure Lecoin, Ingrid Fliniaux, et al.. (2004). Drm/Gremlin, a BMP antagonist, defines the interbud region during feather development.. The International Journal of Developmental Biology. 48(2-3). 149–156. 25 indexed citations

15.

Bardot, Boris, et al.. (2004). The Notch amino terminus regulates protein levels and Delta-induced clustering of Drosophila Notch receptors. Experimental Cell Research. 304(1). 202–223. 27 indexed citations

16.

Ahimou, François, et al.. (2004). The adhesion force of Notch with Delta and the rate of Notch signaling. The Journal of Cell Biology. 167(6). 1217–1229. 73 indexed citations

17.

Picaud, Sandrine, Boris Bardot, Edward De Maeyer, & Isabelle Seif. (2002). Enhanced Tumor Development in Mice Lacking a Functional Type I Interferon Receptor. Journal of Interferon & Cytokine Research. 22(4). 457–462. 43 indexed citations

18.

Bardot, Boris, Laure Lecoin, Emmanuelle Huillard, Georges Calothy, & Maria Marx. (2001). Expression pattern of the drm/gremlin gene during chicken embryonic development. Mechanisms of Development. 101(1-2). 263–265. 23 indexed citations

19.

Topol, Lilia, Boris Bardot, Qingyun Zhang, et al.. (2000). Biosynthesis, Post-translation Modification, and Functional Characterization of Drm/Gremlin. Journal of Biological Chemistry. 275(12). 8785–8793. 125 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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