Bolormaa Vandanmagsar

4.6k total citations · 1 hit paper
30 papers, 3.7k citations indexed

About

Bolormaa Vandanmagsar is a scholar working on Physiology, Molecular Biology and Epidemiology. According to data from OpenAlex, Bolormaa Vandanmagsar has authored 30 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Physiology, 14 papers in Molecular Biology and 9 papers in Epidemiology. Recurrent topics in Bolormaa Vandanmagsar's work include Adipose Tissue and Metabolism (15 papers), Adipokines, Inflammation, and Metabolic Diseases (8 papers) and Mitochondrial Function and Pathology (6 papers). Bolormaa Vandanmagsar is often cited by papers focused on Adipose Tissue and Metabolism (15 papers), Adipokines, Inflammation, and Metabolic Diseases (8 papers) and Mitochondrial Function and Pathology (6 papers). Bolormaa Vandanmagsar collaborates with scholars based in United States, Singapore and Poland. Bolormaa Vandanmagsar's co-authors include Vishwa Deep Dixit, Yun‐Hee Youm, Randall L. Mynatt, Anthony Ravussin, Jacqueline M. Stephens, Éric Ravussin, José E. Galgani, Krisztián Stadler, Hyunwon Yang and Jeffrey M. Gimble and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Bolormaa Vandanmagsar

30 papers receiving 3.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance

2011 2.1k citations Bolormaa Vandanmagsar, Yun‐Hee Youm et al. Nature Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Bolormaa Vandanmagsar United States	20	1.7k	1.4k	1.1k	1.0k	384	30	3.7k
Anthony Ravussin United States	12	1.7k 1.0×	1.4k 1.0×	936 0.9×	1.1k 1.0×	364 0.9×	15	3.6k
Jachelle M. Ofrecio United States	20	2.1k 1.2×	1.4k 1.0×	1.5k 1.4×	750 0.7×	376 1.0×	22	4.1k
Meilian Liu China	35	1.5k 0.9×	1.8k 1.3×	1.6k 1.5×	684 0.7×	443 1.2×	95	4.4k
Atsunori Fukuhara Japan	32	1.5k 0.9×	1.4k 1.0×	1.3k 1.2×	518 0.5×	566 1.5×	88	4.1k
Hae Jin Kim South Korea	13	1.1k 0.6×	1.5k 1.1×	1.2k 1.1×	668 0.6×	228 0.6×	18	3.5k
Janna A. van Diepen Netherlands	24	1.5k 0.8×	1.1k 0.8×	674 0.6×	751 0.7×	386 1.0×	55	3.1k
Ryan W. Grant United States	21	1.5k 0.9×	971 0.7×	1.7k 1.6×	786 0.8×	286 0.7×	38	4.1k
Sonia Fernández‐Veledo Spain	32	1.8k 1.0×	950 0.7×	1.4k 1.3×	452 0.4×	590 1.5×	106	4.0k
Jun Eguchi Japan	29	1.3k 0.7×	1.1k 0.8×	1.0k 1.0×	531 0.5×	504 1.3×	68	3.5k
Megan R. McMullen United States	38	1.4k 0.8×	2.1k 1.5×	656 0.6×	894 0.9×	263 0.7×	79	4.1k

Countries citing papers authored by Bolormaa Vandanmagsar

Since Specialization

Citations

This map shows the geographic impact of Bolormaa Vandanmagsar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bolormaa Vandanmagsar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bolormaa Vandanmagsar more than expected).

Fields of papers citing papers by Bolormaa Vandanmagsar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bolormaa Vandanmagsar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bolormaa Vandanmagsar. The network helps show where Bolormaa Vandanmagsar may publish in the future.

Co-authorship network of co-authors of Bolormaa Vandanmagsar

This figure shows the co-authorship network connecting the top 25 collaborators of Bolormaa Vandanmagsar. A scholar is included among the top collaborators of Bolormaa Vandanmagsar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bolormaa Vandanmagsar. Bolormaa Vandanmagsar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dubuisson, Olga, Elizabeth R. M. Zunica, Bolormaa Vandanmagsar, et al.. (2024). Restricting bioenergetic efficiency enhances longevity and mitochondrial redox capacity in Drosophila melanogaster. Aging Cell. 23(5). e14107–e14107. 3 indexed citations

Axelrod, Christopher L., Ingeborg M. Langohr, Wagner S. Dantas, et al.. (2023). Weight‐independent effects of Roux‐en‐Y gastric bypass surgery on remission of nonalcoholic fatty liver disease in mice. Obesity. 31(12). 2960–2971. 3 indexed citations

Menikdiwela, Kalhara R., Shane Scoggin, Bolormaa Vandanmagsar, et al.. (2023). Genetic Deletion of DNAJB3 Using CRISPR-Cas9, Produced Discordant Phenotypes. Genes. 14(10). 1857–1857. 2 indexed citations

Warfel, Jaycob D., Carrie M. Elks, Bolormaa Vandanmagsar, et al.. (2023). Rats lackingUcp1present a novel translational tool for the investigation of thermogenic adaptation during cold challenge. Acta Physiologica. 238(1). e13935–e13935. 4 indexed citations

Mulya, Anny, Stephan Nieuwoudt, Bolormaa Vandanmagsar, et al.. (2023). GDF15 Mediates the Effect of Skeletal Muscle Contraction on Glucose-Stimulated Insulin Secretion. Diabetes. 72(8). 1070–1082. 24 indexed citations

Vandanmagsar, Bolormaa, Yang Yu, Charlotte Simmler, et al.. (2021). Bioactive compounds from Artemisia dracunculus L. activate AMPK signaling in skeletal muscle. Biomedicine & Pharmacotherapy. 143. 112188–112188. 8 indexed citations

Ghosh, Sujoy, Shawna Wicks, Bolormaa Vandanmagsar, et al.. (2019). Extensive metabolic remodeling after limiting mitochondrial lipid burden is consistent with an improved metabolic health profile. Journal of Biological Chemistry. 294(33). 12313–12327. 20 indexed citations

Mynatt, Randall L., Robert C. Noland, Carrie M. Elks, et al.. (2019). The RNA binding protein HuR influences skeletal muscle metabolic flexibility in rodents and humans. Metabolism. 97. 40–49. 19 indexed citations

Vandanmagsar, Bolormaa, et al.. (2017). Myeloid-specific deletion of NOX2 prevents the metabolic and neurologic consequences of high fat diet. PLoS ONE. 12(8). e0181500–e0181500. 24 indexed citations

10.

Vandanmagsar, Bolormaa, Jaycob D. Warfel, Shawna Wicks, et al.. (2016). Impaired Mitochondrial Fat Oxidation Induces FGF21 in Muscle. Cell Reports. 15(8). 1686–1699. 76 indexed citations

11.

Warfel, Jaycob D., Tamra Mendoza, Sujoy Ghosh, et al.. (2016). Mitochondrial fat oxidation is essential for lipid-induced inflammation in skeletal muscle in mice. Scientific Reports. 6(1). 37941–37941. 35 indexed citations

12.

Haynie, Kimberly R., et al.. (2013). Inhibition of carnitine palymitoyltransferase1b induces cardiac hypertrophy and mortality in mice. Diabetes Obesity and Metabolism. 16(8). 757–760. 48 indexed citations

13.

Youm, Yun‐Hee, Thirumala‐Devi Kanneganti, Bolormaa Vandanmagsar, et al.. (2012). The NLRP3 Inflammasome Promotes Age-Related Thymic Demise and Immunosenescence. Cell Reports. 1(1). 56–68. 132 indexed citations

14.

Vandanmagsar, Bolormaa, Yun‐Hee Youm, Anthony Ravussin, et al.. (2011). NLRP3 inflammasome regulates obesity induced systemic inflammation and insulin signaling (117.4). The Journal of Immunology. 186(1_Supplement). 117.4–117.4. 1 indexed citations

15.

Vandanmagsar, Bolormaa, Yun‐Hee Youm, Anthony Ravussin, et al.. (2011). The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance. Nature Medicine. 17(2). 179–188. 2116 indexed citations breakdown →

16.

Yang, Hyunwon, Yun‐Hee Youm, Bolormaa Vandanmagsar, et al.. (2010). Obesity Increases the Production of Proinflammatory Mediators from Adipose Tissue T Cells and Compromises TCR Repertoire Diversity: Implications for Systemic Inflammation and Insulin Resistance. The Journal of Immunology. 185(3). 1836–1845. 363 indexed citations

17.

Ghosh, Manik C., Gary D. Collins, Bolormaa Vandanmagsar, et al.. (2009). Activation of Wnt5A signaling is required for CXC chemokine ligand 12–mediated T-cell migration. Blood. 114(7). 1366–1373. 54 indexed citations

18.

Yang, Hyunwon, Yun‐Hee Youm, Yuxiang Sun, et al.. (2009). Axin expression in thymic stromal cells contributes to an age-related increase in thymic adiposity and is associated with reduced thymopoiesis independently of ghrelin signaling. Journal of Leukocyte Biology. 85(6). 928–938. 35 indexed citations

19.

Lustig, Ana, Arnell Carter, Bolormaa Vandanmagsar, et al.. (2009). Transcriptome analysis of murine thymocytes reveals age-associated changes in thymic gene expression. International Journal of Medical Sciences. 6(1). 51–64. 20 indexed citations

20.

Yang, Hyunwon, Vishwa Deep Dixit, Kalpesh Patel, et al.. (2008). Reduction in hypophyseal growth hormone and prolactin expression due to deficiency in ghrelin receptor signaling is associated with Pit-1 suppression: Relevance to the immune system. Brain Behavior and Immunity. 22(8). 1138–1145. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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