Bo Liang

578 citations
21 papers · 470 indexed · h-index 11
Topics
MicroRNA in disease regulation (5 papers)Circular RNAs in diseases (5 papers)Cancer-related molecular mechanisms research (4 papers)
Partner nations
ChinaUnited States

In The Last Decade

Bo Liang

20 papers receiving 469 citations

Peers

Bo Liang
Comparison fields: 5 of 75
  • Molecular Biology 292
  • Cancer Research 218
  • Oncology 88
  • Biomaterials 43
  • Surgery 41
Replace Chengmeng Jin with:
Chengmeng Jin China
Minhee Kim South Korea
Nathalia Meireles Da Costa Brazil
Xiongjie Zhu China
Shan Pan China
Hamidreza Saebfar Iran
Tian Lin China
Patrycja Tudrej Poland
Doxakis Anestakis Greece
Xiaoqi He China
Bo Liang relative to Chengmeng Jin China Chengmeng Jin's profile →
Citations per field
00.5×1.5×2.2×
Chengmeng Jin · 1×
Citations per year

Countries citing papers authored by Bo Liang

Since Specialization
Citations

This map shows the geographic impact of Bo Liang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bo Liang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bo Liang more than expected).

Fields of papers citing papers by Bo Liang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bo Liang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bo Liang. The network helps show where Bo Liang may publish in the future.

Co-authorship network of co-authors of Bo Liang

This figure shows the co-authorship network connecting the top 25 collaborators of Bo Liang. A scholar is included among the top collaborators of Bo Liang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bo Liang. Bo Liang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 1
3 1
4 2
5 8
6 1
7 24
8 32
9 38
10 38
11 3
12
Anti-angiogenic and anti-proliferative effects of inhibition of HIF-1α by p-HIF-1α RNAi in colorectal cancer.
3
13
MicroRNA-204 inhibits cell proliferation in T-cell acute lymphoblastic leukemia by down-regulating SOX4.
30
14
MiR-301a promotes cell proliferation by directly targeting TIMP2 in multiple myeloma.
27
15 38
16 99
17 4
18 39
19 19
20 60

About Bo Liang

Bo Liang is a scholar working on Cancer Research, Oncology and Molecular Biology, having authored 21 papers that have together received 470 indexed citations. Recurring topics across this work include MicroRNA in disease regulation (5 papers), Circular RNAs in diseases (5 papers) and Cancer-related molecular mechanisms research (4 papers). The work is most often cited by research in Cancer Research (218 citations), Molecular Biology (292 citations) and Biomaterials (43 citations). Bo Liang has collaborated with scholars based in China and United States. Frequent co-authors include Jun‐Jie Yin, Peihao Yin, Yue He, Ping Yang, Yu Zhou, Jun Liu, Hui Zhao, Yu Wang, Xingang Wang and Qiang Hu. Their work appears in journals such as RSC Advances, International Journal of Nanomedicine and Breast Cancer Research and Treatment.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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