Blaine W. Robinson

614 total citations
17 papers, 266 citations indexed

About

Blaine W. Robinson is a scholar working on Molecular Biology, Hematology and Oncology. According to data from OpenAlex, Blaine W. Robinson has authored 17 papers receiving a total of 266 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Hematology and 4 papers in Oncology. Recurrent topics in Blaine W. Robinson's work include Acute Myeloid Leukemia Research (6 papers), DNA Repair Mechanisms (4 papers) and Acute Lymphoblastic Leukemia research (4 papers). Blaine W. Robinson is often cited by papers focused on Acute Myeloid Leukemia Research (6 papers), DNA Repair Mechanisms (4 papers) and Acute Lymphoblastic Leukemia research (4 papers). Blaine W. Robinson collaborates with scholars based in United States and Italy. Blaine W. Robinson's co-authors include Donna S. Shewach, Michael M. Im, Carolyn A. Felix, Mats Ljungman, Françoise Praz, Timothy G. Strein, Leo J. Ostruszka, Andrew J. Carroll, Cheryl L. Willman and Jeffrey S. Barrett and has published in prestigious journals such as Blood, British Journal of Haematology and Molecular Cancer Therapeutics.

In The Last Decade

Blaine W. Robinson

17 papers receiving 263 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Blaine W. Robinson United States 8 130 74 68 58 51 17 266
Alan Fotoohi Sweden 12 110 0.8× 140 1.9× 79 1.2× 54 0.9× 56 1.1× 17 366
EJ Estlin United Kingdom 12 137 1.1× 119 1.6× 82 1.2× 40 0.7× 95 1.9× 25 454
B‐M Frost Sweden 8 77 0.6× 86 1.2× 66 1.0× 78 1.3× 66 1.3× 13 256
Maribel Tírado-Gómez Puerto Rico 11 226 1.7× 36 0.5× 114 1.7× 76 1.3× 26 0.5× 35 404
Xiaojiao Wang China 11 80 0.6× 41 0.6× 40 0.6× 17 0.3× 44 0.9× 46 310
JY Perrot France 8 162 1.2× 36 0.5× 146 2.1× 83 1.4× 44 0.9× 9 390
Gábor Kovács Hungary 10 102 0.8× 46 0.6× 77 1.1× 27 0.5× 28 0.5× 31 320
Rafael Henriques Jácomo Brazil 11 178 1.4× 60 0.8× 60 0.9× 117 2.0× 10 0.2× 28 332
Wendy P. Gati Canada 10 115 0.9× 49 0.7× 80 1.2× 39 0.7× 29 0.6× 19 368
Britt Sundman‐Engberg Sweden 12 161 1.2× 55 0.7× 184 2.7× 119 2.1× 31 0.6× 20 346

Countries citing papers authored by Blaine W. Robinson

Since Specialization
Citations

This map shows the geographic impact of Blaine W. Robinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Blaine W. Robinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Blaine W. Robinson more than expected).

Fields of papers citing papers by Blaine W. Robinson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Blaine W. Robinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Blaine W. Robinson. The network helps show where Blaine W. Robinson may publish in the future.

Co-authorship network of co-authors of Blaine W. Robinson

This figure shows the co-authorship network connecting the top 25 collaborators of Blaine W. Robinson. A scholar is included among the top collaborators of Blaine W. Robinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Blaine W. Robinson. Blaine W. Robinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Urtishak, Karen, Blaine W. Robinson, Eric Rappaport, et al.. (2016). Unique Familial MLL(KMT2A)‐Rearranged Precursor B‐Cell Infant Acute Lymphoblastic Leukemia in Non‐twin Siblings. Pediatric Blood & Cancer. 63(7). 1175–1180. 3 indexed citations
2.
Dreyer, ZoAnn E., Joanne M. Hilden, Tamekia L. Jones, et al.. (2014). Intensified chemotherapy without SCT in infant ALL: Results from COG P9407 (Cohort 3). Pediatric Blood & Cancer. 62(3). 419–426. 61 indexed citations
3.
Robinson, Blaine W., Giuseppe Germanò, Yuanquan Song, et al.. (2010). mll ortholog containing functional domains of human MLL is expressed throughout the zebrafish lifespan and in haematopoietic tissues. British Journal of Haematology. 152(3). 307–321. 5 indexed citations
4.
Robinson, Blaine W. & Carolyn A. Felix. (2009). Panhandle PCR Approaches to Cloning MLL Genomic Breakpoint Junctions and Fusion Transcript Sequences. Methods in molecular biology. 538. 85–114. 5 indexed citations
5.
6.
Robinson, Blaine W., Kathryn C. Behling, Manish Gupta, et al.. (2008). Abundant anti‐apoptotic BCL‐2 is a molecular target in leukaemias with t(4;11) translocation. British Journal of Haematology. 141(6). 827–839. 33 indexed citations
7.
Robinson, Blaine W., Nai‐Kong V. Cheung, Suresh C. Jhanwar, et al.. (2008). Prospective tracing of MLL-FRYL clone with low MEIS1 expression from emergence during neuroblastoma treatment to diagnosis of myelodysplastic syndrome. Blood. 111(7). 3802–3812. 9 indexed citations
8.
Robinson, Blaine W., Li‐San Wang, Jeffrey S. Barrett, et al.. (2008). Pan-Anti-Apoptotic BCL-2 Family Inhibitor, Obatoclax, Activates Autophagic Cell Death Pathway and Has Potent Cytotoxicity in Infant and Pediatric MLL-Rearranged Leukemias. Blood. 112(11). 2647–2647. 1 indexed citations
9.
Robinson, Blaine W., et al.. (2007). Mismatched nucleotides as the lesions responsible for radiosensitization with gemcitabine: a new paradigm for antimetabolite radiosensitizers. Molecular Cancer Therapeutics. 6(6). 1858–1868. 31 indexed citations
10.
Robinson, Blaine W., Diana J. Slater, & Carolyn A. Felix. (2006). BglII‐based panhandle and reverse panhandle PCR approaches increase capability for cloning der(II) and der(other) genomic breakpoint junctions of MLL translocations. Genes Chromosomes and Cancer. 45(8). 740–753. 7 indexed citations
11.
Robinson, Blaine W., Jeffrey S. Barrett, Jonni S. Moore, et al.. (2005). Pre-Clinical In Vitro Evaluation of BCL-2 Antisense Compound GenasenseTM (G3139; Genta, Inc.) as a Targeted Pro-Apoptotic Agent for Leukemias with t(4;11)(q21;q23) Translocations.. Blood. 106(11). 3360–3360. 2 indexed citations
12.
Germanò, Giuseppe, Blaine W. Robinson, & Carolyn A. Felix. (2005). Cryptic MLL, AF10, ARMC3 Rearrangement in Secondary AML Creates ARMC3-MLL Fusion That Disrupts Gene Encoding Armadillo Repeats Similar to Major Cancer Genes.. Blood. 106(11). 4345–4345. 2 indexed citations
13.
14.
Robinson, Blaine W., Leo J. Ostruszka, Michael M. Im, & Donna S. Shewach. (2004). Promising combination therapies with gemcitabine. Seminars in Oncology. 31(2 Suppl 5). 2–12. 31 indexed citations
15.
Robinson, Blaine W., Michael M. Im, Mats Ljungman, Françoise Praz, & Donna S. Shewach. (2003). Enhanced radiosensitization with gemcitabine in mismatch repair-deficient HCT116 cells.. PubMed. 63(20). 6935–41. 48 indexed citations
16.
Robinson, Blaine W., et al.. (2002). Determination of uric acid in human serum by capillary electrophoresis with polarity reversal and electrochemical detection. Electrophoresis. 23(21). 3705–3710. 23 indexed citations
17.
Shewach, Donna S., Patrick J. Murphy, Blaine W. Robinson, et al.. (2002). Multi-Log Cytotoxicity of Carbocyclic 2′-Deoxyguanosine in HSV-TK-Expressing Human Tumor Cells. Human Gene Therapy. 13(4). 543–551. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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