Michael M. Im

1.9k total citations · 1 hit paper
17 papers, 1.5k citations indexed

About

Michael M. Im is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Michael M. Im has authored 17 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 5 papers in Oncology and 3 papers in Epidemiology. Recurrent topics in Michael M. Im's work include DNA Repair Mechanisms (7 papers), Mitochondrial Function and Pathology (4 papers) and Photosynthetic Processes and Mechanisms (4 papers). Michael M. Im is often cited by papers focused on DNA Repair Mechanisms (7 papers), Mitochondrial Function and Pathology (4 papers) and Photosynthetic Processes and Mechanisms (4 papers). Michael M. Im collaborates with scholars based in United States, Russia and Israel. Michael M. Im's co-authors include Donna S. Shewach, Batsheva Kerem, Assaf C. Bester, Gideon Zamir, Aaron Bensimon, Yifat S. Oren, Maayan Roniger, William P. Fay, Charles F. Yocum and Hana Popelková and has published in prestigious journals such as Cell, Blood and Biochemistry.

In The Last Decade

Michael M. Im

17 papers receiving 1.5k citations

Hit Papers

Nucleotide Deficiency Promotes Genomic Instability in Ear... 2011 2026 2016 2021 2011 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Nucleotide Deficiency Promotes Genomic Instability in Early Stages of Cancer Development
    2011 635 citations Assaf C. Bester, Maayan Roniger et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael M. Im United States 14 1.1k 331 280 169 144 17 1.5k
Felix Bachmann Switzerland 18 797 0.7× 341 1.0× 206 0.7× 136 0.8× 101 0.7× 60 1.7k
Mati Shaklai Israel 23 826 0.8× 317 1.0× 123 0.4× 296 1.8× 75 0.5× 83 1.7k
Igor B. Rosenwald United States 19 1.9k 1.8× 418 1.3× 236 0.8× 110 0.7× 92 0.6× 24 2.2k
Xincheng Lu China 16 1.2k 1.1× 357 1.1× 624 2.2× 71 0.4× 95 0.7× 30 1.6k
Béatrice Cambien France 17 479 0.4× 368 1.1× 141 0.5× 305 1.8× 80 0.6× 29 1.6k
Jianzhong Huang United States 23 1.1k 1.0× 432 1.3× 483 1.7× 38 0.2× 83 0.6× 39 1.7k
Ryushin Mizuta Japan 15 1.8k 1.7× 550 1.7× 378 1.4× 53 0.3× 276 1.9× 29 2.5k
Olaf R.F. Mook Netherlands 18 919 0.9× 361 1.1× 498 1.8× 72 0.4× 229 1.6× 26 1.6k
Stephanie Taylor United States 8 674 0.6× 154 0.5× 261 0.9× 100 0.6× 74 0.5× 12 1.2k

Countries citing papers authored by Michael M. Im

Since Specialization
Citations

This map shows the geographic impact of Michael M. Im's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael M. Im with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael M. Im more than expected).

Fields of papers citing papers by Michael M. Im

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael M. Im. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael M. Im. The network helps show where Michael M. Im may publish in the future.

Co-authorship network of co-authors of Michael M. Im

This figure shows the co-authorship network connecting the top 25 collaborators of Michael M. Im. A scholar is included among the top collaborators of Michael M. Im based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael M. Im. Michael M. Im is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
2.
Im, Michael M., et al.. (2016). Late DNA Damage Mediated by Homologous Recombination Repair Results in Radiosensitization with Gemcitabine. Radiation Research. 186(5). 466–477. 5 indexed citations
3.
Im, Michael M., et al.. (2015). Drug Metabolism and Homologous Recombination Repair in Radiosensitization with Gemcitabine. Radiation Research. 183(1). 114–123. 7 indexed citations
4.
Lamm, Noa, Assaf C. Bester, Michael M. Im, et al.. (2015). Folate levels modulate oncogene‐induced replication stress and tumorigenicity. EMBO Molecular Medicine. 7(9). 1138–1152. 28 indexed citations
5.
Wawrzyniak, Joseph A., Anna Bianchi, Wiam Bshara, et al.. (2013). A Purine Nucleotide Biosynthesis Enzyme Guanosine Monophosphate Reductase Is a Suppressor of Melanoma Invasion. Cell Reports. 5(2). 493–507. 41 indexed citations
6.
Mannava, Sudha, Kalyana Moparthy, Linda J. Wheeler, et al.. (2012). Depletion of Deoxyribonucleotide Pools Is an Endogenous Source of DNA Damage in Cells Undergoing Oncogene-Induced Senescence. American Journal Of Pathology. 182(1). 142–151. 67 indexed citations
7.
Bester, Assaf C., Maayan Roniger, Yifat S. Oren, et al.. (2011). Nucleotide Deficiency Promotes Genomic Instability in Early Stages of Cancer Development. Cell. 145(3). 435–446. 635 indexed citations breakdown →
8.
Mannava, Sudha, Vladimir Grachtchouk, Linda J. Wheeler, et al.. (2008). Direct role of nucleotide metabolism in C-MYC-dependent proliferation of melanoma cells. Cell Cycle. 7(15). 2392–2400. 190 indexed citations
9.
Boucher, Paul D., Michael M. Im, Svend O. Freytag, & Donna S. Shewach. (2006). A Novel Mechanism of Synergistic Cytotoxicity with 5-Fluorocytosine and Ganciclovir in Double Suicide Gene Therapy. Cancer Research. 66(6). 3230–3237. 55 indexed citations
10.
11.
Robinson, Blaine W., Leo J. Ostruszka, Michael M. Im, & Donna S. Shewach. (2004). Promising combination therapies with gemcitabine. Seminars in Oncology. 31(2 Suppl 5). 2–12. 31 indexed citations
12.
Day, Sharlene M., Jennifer Reeve, Brian Pedersen, et al.. (2004). Macrovascular thrombosis is driven by tissue factor derived primarily from the blood vessel wall. Blood. 105(1). 192–198. 229 indexed citations
13.
Im, Michael M., Andrew Heath, Ulrika Ringdahl, et al.. (2003). Plasminogen Enhances Virulence of Group A Streptococci by Streptokinase‐Dependent and Streptokinase‐Independent Mechanisms. The Journal of Infectious Diseases. 188(4). 497–505. 56 indexed citations
14.
Robinson, Blaine W., Michael M. Im, Mats Ljungman, Françoise Praz, & Donna S. Shewach. (2003). Enhanced radiosensitization with gemcitabine in mismatch repair-deficient HCT116 cells.. PubMed. 63(20). 6935–41. 48 indexed citations
15.
Popelková, Hana, Michael M. Im, & Charles F. Yocum. (2003). Binding of Manganese Stabilizing Protein to Photosystem II:  Identification of Essential N-Terminal Threonine Residues and Domains that Prevent Nonspecific Binding. Biochemistry. 42(20). 6193–6200. 30 indexed citations
16.
Popelková, Hana, Michael M. Im, John C. D’Auria, et al.. (2002). N-Terminus of the Photosystem II Manganese Stabilizing Protein:  Effects of Sequence Elongation and Truncation. Biochemistry. 41(8). 2702–2711. 35 indexed citations
17.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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