Beth Coyle
- Genetics top 0.5%
- Glioma Diagnosis and Treatment 28
- Sensory Systems top 1%
- Hearing, Cochlea, Tinnitus, Genetics 6
- Otorhinolaryngology top 2%
- Neurology top 5%
- Cancer Research top 5%
- MicroRNA in disease regulation 5
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- Cancer Cells and Metastasis 9
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- Hedgehog Signaling Pathway Studies 9
- Epigenetics and DNA Methylation 8
- Cancer therapeutics and mechanisms 4
- Histone Deacetylase Inhibitors Research 4
- Co-authors
- Richard G. GrundyJames LoweAnna M. GrabowskaDavid WalkerDelyan P. IvanovMartyna Adamowicz‐BriceDavid W. EllisonRichard C. Trembath
- Partner nations
- United KingdomUnited StatesGermany
In The Last Decade
Beth Coyle
58 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 98
- Genetics 1.0k
- Sensory Systems 305
- Otorhinolaryngology 162
- Neurology 208
- Cancer Research 378
Countries citing papers authored by Beth Coyle
This map shows the geographic impact of Beth Coyle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth Coyle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth Coyle more than expected).
Fields of papers citing papers by Beth Coyle
This network shows the impact of papers produced by Beth Coyle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth Coyle. The network helps show where Beth Coyle may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Beth Coyle, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 2 | |
| 2 | 2023 | 9 | |
| 3 | 2023 | 15 | |
| 4 | 2018 | 55 | |
| 5 | 2018 | 4 | |
| 6 | 2016 | 155 | |
| 7 | 2016 | 1 | |
| 8 | 2016 | 3 | |
| 9 | 2013 | 14 | |
| 10 | 2012 | 72 | |
| 11 | 2012 | 18 | |
| 12 | 2010 | 31 | |
| 13 | 2009 | 31 | |
| 14 | 2008 | 77 | |
| 15 | 2008 | 26 | |
| 16 | 2003 | 16 | |
| 17 | 2003 | 33 | |
| 18 | 1999 | 40 | |
| 19 | 1998 | 180 | |
| 20 | 1996 | 13 |
About Beth Coyle
Beth Coyle is a scholar working on Genetics, Sensory Systems, Oncology, Cancer Research and Molecular Biology, having authored 59 papers that have together received 2.1k indexed citations. Recurring topics across this work include Glioma Diagnosis and Treatment (28 papers), Cancer Cells and Metastasis (9 papers), Hedgehog Signaling Pathway Studies (9 papers), Epigenetics and DNA Methylation (8 papers), Hearing, Cochlea, Tinnitus, Genetics (6 papers), MicroRNA in disease regulation (5 papers), Cancer therapeutics and mechanisms (4 papers) and Histone Deacetylase Inhibitors Research (4 papers). The work is most often cited by research in Genetics (1.0k citations), Sensory Systems (305 citations), Otorhinolaryngology (162 citations), Neurology (208 citations) and Cancer Research (378 citations). Beth Coyle has collaborated with scholars based in United Kingdom, United States and Germany. Frequent co-authors include Richard G. Grundy, James Lowe, Anna M. Grabowska, David Walker, Delyan P. Ivanov, Martyna Adamowicz‐Brice, David W. Ellison, Richard C. Trembath, William Reardon and John‐Paul Kilday. Their work appears in journals such as Neuro-Oncology, British Journal of Cancer, Cancers, Clinical Cancer Research and Acta Neuropathologica Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.