Bernhard E. Flucher

5.3k total citations
107 papers, 4.3k citations indexed

About

Bernhard E. Flucher is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Bernhard E. Flucher has authored 107 papers receiving a total of 4.3k indexed citations (citations by other indexed papers that have themselves been cited), including 104 papers in Molecular Biology, 79 papers in Cellular and Molecular Neuroscience and 38 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Bernhard E. Flucher's work include Ion channel regulation and function (93 papers), Neuroscience and Neuropharmacology Research (36 papers) and Neuroscience and Neural Engineering (35 papers). Bernhard E. Flucher is often cited by papers focused on Ion channel regulation and function (93 papers), Neuroscience and Neuropharmacology Research (36 papers) and Neuroscience and Neural Engineering (35 papers). Bernhard E. Flucher collaborates with scholars based in Austria, United States and Germany. Bernhard E. Flucher's co-authors include Gerald J. Obermair, Mathew P. Daniels, Petronel Tuluc, Marta Campiglio, Manfred Grabner, Clara Franzini‐Armstrong, C Franzini-Armstrong, Jeanne A. Powell, Hiroaki Takekura and S.B. Andrews and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Bernhard E. Flucher

103 papers receiving 4.2k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Bernhard E. Flucher 3.8k 2.3k 1.5k 605 398 107 4.3k
Geoffrey S. Pitt 4.1k 1.1× 1.9k 0.8× 2.2k 1.5× 676 1.1× 258 0.6× 91 5.1k
Xiangang Zong 2.8k 0.7× 2.1k 0.9× 1.3k 0.8× 160 0.3× 504 1.3× 40 4.1k
Jeanne A. Powell 2.3k 0.6× 1.4k 0.6× 844 0.6× 256 0.4× 229 0.6× 40 2.6k
C Franzini-Armstrong 2.4k 0.6× 931 0.4× 1.2k 0.8× 400 0.7× 243 0.6× 24 2.8k
Eleazar Vega‐Saenz de Miera 3.0k 0.8× 1.8k 0.8× 1.1k 0.7× 162 0.3× 230 0.6× 49 3.7k
Delphine Bichet 2.0k 0.5× 1.1k 0.5× 657 0.4× 197 0.3× 321 0.8× 34 2.5k
Tatiana M. Vinogradova 2.7k 0.7× 937 0.4× 2.3k 1.5× 932 1.5× 120 0.3× 57 3.8k
Alan H. Sharp 6.0k 1.6× 5.3k 2.4× 494 0.3× 914 1.5× 582 1.5× 54 7.6k
Dominik Oliver 2.1k 0.6× 1.1k 0.5× 562 0.4× 214 0.4× 191 0.5× 62 3.6k
Ilana Lotan 2.6k 0.7× 1.5k 0.7× 895 0.6× 446 0.7× 191 0.5× 69 2.9k

Countries citing papers authored by Bernhard E. Flucher

Since Specialization
Citations

This map shows the geographic impact of Bernhard E. Flucher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernhard E. Flucher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernhard E. Flucher more than expected).

Fields of papers citing papers by Bernhard E. Flucher

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bernhard E. Flucher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernhard E. Flucher. The network helps show where Bernhard E. Flucher may publish in the future.

Co-authorship network of co-authors of Bernhard E. Flucher

This figure shows the co-authorship network connecting the top 25 collaborators of Bernhard E. Flucher. A scholar is included among the top collaborators of Bernhard E. Flucher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bernhard E. Flucher. Bernhard E. Flucher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fernández‐Quintero, Monica L., Marta Campiglio, Johannes R. Loeffler, et al.. (2025). Voltage-sensor gating charge interactions bimodally regulate voltage dependence and kinetics of calcium channel activation. The Journal of General Physiology. 157(5).
2.
Fernández‐Quintero, Monica L., et al.. (2024). CaV1.1 voltage-sensing domain III exclusively controls skeletal muscle excitation-contraction coupling. Nature Communications. 15(1). 7440–7440. 4 indexed citations
3.
Liedl, Klaus R., et al.. (2023). Asymmetric contribution of a selectivity filter gate in triggering inactivation of CaV1.3 channels. The Journal of General Physiology. 156(2). 1 indexed citations
4.
Fernández‐Quintero, Monica L., et al.. (2021). Structural determinants of voltage-gating properties in calcium channels. eLife. 10. 30 indexed citations
5.
Fernández‐Quintero, Monica L., Abeltje M. Polstra, Johanna M. van Hagen, et al.. (2021). CACNA1I gain-of-function mutations differentially affect channel gating and cause neurodevelopmental disorders. Brain. 144(7). 2092–2106. 31 indexed citations
6.
Bierhals, Tatjana, Marta Campiglio, Jessika Johannsen, et al.. (2020). A homozygous missense variant in CACNB4 encoding the auxiliary calcium channel beta4 subunit causes a severe neurodevelopmental disorder and impairs channel and non-channel functions. PLoS Genetics. 16(3). e1008625–e1008625. 26 indexed citations
7.
Polstra, Abeltje M., Johanna M. van Hagen, Marta Campiglio, et al.. (2020). Two CaV3.3 (CACNA1I) Gain-of-Function Mutations Linked to Epilepsy and Intellectual Disability Affect Gating Properties and the Window Current. Biophysical Journal. 118(3). 106a–106a. 1 indexed citations
8.
Flucher, Bernhard E., et al.. (2020). Multiple Sequence Variants in STAC3 Affect Interactions with CaV1.1 and Excitation-Contraction Coupling. Structure. 28(8). 922–932.e5. 21 indexed citations
9.
Flucher, Bernhard E., et al.. (2019). Postsynaptic CaV1.1-driven calcium signaling coordinates presynaptic differentiation at the developing neuromuscular junction. Scientific Reports. 9(1). 18450–18450. 4 indexed citations
10.
Campiglio, Marta, et al.. (2018). STAC proteins associate to the IQ domain of Ca V 1.2 and inhibit calcium-dependent inactivation. Proceedings of the National Academy of Sciences. 115(6). 1376–1381. 37 indexed citations
11.
Campiglio, Marta, et al.. (2018). STAC Proteins Associate to the IQ Domain of CaV1.2 and Inhibit Calcium-Dependent Inactivation. Biophysical Journal. 114(3). 638a–638a. 3 indexed citations
12.
Flucher, Bernhard E.. (2016). Specific contributions of the four voltage-sensing domains in L-type calcium channels to gating and modulation. The Journal of General Physiology. 148(2). 91–95. 8 indexed citations
13.
Gstir, Ronald, Simon Schafferer, Marcel Scheideler, et al.. (2014). Generation of a neuro-specific microarray reveals novel differentially expressed noncoding RNAs in mouse models for neurodegenerative diseases. RNA. 20(12). 1929–1943. 29 indexed citations
14.
Tuluc, Petronel, et al.. (2014). Calcium Channel α2δ-1 Subunit Knockout Causes Diabetes Due to Impaired Insulin Release. Biophysical Journal. 106(2). 331a–331a. 1 indexed citations
15.
Flucher, Bernhard E., Gerald J. Obermair, Petronel Tuluc, et al.. (2005). The role of auxiliary dihydropyridine receptor subunits in muscle. Journal of Muscle Research and Cell Motility. 26(1). 1–6. 33 indexed citations
16.
Takekura, Hiroaki, et al.. (2004). Differential Contribution of Skeletal and Cardiac II-III Loop Sequences to the Assembly of Dihydropyridine-Receptor Arrays in Skeletal Muscle. Molecular Biology of the Cell. 15(12). 5408–5419. 43 indexed citations
17.
Obermair, Gerald J., et al.. (2003). Cardiac-type EC-Coupling in Dysgenic Myotubes Restored with Ca2+ Channel Subunit Isoforms α1C and α1D Does not Correlate with Current Density. Biophysical Journal. 84(6). 3816–3828. 24 indexed citations
18.
Takekura, Hiroaki, Bernhard E. Flucher, & Clara Franzini‐Armstrong. (2001). Sequential Docking, Molecular Differentiation, and Positioning of T-Tubule/SR Junctions in Developing Mouse Skeletal Muscle. Developmental Biology. 239(2). 204–214. 86 indexed citations
19.
Hofmann, Franz, et al.. (2000). Effects of the dihydropyridine receptor subunits γ and α2δ on the kinetics of heterologously expressed L-type Ca2+ channels. Pflügers Archiv - European Journal of Physiology. 439(6). 691–699. 24 indexed citations
20.
Flucher, Bernhard E., S.B. Andrews, & Mathew P. Daniels. (1994). Molecular organization of transverse tubule/sarcoplasmic reticulum junctions during development of excitation-contraction coupling in skeletal muscle.. Molecular Biology of the Cell. 5(10). 1105–1118. 71 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Breakdown of academic impact, for papers by Geoffrey S. Pitt Breakdown of academic impact, for papers by Xiangang Zong Breakdown of academic impact, for papers by Jeanne A. Powell Breakdown of academic impact, for papers by C Franzini-Armstrong Breakdown of academic impact, for papers by Eleazar Vega‐Saenz de Miera Breakdown of academic impact, for papers by Delphine Bichet Breakdown of academic impact, for papers by Tatiana M. Vinogradova Breakdown of academic impact, for papers by Alan H. Sharp Breakdown of academic impact, for papers by Dominik Oliver Breakdown of academic impact, for papers by Ilana Lotan
Rankless by CCL
2026