Benoît Langlois
Impact in
- Immunology and Allergy top 2%
- Cell Adhesion Molecules Research
- Cancer Research top 10%
- Protease and Inhibitor Mechanisms
- Cancer, Lipids, and Metabolism
Papers in ⓘ
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- Cell Adhesion Molecules Research 12
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- Protease and Inhibitor Mechanisms 10
- Co-authors
- Gertraud Orend (4 shared papers)Thomas Hussenet (3 shared papers)Kim S. Midwood (1 shared paper)Stéphane Dedieu (12 shared papers)Hervé Emonard (5 shared papers)Christophe Schneider (5 shared papers)Hervé Sartelet (4 shared papers)Patrick Henriet (2 shared papers)
In The Last Decade
Benoît Langlois
18 papers receiving 841 citations
Peers
Comparison fields: 5 of 89
- Immunology and Allergy 250
- Cancer Research 247
- Cell Biology 226
- Equine 20
- Oncology 179
Countries citing papers authored by Benoît Langlois
This map shows the geographic impact of Benoît Langlois's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benoît Langlois with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benoît Langlois more than expected).
Fields of papers citing papers by Benoît Langlois
This network shows the impact of papers produced by Benoît Langlois. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benoît Langlois. The network helps show where Benoît Langlois may publish in the future.
Co-authors
The 25 scholars most cited alongside Benoît Langlois, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 262 | |
| 2 | 2016 | 73 | |
| 3 | 2010 | 73 | |
| 4 | 2008 | 64 | |
| 5 | 2014 | 53 | |
| 6 | 2008 | 46 | |
| 7 | 2012 | 41 | |
| 8 | 2017 | 36 | |
| 9 | 2020 | 36 | |
| 10 | 2008 | 34 | |
| 11 | 2018 | 33 | |
| 12 | 2008 | 29 | |
| 13 | 2020 | 23 | |
| 14 | 2017 | 23 | |
| 15 | 1980 | 21 | |
| 16 | 2009 | 6 | |
| 17 | 2022 | 2 | |
| 18 | 1981 | 2 | |
| 19 | 2025 | 0 |
About Benoît Langlois
Benoît Langlois is a scholar working on Immunology and Allergy, Cancer Research, Tourism, Leisure and Hospitality Management, Cell Biology and Oncology, having authored 19 papers that have together received 857 indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (12 papers), Protease and Inhibitor Mechanisms (10 papers), Cellular Mechanics and Interactions (3 papers), Angiogenesis and VEGF in Cancer (2 papers), Cancer Cells and Metastasis (2 papers), Genetic and phenotypic traits in livestock (2 papers), Cellular transport and secretion (2 papers) and Peptidase Inhibition and Analysis (2 papers). The work is most often cited by research in Immunology and Allergy (250 citations), Cancer Research (247 citations), Cell Biology (226 citations), Equine (20 citations) and Oncology (179 citations). Benoît Langlois has collaborated with scholars based in France, Belgium and Germany. Frequent co-authors include Gertraud Orend, Thomas Hussenet, Kim S. Midwood, Stéphane Dedieu, Hervé Emonard, Christophe Schneider, Hervé Sartelet, Patrick Henriet, Laurent Martiny and Jérôme Devy. Their work appears in journals such as Frontiers in Oncology, Genetics Selection Evolution, The International Journal of Biochemistry & Cell Biology, Oncotarget and Cell Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.