Benjamin Wayment

1.3k total citations
11 papers, 1.0k citations indexed

About

Benjamin Wayment is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, Benjamin Wayment has authored 11 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Cardiology and Cardiovascular Medicine and 7 papers in Physiology. Recurrent topics in Benjamin Wayment's work include Cardiovascular Function and Risk Factors (7 papers), Adipose Tissue and Metabolism (6 papers) and Mitochondrial Function and Pathology (4 papers). Benjamin Wayment is often cited by papers focused on Cardiovascular Function and Risk Factors (7 papers), Adipose Tissue and Metabolism (6 papers) and Mitochondrial Function and Pathology (4 papers). Benjamin Wayment collaborates with scholars based in United States, France and South Korea. Benjamin Wayment's co-authors include E. Dale Abel, Sheldon E. Litwin, Heather Theobald, Heiko Bugger, Vlad G. Zaha, Sihem Boudina, Adam R. Wende, Sandra Sena, Joseph Tuinei and Jaetaek Kim and has published in prestigious journals such as Circulation, PLoS ONE and Circulation Research.

In The Last Decade

Benjamin Wayment

11 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Benjamin Wayment United States 10 593 590 330 145 103 11 1.0k
Joseph Tuinei United States 10 527 0.9× 576 1.0× 436 1.3× 146 1.0× 182 1.8× 13 1.1k
Brett O. Schönekess Canada 10 685 1.2× 724 1.2× 347 1.1× 135 0.9× 57 0.6× 14 1.3k
Jodil Willems Netherlands 15 361 0.6× 855 1.4× 411 1.2× 239 1.6× 81 0.8× 19 1.2k
Rolf Schreckenberg Germany 20 358 0.6× 375 0.6× 168 0.5× 158 1.1× 49 0.5× 58 847
Suneet Purohit United States 5 288 0.5× 412 0.7× 188 0.6× 158 1.1× 77 0.7× 5 753
Yanqiu Xing China 12 269 0.5× 430 0.7× 170 0.5× 190 1.3× 109 1.1× 23 730
David Coven United States 6 278 0.5× 601 1.0× 201 0.6× 313 2.2× 143 1.4× 8 896
Haruyo Yamashita Japan 10 376 0.6× 389 0.7× 266 0.8× 124 0.9× 110 1.1× 25 854
Sang Yong Ji United States 8 297 0.5× 350 0.6× 328 1.0× 138 1.0× 56 0.5× 10 851
Ashraf Abrahani Canada 22 402 0.7× 598 1.0× 246 0.7× 185 1.3× 93 0.9× 25 1.1k

Countries citing papers authored by Benjamin Wayment

Since Specialization
Citations

This map shows the geographic impact of Benjamin Wayment's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Wayment with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Wayment more than expected).

Fields of papers citing papers by Benjamin Wayment

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Wayment. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Wayment. The network helps show where Benjamin Wayment may publish in the future.

Co-authorship network of co-authors of Benjamin Wayment

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Wayment. A scholar is included among the top collaborators of Benjamin Wayment based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Wayment. Benjamin Wayment is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Wende, Adam R., Jaetaek Kim, William L. Holland, et al.. (2017). Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses. American Journal of Physiology-Heart and Circulatory Physiology. 313(6). H1098–H1108. 44 indexed citations
2.
Ilkun, Olesya, Nicole Wilde, Joseph Tuinei, et al.. (2015). Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome. Journal of Molecular and Cellular Cardiology. 85. 104–116. 26 indexed citations
3.
Zhu, Yi, Karla Maria Pereira Pires, Kevin J. Whitehead, et al.. (2013). Mechanistic Target of Rapamycin (Mtor) Is Essential for Murine Embryonic Heart Development and Growth. PLoS ONE. 8(1). e54221–e54221. 74 indexed citations
4.
Bugger, Heiko, Christian Riehle, Bharat P. Jaishy, et al.. (2012). Genetic loss of insulin receptors worsens cardiac efficiency in diabetes. Journal of Molecular and Cellular Cardiology. 52(5). 1019–1026. 59 indexed citations
5.
Riehle, Christian, Adam R. Wende, Vlad G. Zaha, et al.. (2011). PGC-1β Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy. Circulation Research. 109(7). 783–793. 114 indexed citations
6.
Sena, Sandra, Ping Hu, Dongfang Zhang, et al.. (2009). Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarction. Journal of Molecular and Cellular Cardiology. 46(6). 910–918. 57 indexed citations
7.
Boudina, Sihem, Heiko Bugger, Sandra Sena, et al.. (2009). Contribution of Impaired Myocardial Insulin Signaling to Mitochondrial Dysfunction and Oxidative Stress in the Heart. Circulation. 119(9). 1272–1283. 235 indexed citations
8.
Kim, Jaetaek, Adam R. Wende, Sandra Sena, et al.. (2008). Insulin-Like Growth Factor I Receptor Signaling Is Required for Exercise-Induced Cardiac Hypertrophy. Molecular Endocrinology. 22(11). 2531–2543. 172 indexed citations
9.
Bugger, Heiko, Sihem Boudina, Xiao Xuan Hu, et al.. (2008). Type 1 Diabetic Akita Mouse Hearts Are Insulin Sensitive but Manifest Structurally Abnormal Mitochondria That Remain Coupled Despite Increased Uncoupling Protein 3. Diabetes. 57(11). 2924–2932. 150 indexed citations
10.
Riehle, Christian, Benjamin Wayment, Karla Maria Pereira Pires, et al.. (2008). Abstract 3559: Insulin Receptor Substrates (IRS) Signaling are Essential Regulators of Mitochondrial Function and Cardiomyocyte Survival. Circulation. 118(suppl_18). 3 indexed citations
11.
O’Neill, Brian T., Jaetaek Kim, Adam R. Wende, et al.. (2007). A Conserved Role for Phosphatidylinositol 3-Kinase but Not Akt Signaling in Mitochondrial Adaptations that Accompany Physiological Cardiac Hypertrophy. Cell Metabolism. 6(4). 294–306. 112 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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