Benjamin Schattling

1.4k total citations · 1 hit paper
13 papers, 945 citations indexed

About

Benjamin Schattling is a scholar working on Molecular Biology, Neurology and Pathology and Forensic Medicine. According to data from OpenAlex, Benjamin Schattling has authored 13 papers receiving a total of 945 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Neurology and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Benjamin Schattling's work include Multiple Sclerosis Research Studies (4 papers), Ion Channels and Receptors (4 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Benjamin Schattling is often cited by papers focused on Multiple Sclerosis Research Studies (4 papers), Ion Channels and Receptors (4 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Benjamin Schattling collaborates with scholars based in Germany, Slovakia and Switzerland. Benjamin Schattling's co-authors include Manuel A. Friese, Lars Fugger, Friederike Ufer, Marc Freichel, Matthias Kneussel, Rudi Vennekens, Aurélie Menigoz, Olaf Pongs, Edda Thies and Martin Kruse and has published in prestigious journals such as Nature Medicine, Stroke and Science Advances.

In The Last Decade

Benjamin Schattling

13 papers receiving 939 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanisms of neurodegeneration and axonal dysfunction in multiple sclerosis

2014 455 citations Manuel A. Friese, Benjamin Schattling et al. Nature Reviews Neurology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Benjamin Schattling Germany	12	375	338	200	152	145	13	945
Paul D. Storer United States	13	348 0.9×	163 0.5×	188 0.9×	43 0.3×	149 1.0×	19	852
Stefan Gingele Germany	19	352 0.9×	527 1.6×	404 2.0×	28 0.2×	241 1.7×	59	1.3k
Yingyan Pu China	18	396 1.1×	124 0.4×	259 1.3×	33 0.2×	162 1.1×	34	909
Yimin Yuan China	18	328 0.9×	246 0.7×	257 1.3×	68 0.4×	70 0.5×	32	993
Jeong‐Sun Choi South Korea	21	418 1.1×	68 0.2×	211 1.1×	23 0.2×	101 0.7×	37	943
Elena Ambrosini Italy	20	603 1.6×	128 0.4×	547 2.7×	24 0.2×	365 2.5×	35	1.5k
Ettore Salsano Italy	22	638 1.7×	77 0.2×	322 1.6×	106 0.7×	59 0.4×	72	1.6k
Catherine D. Sorbara Germany	7	394 1.1×	342 1.0×	306 1.5×	10 0.1×	124 0.9×	8	965
Gianluca Manni Italy	32	734 2.0×	325 1.0×	658 3.3×	27 0.2×	25 0.2×	93	3.3k

Countries citing papers authored by Benjamin Schattling

Since Specialization

Citations

This map shows the geographic impact of Benjamin Schattling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Schattling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Schattling more than expected).

Fields of papers citing papers by Benjamin Schattling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Schattling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Schattling. The network helps show where Benjamin Schattling may publish in the future.

Co-authorship network of co-authors of Benjamin Schattling

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Schattling. A scholar is included among the top collaborators of Benjamin Schattling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Schattling. Benjamin Schattling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Spohn, Michael, et al.. (2025). Genetic Risk Profiling Reveals Altered Glycosyltransferase Expression as a Predictor for Patient Outcome in Neuroblastoma. Journal of Clinical Medicine. 14(2). 527–527. 2 indexed citations

Engler, Jan Broder, Simone Bauer, Giovanni Di Liberto, et al.. (2023). MicroRNA-92a–CPEB3 axis protects neurons against inflammatory neurodegeneration. Science Advances. 9(47). eadi6855–eadi6855. 13 indexed citations

Rosenkranz, Sina C., Artem Shaposhnykov, Jan Broder Engler, et al.. (2021). Enhancing mitochondrial activity in neurons protects against neurodegeneration in a mouse model of multiple sclerosis. eLife. 10. 49 indexed citations

Rosenkranz, Sina C., Artem Shaposhnykov, Lisa Epping, et al.. (2020). TRPV4-Mediated Regulation of the Blood Brain Barrier Is Abolished During Inflammation. Frontiers in Cell and Developmental Biology. 8. 849–849. 18 indexed citations

Schattling, Benjamin, Walid Fazeli, Birgit Engeland, et al.. (2016). Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis. JCI Insight. 1(19). e89810–e89810. 23 indexed citations

Ufer, Friederike, Pablo Vargas, Jan Broder Engler, et al.. (2016). Arc/Arg3.1 governs inflammatory dendritic cell migration from the skin and thereby controls T cell activation. Science Immunology. 1(3). eaaf8665–eaaf8665. 31 indexed citations

Friese, Manuel A., Benjamin Schattling, & Lars Fugger. (2014). Mechanisms of neurodegeneration and axonal dysfunction in multiple sclerosis. Nature Reviews Neurology. 10(4). 225–238. 455 indexed citations breakdown →

Schattling, Benjamin, et al.. (2014). Acquired channelopathies as contributors to development and progression of multiple sclerosis. Experimental Neurology. 262. 28–36. 15 indexed citations

Gelderblom, Mathias, Nico Melzer, Benjamin Schattling, et al.. (2014). Transient Receptor Potential Melastatin Subfamily Member 2 Cation Channel Regulates Detrimental Immune Cell Invasion in Ischemic Stroke. Stroke. 45(11). 3395–3402. 75 indexed citations

10.

Gelderblom, Mathias, Benjamin Schattling, Peter Ludewig, et al.. (2013). Plasma levels of neuron specific enolase quantify the extent of neuronal injury in murine models of ischemic stroke and multiple sclerosis. Neurobiology of Disease. 59. 177–182. 18 indexed citations

11.

Schattling, Benjamin, Karin Steinbach, Edda Thies, et al.. (2012). TRPM4 cation channel mediates axonal and neuronal degeneration in experimental autoimmune encephalomyelitis and multiple sclerosis. Nature Medicine. 18(12). 1805–1811. 165 indexed citations

12.

Choe, Chi‐un, Evangelos Karamatskos, Benjamin Schattling, et al.. (2012). A clinical and neurobiological case of IgM NMDA receptor antibody associated encephalitis mimicking bipolar disorder. Psychiatry Research. 208(2). 194–196. 29 indexed citations

13.

Schattling, Benjamin, Klaus H. Hoffmann, Markus Stüecker, et al.. (2009). Upregulation of cathepsin S in psoriatic keratinocytes. Experimental Dermatology. 19(8). e80–8. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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