Benjamin N. Ostendorf

1.2k total citations · 1 hit paper
19 papers, 493 citations indexed

About

Benjamin N. Ostendorf is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Benjamin N. Ostendorf has authored 19 papers receiving a total of 493 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Hematology and 4 papers in Genetics. Recurrent topics in Benjamin N. Ostendorf's work include Chronic Myeloid Leukemia Treatments (5 papers), Chronic Lymphocytic Leukemia Research (4 papers) and Eosinophilic Disorders and Syndromes (2 papers). Benjamin N. Ostendorf is often cited by papers focused on Chronic Myeloid Leukemia Treatments (5 papers), Chronic Lymphocytic Leukemia Research (4 papers) and Eosinophilic Disorders and Syndromes (2 papers). Benjamin N. Ostendorf collaborates with scholars based in Germany and United States. Benjamin N. Ostendorf's co-authors include Sohail F. Tavazoie, Bernardo Tavora, Hani Goodarzi, Veena Padmanaban, Isabel Keller, Zachary Kerner, Roger Vaughan, Alana L. Welm, Xuhang Liu and Olav Olsen and has published in prestigious journals such as Nature, Nucleic Acids Research and Nature Medicine.

In The Last Decade

Benjamin N. Ostendorf

17 papers receiving 488 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Neuronal substance P drives metastasis through an extracellular RNA–TLR7 axis

2024 74 citations Veena Padmanaban, Isabel Keller et al. Nature profile →

Peers

Countries citing papers authored by Benjamin N. Ostendorf

Since Specialization

Citations

This map shows the geographic impact of Benjamin N. Ostendorf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin N. Ostendorf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin N. Ostendorf more than expected).

Fields of papers citing papers by Benjamin N. Ostendorf

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin N. Ostendorf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin N. Ostendorf. The network helps show where Benjamin N. Ostendorf may publish in the future.

Co-authorship network of co-authors of Benjamin N. Ostendorf

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin N. Ostendorf. A scholar is included among the top collaborators of Benjamin N. Ostendorf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin N. Ostendorf. Benjamin N. Ostendorf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

#	Work	Indexed citations
1	BEscreen: a versatile toolkit to design base editing libraries 2025 ·Nucleic Acids Research ·(unknown), Shuang Liu, Lars Bullinger, Benjamin N. Ostendorf	0
2	Neuronal substance P drives metastasis through an extracellular RNA–TLR7 axis breakdown → 2024 ·Nature ·Veena Padmanaban, Isabel Keller, (unknown), Benjamin N. Ostendorf, Zachary Kerner, Sohail F. Tavazoie	74
3	Apolipoprotein E2 Stimulates Protein Synthesis and Promotes Melanoma Progression and Metastasis 2023 ·Cancer Research ·(unknown), Benjamin N. Ostendorf, (unknown), Sohail F. Tavazoie	5
4	Functional genetic screen identifies ITPR3/calcium/RELB axis as a driver of colorectal cancer metastatic liver colonization 2022 ·Developmental Cell ·Ryan H. Moy, Alexander Nguyen, Jia Min Loo, Norihiro Yamaguchi, (unknown), Balaji Santhanam, Benjamin N. Ostendorf, Yushuai Wu, Saeed Tavazoie, Sohail F. Tavazoie, (unknown), (unknown)	13
5	Common human genetic variants of APOE impact murine COVID-19 mortality 2022 ·Nature ·Benjamin N. Ostendorf, Mira Patel, (unknown), Hans-Heinrich Hoffmann, Sebastian E. Carrasco, Charles M. Rice, Sohail F. Tavazoie	38
6	Functional Genomics In Vivo Reveal Metabolic Dependencies of Pancreatic Cancer Cells 2020 ·Cell Metabolism ·Xiphias Ge Zhu, Aleksey Chudnovskiy, Lou Baudrier, (unknown), Yuyang Liu, Benjamin N. Ostendorf, Norihiro Yamaguchi, (unknown), Bernardo Tavora, Rebecca C. Timson, Søren Heissel, Elisa de Stanchina, Henrik Molina, Gabriel D. Victora, Hani Goodarzi, Kıvanç Birsoy	90
7	Tumoural activation of TLR3–SLIT2 axis in endothelium drives metastasis 2020 ·Nature ·Bernardo Tavora, (unknown), (unknown), (unknown), (unknown), (unknown), Benjamin N. Ostendorf, Xuhang Liu, (unknown), Olav Olsen, Alana L. Welm, Hani Goodarzi, Sohail F. Tavazoie	106
8	Common germline variants of the human APOE gene modulate melanoma progression and survival 2020 ·Nature Medicine ·Benjamin N. Ostendorf, (unknown), (unknown), (unknown), Bernardo Tavora, Roger Vaughan, Sohail F. Tavazoie	70
9	Phenotypic characterization of aberrant stem and progenitor cell populations in myelodysplastic syndromes 2018 ·PLoS ONE ·Benjamin N. Ostendorf, (unknown), Anne Flörcken, Jörg Westermann	19
10	Myeloid-derived suppressor cells in human peripheral blood: Optimized quantification in healthy donors and patients with metastatic renal cell carcinoma 2015 ·Immunology Letters ·Anne Flörcken, (unknown), (unknown), Anne Gerhardt, Benjamin N. Ostendorf, Bernd Dörken, Antonio Pezzutto, Jörg Westermann	21
11	BCR-ABL Mutation-Guided Therapy for CML Blast Crisis: A Case Report 2015 ·SHILAP Revista de lepidopterología ·Benjamin N. Ostendorf	2
12	BCR-ABL Mutation-Guided Therapy for CML Blast Crisis: A Case Report 2015 ·Biomarker Insights ·Benjamin N. Ostendorf, Hendrik Nogai, Claudia D. Baldus, Thomas Burmeister, Renate Arnold	0
13	Association between low uric acid levels and acute graft-versus-host disease 2014 ·Annals of Hematology ·Benjamin N. Ostendorf, Olga Blau, Lutz Uharek, Igor Wolfgang Blau, Olaf Penack	14
14	Synchronous tuberculosis, Epstein-Barr virus-associated lymphoproliferative disorder and cytomegalovirus infection in an allogeneic transplant recipient: a case report 2014 ·SpringerPlus ·Benjamin N. Ostendorf, Christian Jehn, Lam Vuong, Hendrik Nogai, (unknown), Bernhard Gebauer, Olaf Penack, Igor Wolfgang Blau, Ioannis Anagnostopoulos, Renate Arnold	6
15	Nilotinib 2014 ·Recent results in cancer research ·Benjamin N. Ostendorf, Philipp le Coutre, Theo D. Kim, Alfonso Quintás‐Cardama	16
16	Severe radiotoxicity in an allogeneic transplant recipient with a heterozygous ATM mutation 2014 ·European Journal Of Haematology ·Benjamin N. Ostendorf, Theis H. Terwey, Philipp Hemmati, Dirk Böhmer, Uwe Pleyer, Renate Arnold	5
17	A Novel Domain Regulating Degradation of the Glomerular Slit Diaphragm Protein Podocin in Cell Culture Systems 2013 ·PLoS ONE ·Markus Gödel, Benjamin N. Ostendorf, (unknown), (unknown), Tobias B. Huber	11
18	Allogeneic Stem Cell Transplantation In Adults With High Risk Acute Lymphoblastic Leukemia (ALL) In 1. Remission (CR1): Promising, But Strongly Influenced By ALL Subtypes 2013 ·Blood ·Renate Arnold, Theis H. Terwey, Giang Lam Vuong, Benjamin N. Ostendorf, Olaf Penack, Gero Massenkeil, Nicola Gökbuget, Philipp Hemmati	2
19	Human Staphylococcus intermedius Infection in a Patient With Postradiation Changes 2011 ·Infectious Diseases in Clinical Practice ·Benjamin N. Ostendorf, Richard L. Oehler, John N. Greene	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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