Benjamin J. Lamont

1.4k total citations
17 papers, 1.2k citations indexed

About

Benjamin J. Lamont is a scholar working on Surgery, Endocrinology, Diabetes and Metabolism and Physiology. According to data from OpenAlex, Benjamin J. Lamont has authored 17 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Surgery, 9 papers in Endocrinology, Diabetes and Metabolism and 7 papers in Physiology. Recurrent topics in Benjamin J. Lamont's work include Pancreatic function and diabetes (11 papers), Diabetes Treatment and Management (8 papers) and Metabolism, Diabetes, and Cancer (6 papers). Benjamin J. Lamont is often cited by papers focused on Pancreatic function and diabetes (11 papers), Diabetes Treatment and Management (8 papers) and Metabolism, Diabetes, and Cancer (6 papers). Benjamin J. Lamont collaborates with scholars based in Australia, Canada and United States. Benjamin J. Lamont's co-authors include Daniel J. Drucker, Adriano Maida, Safina Ali, Sofianos Andrikopoulos, Laurie L. Baggio, Jacqueline A. Koehler, Herbert Y. Gaisano, Yazhou Li, Edwin Kwan and Theodore J. Brown and has published in prestigious journals such as Journal of Clinical Investigation, Nature Medicine and Diabetes.

In The Last Decade

Benjamin J. Lamont

17 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Benjamin J. Lamont Australia 16 729 551 521 234 121 17 1.2k
Sandrine Gremlich Switzerland 13 574 0.8× 681 1.2× 505 1.0× 219 0.9× 68 0.6× 22 1.2k
Kazuyo Suzuki Japan 19 487 0.7× 304 0.6× 531 1.0× 336 1.4× 73 0.6× 40 1.1k
Scott B. Widenmaier Canada 14 393 0.5× 427 0.8× 672 1.3× 230 1.0× 202 1.7× 23 1.2k
Lingguang Cui United States 15 525 0.7× 600 1.1× 653 1.3× 480 2.1× 170 1.4× 17 1.6k
Gustaf N. Hendrick United States 14 589 0.8× 522 0.9× 374 0.7× 312 1.3× 114 0.9× 18 1.1k
Elodie M. Varin Canada 15 509 0.7× 358 0.6× 325 0.6× 152 0.6× 113 0.9× 18 885
Mitsuru Hashiramoto Japan 19 342 0.5× 470 0.9× 675 1.3× 289 1.2× 267 2.2× 44 1.3k
Patrizia Borboni Italy 14 314 0.4× 300 0.5× 456 0.9× 158 0.7× 89 0.7× 21 881
Yukiko Toyofuku Japan 10 390 0.5× 546 1.0× 266 0.5× 158 0.7× 187 1.5× 13 1.1k
Kimitaka Shibue Japan 12 369 0.5× 284 0.5× 245 0.5× 160 0.7× 55 0.5× 26 670

Countries citing papers authored by Benjamin J. Lamont

Since Specialization
Citations

This map shows the geographic impact of Benjamin J. Lamont's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin J. Lamont with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin J. Lamont more than expected).

Fields of papers citing papers by Benjamin J. Lamont

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin J. Lamont. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin J. Lamont. The network helps show where Benjamin J. Lamont may publish in the future.

Co-authorship network of co-authors of Benjamin J. Lamont

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin J. Lamont. A scholar is included among the top collaborators of Benjamin J. Lamont based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin J. Lamont. Benjamin J. Lamont is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Mangiafico, Salvatore, et al.. (2017). Dapagliflozin improves insulin resistance and glucose intolerance in a novel transgenic rat model of chronic glucose overproduction and glucose toxicity. Diabetes Obesity and Metabolism. 19(8). 1135–1146. 28 indexed citations
2.
Xirouchaki, Chrysovalantou E., Salvatore Mangiafico, Zheng Ruan, et al.. (2016). Impaired glucose metabolism and exercise capacity with muscle-specific glycogen synthase 1 (gys1) deletion in adult mice. Molecular Metabolism. 5(3). 221–232. 50 indexed citations
3.
Lamont, Benjamin J., et al.. (2016). A low-carbohydrate high-fat diet increases weight gain and does not improve glucose tolerance, insulin secretion or β-cell mass in NZO mice. Nutrition and Diabetes. 6(2). e194–e194. 40 indexed citations
4.
Campbell, Jonathan E., John R. Ussher, Erin E. Mulvihill, et al.. (2015). TCF1 links GIPR signaling to the control of beta cell function and survival. Nature Medicine. 22(1). 84–90. 110 indexed citations
5.
Lamont, Benjamin J. & Sofianos Andrikopoulos. (2014). Hope and fear for new classes of type 2 diabetes drugs: is there preclinical evidence that incretin-based therapies alter pancreatic morphology?. Journal of Endocrinology. 221(1). T43–T61. 20 indexed citations
6.
Shulkes, Arthur, Jonathan P. Whitehead, Tony Tiganis, et al.. (2012). The Role of Liver Fructose-1,6-Bisphosphatase in Regulating Appetite and Adiposity. Diabetes. 61(5). 1122–1132. 42 indexed citations
7.
Ali, Safina, Benjamin J. Lamont, Maureen Charron, & Daniel J. Drucker. (2011). Dual elimination of the glucagon and GLP-1 receptors in mice reveals plasticity in the incretin axis. Journal of Clinical Investigation. 121(5). 1917–1929. 86 indexed citations
8.
Lamont, Benjamin J., Yazhou Li, Edwin Kwan, et al.. (2011). Pancreatic GLP-1 receptor activation is sufficient for incretin control of glucose metabolism in mice. Journal of Clinical Investigation. 122(1). 388–402. 143 indexed citations
9.
Maida, Adriano, et al.. (2010). Metformin regulates the incretin receptor axis via a pathway dependent on peroxisome proliferator-activated receptor-α in mice. Diabetologia. 54(2). 339–349. 253 indexed citations
10.
Koehler, Jacqueline A., Laurie L. Baggio, Benjamin J. Lamont, Safina Ali, & Daniel J. Drucker. (2009). Glucagon-Like Peptide-1 Receptor Activation Modulates Pancreatitis-Associated Gene Expression But Does Not Modify the Susceptibility to Experimental Pancreatitis in Mice. Diabetes. 58(9). 2148–2161. 130 indexed citations
11.
Fam, Barbara C., Amy R. Blair, Christian Rantzau, et al.. (2008). Increased glucose production in mice overexpressing human fructose-1,6-bisphosphatase in the liver. American Journal of Physiology-Endocrinology and Metabolism. 295(5). E1132–E1141. 28 indexed citations
12.
Lamont, Benjamin J. & Daniel J. Drucker. (2007). Differential Antidiabetic Efficacy of Incretin Agonists Versus DPP-4 Inhibition in High Fat–Fed Mice. Diabetes. 57(1). 190–198. 107 indexed citations
13.
Lamont, Benjamin J., Barbara C. Fam, Melkam A. Kebede, et al.. (2006). Expression of Human Fructose-1,6-Bisphosphatase in the Liver of Transgenic Mice Results in Increased Glycerol Gluconeogenesis. Endocrinology. 147(6). 2764–2772. 34 indexed citations
14.
Carey, Andrew L., Benjamin J. Lamont, Sofianos Andrikopoulos, et al.. (2003). Interleukin-6 gene expression is increased in insulin-resistant rat skeletal muscle following insulin stimulation. Biochemical and Biophysical Research Communications. 302(4). 837–840. 19 indexed citations
15.
Lamont, Benjamin J., Sofianos Andrikopoulos, Jennifer M. Favaloro, et al.. (2003). Peripheral insulin resistance develops in transgenic rats overexpressing phosphoenolpyruvate carboxykinase in the kidney. Diabetologia. 46(10). 1338–1347. 19 indexed citations
16.
Reuck, Jacques De, D. Decoo, Benjamin J. Lamont, et al.. (1999). Acetazolamide Vasoreactivity in Vascular Dementia: A Positron Emission Tomographic Study. European Neurology. 41(1). 31–36. 39 indexed citations
17.
Driscoll, G. L., et al.. (1991). Positive Chlamydial Serology and its Effect on Factors Influencing Outcome of IVF Treatment. Australian and New Zealand Journal of Obstetrics and Gynaecology. 31(2). 145–147. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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