Ben J. Roberts

801 total citations
10 papers, 685 citations indexed

About

Ben J. Roberts is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, Ben J. Roberts has authored 10 papers receiving a total of 685 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Cancer Research and 3 papers in Epidemiology. Recurrent topics in Ben J. Roberts's work include Cancer, Hypoxia, and Metabolism (5 papers), RNA modifications and cancer (3 papers) and Ubiquitin and proteasome pathways (3 papers). Ben J. Roberts is often cited by papers focused on Cancer, Hypoxia, and Metabolism (5 papers), RNA modifications and cancer (3 papers) and Ubiquitin and proteasome pathways (3 papers). Ben J. Roberts collaborates with scholars based in Australia, United States and Italy. Ben J. Roberts's co-authors include Susan E. Shoaf, Murray L. Whitelaw, Byoung‐Joon Song, Sang S. Park, Yunjo Soh, Martin Lewis, Kyu‐Shik Jeong, Byoung‐Joon Song, Leanne M. Dibbens and Zeeshan Shaukat and has published in prestigious journals such as Journal of Biological Chemistry, Oncogene and Biochemical and Biophysical Research Communications.

In The Last Decade

Ben J. Roberts

10 papers receiving 669 citations

Peers

Countries citing papers authored by Ben J. Roberts

Since Specialization

Citations

This map shows the geographic impact of Ben J. Roberts's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ben J. Roberts with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ben J. Roberts more than expected).

Fields of papers citing papers by Ben J. Roberts

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ben J. Roberts. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ben J. Roberts. The network helps show where Ben J. Roberts may publish in the future.

Co-authorship network of co-authors of Ben J. Roberts

This figure shows the co-authorship network connecting the top 25 collaborators of Ben J. Roberts. A scholar is included among the top collaborators of Ben J. Roberts based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ben J. Roberts. Ben J. Roberts is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Functional Effects of Epilepsy Associated KCNT1 Mutations Suggest Pathogenesis via Aberrant Inhibitory Neuronal Activity 2022 ·International Journal of Molecular Sciences ·Grigori Y. Rychkov, Zeeshan Shaukat, (unknown), (unknown), Ben J. Roberts, Claudia Bonardi, Guido Rubboli, Brandon Meaney, Robyn Whitney, Rikke S. Møller, Michael G. Ricos, Leanne M. Dibbens	4
2	HIF-1α and EPAS ubiquitination mediated by the VHL tumour suppressor involves flexibility in the ubiquitination mechanism, similar to other RING E3 ligases 2006 ·Oncogene ·(unknown), Ben J. Roberts	54
3	Role of CYP2E1 activity in endoplasmic reticulum ubiquitination, proteasome association, and the unfolded protein response 2005 ·Archives of Biochemistry and Biophysics ·Martin Lewis, Ben J. Roberts	31
4	Role of the von Hippel-Lindau tumour suppressor protein in the regulation of HIF-1α and its oxygen-regulated transactivation domains at high cell density 2005 ·Oncogene ·(unknown), Ben J. Roberts	15
5	Role of nuclear and cytoplasmic localization in the tumour-suppressor activity of the von Hippel-Lindau protein 2003 ·Oncogene ·Martin Lewis, Ben J. Roberts	24
6	Role of the C-terminal α-helical domain of the von Hippel–Lindau protein in its E3 ubiquitin ligase activity 2003 ·Oncogene ·Martin Lewis, Ben J. Roberts	15
7	Degradation of the Basic Helix-Loop-Helix/Per-ARNT-Sim Homology Domain Dioxin Receptor Via the Ubiquitin/Proteasome Pathway 1999 ·Journal of Biological Chemistry ·Ben J. Roberts, Murray L. Whitelaw	107
8	Evidence of Proteasome-mediated Cytochrome P-450 Degradation 1997 ·Journal of Biological Chemistry ·Ben J. Roberts	99
9	Ethanol Induces CYP2E1 by Protein Stabilization 1995 ·Journal of Biological Chemistry ·Ben J. Roberts, Byoung‐Joon Song, Yunjo Soh, Sang S. Park, Susan E. Shoaf	230
10	Induction of CYP2E1 in Liver, Kidney, Brain and Intestine During Chronic Ethanol Administration and Withdrawal: Evidence That CYP2E1 Possesses a Rapid Phase Half-Life of 6 Hours or Less 1994 ·Biochemical and Biophysical Research Communications ·Ben J. Roberts, Susan E. Shoaf, Kyu‐Shik Jeong, Byoung‐Joon Song	106

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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