Beili Xu

776 total citations
13 papers, 644 citations indexed

About

Beili Xu is a scholar working on Epidemiology, Clinical Biochemistry and Hepatology. According to data from OpenAlex, Beili Xu has authored 13 papers receiving a total of 644 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Epidemiology, 5 papers in Clinical Biochemistry and 5 papers in Hepatology. Recurrent topics in Beili Xu's work include Liver Disease Diagnosis and Treatment (6 papers), Liver physiology and pathology (5 papers) and Advanced Glycation End Products research (5 papers). Beili Xu is often cited by papers focused on Liver Disease Diagnosis and Treatment (6 papers), Liver physiology and pathology (5 papers) and Advanced Glycation End Products research (5 papers). Beili Xu collaborates with scholars based in China. Beili Xu's co-authors include Qunyan Yao, Shuncai Zhang, Chuantao Tu, Xi Li, Jiyao Wang, Hongchun Liu, Lixin Li, Chaohui Zhou, Shuyu Li and Zheng Li and has published in prestigious journals such as Food and Chemical Toxicology, Frontiers in Pharmacology and Toxicology Letters.

In The Last Decade

Beili Xu

13 papers receiving 635 citations

Peers

Beili Xu
Beili Xu
Citations per year, relative to Beili Xu Beili Xu (= 1×) peers Karina Reyes‐Gordillo

Countries citing papers authored by Beili Xu

Since Specialization
Citations

This map shows the geographic impact of Beili Xu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beili Xu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beili Xu more than expected).

Fields of papers citing papers by Beili Xu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beili Xu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beili Xu. The network helps show where Beili Xu may publish in the future.

Co-authorship network of co-authors of Beili Xu

This figure shows the co-authorship network connecting the top 25 collaborators of Beili Xu. A scholar is included among the top collaborators of Beili Xu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beili Xu. Beili Xu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Zou, Yanting, et al.. (2023). Anti-PCSK9 Treatment Attenuates Liver Fibrosis via Inhibiting Hypoxia-Induced Autophagy in Hepatocytes. Inflammation. 46(6). 2102–2119. 7 indexed citations
2.
Xu, Beili, Shuyu Li, Yong Fang, et al.. (2021). Proprotein Convertase Subtilisin/Kexin Type 9 Promotes Gastric Cancer Metastasis and Suppresses Apoptosis by Facilitating MAPK Signaling Pathway Through HSP70 Up-Regulation. Frontiers in Oncology. 10. 609663–609663. 40 indexed citations
3.
Xu, Beili, et al.. (2020). S100A6 promotes proliferation and migration of HepG2 cells via increased ubiquitin-dependent degradation of p53. Open Medicine. 15(1). 317–326. 12 indexed citations
4.
Zou, Yanting, Shuyu Li, Beili Xu, et al.. (2019). Inhibition of Proprotein Convertase Subtilisin/Kexin Type 9 Ameliorates Liver Fibrosis via Mitigation of Intestinal Endotoxemia. Inflammation. 43(1). 251–263. 15 indexed citations
5.
Li, Xi, Qunyan Yao, Jiying Huang, et al.. (2019). Morin Hydrate Inhibits TREM-1/TLR4-Mediated Inflammatory Response in Macrophages and Protects Against Carbon Tetrachloride-Induced Acute Liver Injury in Mice. Frontiers in Pharmacology. 10. 1089–1089. 29 indexed citations
6.
Li, Xi, Qunyan Yao, Beili Xu, et al.. (2018). The Flavonoid Quercetin Ameliorates Liver Inflammation and Fibrosis by Regulating Hepatic Macrophages Activation and Polarization in Mice. Frontiers in Pharmacology. 9. 72–72. 135 indexed citations
7.
9.
Li, Xi, Hong‐Chun Liu, Qunyan Yao, et al.. (2015). Quercetin Protects Mice from ConA-Induced Hepatitis by Inhibiting HMGB1-TLR Expression and Down-Regulating the Nuclear Factor Kappa B Pathway. Inflammation. 39(1). 96–106. 45 indexed citations
10.
Tu, Chuantao, Qunyan Yao, Beili Xu, et al.. (2012). Protective effects of curcumin against hepatic fibrosis induced by carbon tetrachloride: Modulation of high-mobility group box 1, Toll-like receptor 4 and 2 expression. Food and Chemical Toxicology. 50(9). 3343–3351. 82 indexed citations
11.
Tu, Chuantao, Qunyan Yao, Beili Xu, & Shuncai Zhang. (2012). Curcumin Protects Against Concanavalin A-Induced Hepatitis in Mice Through Inhibiting the Cytoplasmic Translocation and Expression of High Mobility Group Box 1. Inflammation. 36(1). 206–215. 23 indexed citations
12.
Yao, Qunyan, Beili Xu, Jiyao Wang, et al.. (2012). Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats. BMC Complementary and Alternative Medicine. 12(1). 156–156. 104 indexed citations
13.
Jiang, Wanglin, et al.. (2009). Cardioprotection with forsythoside B in rat myocardial ischemia-reperfusion injury: Relation to inflammation response. Phytomedicine. 17(8-9). 635–639. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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